Eduardo G. Lapetina
- Molecular Biology top 5%
- Physiology top 2%
- Cellular and Molecular Neuroscience top 5%
- Immunology top 5%
- Biochemistry top 0.5%
- Co-authors
- Bernhard BrünePedro CuatrecasasRobert H. MichellSusanne MohrWolfgang SiessThomas S. McCormickM. Motasim BillahMotasim Billah
- Topics
- Protein Kinase Regulation and GTPase Signaling (27 papers)Platelet Disorders and Treatments (20 papers)Nitric Oxide and Endothelin Effects (14 papers)
- Cited by
- BiochemistryHematologyPhysiology
- Partner nations
- United StatesArgentinaUnited Kingdom
In The Last Decade
Eduardo G. Lapetina
87 papers receiving 4.2k citations
Peers
Comparison fields: 5 of 109
- Molecular Biology 2.4k
- Physiology 963
- Cellular and Molecular Neuroscience 540
- Immunology 527
- Biochemistry 517
Countries citing papers authored by Eduardo G. Lapetina
This map shows the geographic impact of Eduardo G. Lapetina's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Eduardo G. Lapetina with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Eduardo G. Lapetina more than expected).
Fields of papers citing papers by Eduardo G. Lapetina
This network shows the impact of papers produced by Eduardo G. Lapetina. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Eduardo G. Lapetina. The network helps show where Eduardo G. Lapetina may publish in the future.
Co-authorship network of co-authors of Eduardo G. Lapetina
This figure shows the co-authorship network connecting the top 25 collaborators of Eduardo G. Lapetina. A scholar is included among the top collaborators of Eduardo G. Lapetina based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Eduardo G. Lapetina. Eduardo G. Lapetina is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 44 | |
| 2 | 10 | |
| 3 | 64 | |
| 4 | 187 | |
| 5 | 57 | |
| 6 | 117 | |
| 7 | 14 | |
| 8 | 27 | |
| 9 | 3 | |
| 10 | 33 | |
| 11 | 64 | |
| 12 | 89 | |
| 13 | 11 | |
| 14 | 25 | |
| 15 | 59 | |
| 16 | 6 | |
| 17 | 7 | |
| 18 | 19 | |
| 19 | 27 | |
| 20 | 13 |
About Eduardo G. Lapetina
Eduardo G. Lapetina is a scholar working on Hematology, Genetics and Physiology, having authored 87 papers that have together received 4.4k indexed citations. Recurring topics across this work include Protein Kinase Regulation and GTPase Signaling (27 papers), Platelet Disorders and Treatments (20 papers) and Nitric Oxide and Endothelin Effects (14 papers). The work is most often cited by research in Biochemistry (517 citations), Hematology (439 citations) and Physiology (963 citations). Eduardo G. Lapetina has collaborated with scholars based in United States, Argentina and United Kingdom. Frequent co-authors include Bernhard Brüne, Pedro Cuatrecasas, Robert H. Michell, Susanne Mohr, Wolfgang Siess, Thomas S. McCormick, M. Motasim Billah, Motasim Billah, Hazem Hallak and Michael F. Crouch. Their work appears in journals such as Nature, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.