Diane Crighton

2.7k total citations · 1 hit paper
24 papers, 2.1k citations indexed

About

Diane Crighton is a scholar working on Oncology, Molecular Biology and Cell Biology. According to data from OpenAlex, Diane Crighton has authored 24 papers receiving a total of 2.1k indexed citations (citations by other indexed papers that have themselves been cited), including 17 papers in Oncology, 16 papers in Molecular Biology and 7 papers in Cell Biology. Recurrent topics in Diane Crighton's work include Cancer-related Molecular Pathways (13 papers), Cellular Mechanics and Interactions (7 papers) and Cell death mechanisms and regulation (5 papers). Diane Crighton is often cited by papers focused on Cancer-related Molecular Pathways (13 papers), Cellular Mechanics and Interactions (7 papers) and Cell death mechanisms and regulation (5 papers). Diane Crighton collaborates with scholars based in United Kingdom, Netherlands and Italy. Diane Crighton's co-authors include Kevin M. Ryan, Simon Wilkinson, Jim O’Prey, Tim Crook, Ornella Garrone, Nelofer Syed, Milena Gasco, Paul R. Harrison, Paul Smith and Michael F. Olson and has published in prestigious journals such as Cell, Journal of Clinical Investigation and The Journal of Cell Biology.

In The Last Decade

Diane Crighton

24 papers receiving 2.1k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

DRAM, a p53-Induced Modulator of Autophagy, Is Critical for Apoptosis

2006 1.1k citations Diane Crighton, Simon Wilkinson et al. Cell profile →

Peers

Countries citing papers authored by Diane Crighton

Since Specialization

Citations

This map shows the geographic impact of Diane Crighton's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Diane Crighton with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Diane Crighton more than expected).

Fields of papers citing papers by Diane Crighton

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Diane Crighton. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Diane Crighton. The network helps show where Diane Crighton may publish in the future.

Co-authorship network of co-authors of Diane Crighton

This figure shows the co-authorship network connecting the top 25 collaborators of Diane Crighton. A scholar is included among the top collaborators of Diane Crighton based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Diane Crighton. Diane Crighton is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

#	Work	Indexed citations
1	p53 directly regulates the glycosidase FUCA1 to promote chemotherapy-induced cell death 2016 ·Cell Cycle ·Alice D. Baudot, Diane Crighton, Jim O’Prey, Joanna Somers, Pablo Sierra Gonzalez, Kevin M. Ryan	25
2	A novel small-molecule MRCK inhibitor blocks cancer cell invasion 2014 ·Cell Communication and Signaling ·Mathieu Unbekandt, Daniel R. Croft, Diane Crighton, Mokdad Mezna, (unknown), Patricia McConnell, Alexander W. Schüttelkopf, (unknown), Andrew Pannifer, (unknown), Justin Bower, Martin J. Drysdale, Michael F. Olson	2
3	Extracellular Adenosine Sensing—A Metabolic Cell Death Priming Mechanism Downstream of p53 2013 ·Molecular Cell ·Jaclyn S. Long, Diane Crighton, (unknown), Gillian Mackay, Liang Zheng, Timothy M. Palmer, Eyal Gottlieb, Kevin M. Ryan	39
4	Trailblazing LIM kinases take the lead in collective tumor cell invasion 2011 ·PubMed ·Diane Crighton, Michael F. Olson	2
5	Co-Crystal Structures of Inhibitors with MRCKβ, a Key Regulator of Tumor Cell Invasion 2011 ·PLoS ONE ·(unknown), E.R. Wheatley, Diane Crighton, Ewald Schröder, (unknown), (unknown), Mokdad Mezna, (unknown), (unknown), Andrew V. Turnbull, Michael F. Olson	37
6	Modeling and Imaging 3-Dimensional Collective Cell Invasion 2011 ·Journal of Visualized Experiments ·(unknown), Diane Crighton, Michael F. Olson	12
7	Modeling and Imaging 3-Dimensional Collective Cell Invasion 2011 ·Journal of Visualized Experiments ·(unknown), Diane Crighton, Michael F. Olson	2
8	p53-mediated transcriptional regulation and activation of the actin cytoskeleton regulatory RhoC to LIMK2 signaling pathway promotes cell survival 2010 ·Cell Research ·Daniel R. Croft, Diane Crighton, Michael S. Samuel, Filipe C. Lourenço, June Munro, (unknown), Karim Bensaad, Karen H. Vousden, Owen J. Sansom, Kevin M. Ryan, Michael F. Olson	64
9	p53-mediated induction of Noxa and p53AIP1 requires NFκB 2010 ·Cell Cycle ·Jim O’Prey, Diane Crighton, Ángel G. Martín, Karen H. Vousden, Howard O. Fearnhead, Kevin M. Ryan	32
10	TSC-22D1 isoforms have opposing roles in mammary epithelial cell survival 2009 ·Cell Death and Differentiation ·Camille Huser, Marie Anne Pringle, Victoria J. Heath, (unknown), Howard Kendrick, Matthew J. Smalley, Diane Crighton, Kevin M. Ryan, B.A. Gusterson, Torsten Stein	12
11	Activation of p73 and induction of Noxa by DNA damage requires NF-kappa B 2009 ·Aging ·Ángel G. Martín, Jason P. Trama, Diane Crighton, Kevin M. Ryan, Howard O. Fearnhead	31
12	TSC22 in mammary gland development and breast cancer 2008 ·Breast Cancer Research ·Camille Huser, Victoria J. Heath, Marie Anne Pringle, (unknown), Diane Crighton, Kevin M. Ryan, Gareth J. Inman, Torsten Stein, B.A. Gusterson	1
13	DRAM Links Autophagy to p53 and Programmed Cell Death 2007 ·Autophagy ·Diane Crighton, Simon Wilkinson, Kevin M. Ryan	168
14	A p53-derived apoptotic peptide derepresses p73 to cause tumor regression in vivo 2007 ·Journal of Clinical Investigation ·Helen Bell, Christine Dufès, Jim O’Prey, Diane Crighton, Daniele Bergamaschi, Xin Lü, Andreas G. Schätzlein, Karen H. Vousden, Kevin M. Ryan	59
15	p73 regulates DRAM-independent autophagy that does not contribute to programmed cell death 2007 ·Cell Death and Differentiation ·Diane Crighton, Jim O’Prey, Helen Bell, Kevin M. Ryan	82
16	DRAM, a p53-Induced Modulator of Autophagy, Is Critical for Apoptosis breakdown → 2006 ·Cell ·Diane Crighton, Simon Wilkinson, Jim O’Prey, Nelofer Syed, Paul Smith, Paul R. Harrison, Milena Gasco, Ornella Garrone, Tim Crook, Kevin M. Ryan	1093
17	Splicing DNA-damage responses to tumour cell death 2004 ·Biochimica et Biophysica Acta (BBA) - Reviews on Cancer ·Diane Crighton, Kevin M. Ryan	48
18	p53 represses RNA polymerase III transcription by targeting TBP and inhibiting promoter occupancy by TFIIIB 2003 ·The EMBO Journal ·Diane Crighton	112
19	Several regions of p53 are involved in repression of RNA polymerase III transcription 2002 ·Oncogene ·Torsten Stein, Diane Crighton, (unknown), Jo Milner, Robert J. White	24
20	RNA polymerase III transcription can be derepressed by oncogenes or mutations that compromise p53 function in tumours and Li-Fraumeni syndrome 2002 ·Oncogene ·Torsten Stein, Diane Crighton, John M. Boyle, Jennifer M. Varley, Robert J. White	29

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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