Michael F. Crouch

2.0k total citations
59 papers, 1.7k citations indexed

About

Michael F. Crouch is a scholar working on Molecular Biology, Cell Biology and Cellular and Molecular Neuroscience. According to data from OpenAlex, Michael F. Crouch has authored 59 papers receiving a total of 1.7k indexed citations (citations by other indexed papers that have themselves been cited), including 44 papers in Molecular Biology, 15 papers in Cell Biology and 14 papers in Cellular and Molecular Neuroscience. Recurrent topics in Michael F. Crouch's work include Protein Kinase Regulation and GTPase Signaling (21 papers), Receptor Mechanisms and Signaling (12 papers) and Cellular transport and secretion (7 papers). Michael F. Crouch is often cited by papers focused on Protein Kinase Regulation and GTPase Signaling (21 papers), Receptor Mechanisms and Signaling (12 papers) and Cellular transport and secretion (7 papers). Michael F. Crouch collaborates with scholars based in Australia, United States and United Kingdom. Michael F. Crouch's co-authors include Francis S. Willard, Leise A. Berven, E G Lapetina, Ian A. Hendry, Eduardo G. Lapetina, Hugh D. Campbell, Ljubov Simson, Clarke R. Raymond, Stephen Redman and Klaus I. Matthaei and has published in prestigious journals such as Nature, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.

In The Last Decade

Michael F. Crouch

58 papers receiving 1.7k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Michael F. Crouch Australia 25 1.1k 370 351 167 160 59 1.7k
Miranda van Triest Netherlands 11 1.1k 1.0× 233 0.6× 283 0.8× 120 0.7× 121 0.8× 11 1.5k
Shinichiro Toki Japan 13 1.3k 1.1× 362 1.0× 182 0.5× 129 0.8× 145 0.9× 28 1.9k
Giorgio Rovelli Switzerland 20 746 0.6× 418 1.1× 245 0.7× 154 0.9× 219 1.4× 24 1.4k
Tammy M. Seasholtz United States 17 1.4k 1.2× 206 0.6× 363 1.0× 114 0.7× 331 2.1× 25 1.8k
Eric Boursier France 2 1.6k 1.4× 436 1.2× 269 0.8× 234 1.4× 342 2.1× 2 2.4k
Craig E. Crosson United States 33 1.6k 1.4× 312 0.8× 223 0.6× 110 0.7× 248 1.6× 82 3.3k
Nicolas Bourmeyster France 24 1.2k 1.0× 310 0.8× 221 0.6× 106 0.6× 166 1.0× 51 1.7k
Vera Novitskaya United Kingdom 23 1.2k 1.0× 400 1.1× 322 0.9× 133 0.8× 286 1.8× 28 1.9k
Sikha Ghosh United States 21 1.1k 0.9× 138 0.4× 492 1.4× 237 1.4× 114 0.7× 38 1.8k
Stephen J. Neame United Kingdom 11 969 0.8× 261 0.7× 306 0.9× 162 1.0× 110 0.7× 17 1.3k

Countries citing papers authored by Michael F. Crouch

Since Specialization
Citations

This map shows the geographic impact of Michael F. Crouch's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Michael F. Crouch with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Michael F. Crouch more than expected).

Fields of papers citing papers by Michael F. Crouch

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Michael F. Crouch. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Michael F. Crouch. The network helps show where Michael F. Crouch may publish in the future.

Co-authorship network of co-authors of Michael F. Crouch

This figure shows the co-authorship network connecting the top 25 collaborators of Michael F. Crouch. A scholar is included among the top collaborators of Michael F. Crouch based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Michael F. Crouch. Michael F. Crouch is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Werry, Tim D., et al.. (2008). Pharmacology of 5HT2C receptor-mediated ERK1/2 phosphorylation: Agonist-specific activation pathways and the impact of RNA editing. Biochemical Pharmacology. 76(10). 1276–1287. 15 indexed citations
2.
Sexton, Patrick M., et al.. (2006). Effective GPCR screening using ERK 1/2: applying SureFire to detect ERK 1/2 phosphorylation. 26(1). 27–28. 1 indexed citations
3.
Osmond, Ronald I. W., et al.. (2005). GPCR Screening via ERK 1/2: A Novel Platform for Screening G Protein–Coupled Receptors. SLAS DISCOVERY. 10(7). 730–737. 60 indexed citations
4.
Campbell, Hugh D., Ian S. McLennan, Leise A. Berven, et al.. (2002). Fliih, a Gelsolin-Related Cytoskeletal Regulator Essential for Early Mammalian Embryonic Development. Molecular and Cellular Biology. 22(10). 3518–3526. 75 indexed citations
5.
Crouch, Michael F., Geoffrey W. Osborne, & Francis S. Willard. (2000). The GTP-binding protein Giα translocates to kinetochores and regulates the M-G1 cell cycle transition of Swiss 3T3 cells. Cellular Signalling. 12(3). 153–163. 17 indexed citations
6.
7.
Berven, Leise A., Ian J. Frew, & Michael F. Crouch. (1999). Nitric Oxide Donors Selectively Potentiate Thrombin-Stimulated p70S6k Activity and Morphological Changes in Swiss 3T3 Cells. Biochemical and Biophysical Research Communications. 266(2). 352–360. 15 indexed citations
8.
Crouch, Michael F.. (1997). Regulation of Thrombin-Induced Stress Fibre Formation in Swiss 3T3 Cells by the 70-kDa S6 Kinase. Biochemical and Biophysical Research Communications. 233(1). 193–199. 36 indexed citations
9.
Crouch, Michael F., et al.. (1996). Signal transduction from membrane to nucleus: The special case for neurons. Neurochemical Research. 21(7). 779–785. 15 indexed citations
10.
Berven, Leise A., et al.. (1995). Evidence That the Pertussis Toxin-sensitive Trimeric GTP-binding Protein Gi2 Is Required for Agonist- and Store-activated Ca2+ Inflow in Hepatocytes. Journal of Biological Chemistry. 270(43). 25893–25897. 33 indexed citations
11.
Crouch, Michael F., et al.. (1995). Retrograde axonal transport of signal transduction proteins in rat sciatic nerve. Brain Research. 690(1). 55–63. 77 indexed citations
12.
Crouch, Michael F., et al.. (1994). Retrograde Axonal Transport of the α‐Subunit of the GTP‐binding Protein Gz in Mouse Sciatic Nerve: a Potential Pathway for Signal Transduction In Neurons. European Journal of Neuroscience. 6(4). 626–631. 22 indexed citations
13.
Crouch, Michael F. & Ljubov Simson. (1994). The GTP-binding protein Giα2 is directly linked to and substrate of a serine kinase in Balb/c3T3 cells. Cellular Signalling. 6(6). 695–706.
14.
15.
Crouch, Michael F. & Ian A. Hendry. (1993). Giα and Giβ are part of a signalling complex in Balb/c3T3 cells: Phosphorylation of Giβ in growth factor-activated fibroblasts. Cellular Signalling. 5(1). 41–52. 9 indexed citations
16.
Torti, Mauro, Michael F. Crouch, & Eduardo G. Lapetina. (1992). Epinephrine induces association of pp60src with Giα in human platelets. Biochemical and Biophysical Research Communications. 186(1). 440–447. 27 indexed citations
17.
Hendry, Ian A. & Michael F. Crouch. (1991). Retrograde axonal transport of the GTP-binding protein Giα: A potential neurotrophic intra-axonal messenger. Neuroscience Letters. 133(1). 29–32. 28 indexed citations
18.
Lapetina, Eduardo G. & Michael F. Crouch. (1989). The Relationship between Phospholipases A2 and C in Signal Transduction. Annals of the New York Academy of Sciences. 559(1). 153–157. 7 indexed citations
19.
Crouch, Michael F. & Eduardo G. Lapetina. (1988). No direct correlation between Ca2+ mobilization and dissociation of Gi during platelet phospholipase A2 activation. Biochemical and Biophysical Research Communications. 153(1). 21–30. 27 indexed citations
20.
Crouch, Michael F. & Eduardo G. Lapetina. (1988). The Na+H+ antiporter is not involved in potentiation of thrombin-induced responses by epinephrine. Biochemical and Biophysical Research Communications. 151(1). 178–186. 11 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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