John M. Boyle

1.2k total citations
21 papers, 872 citations indexed

About

John M. Boyle is a scholar working on Molecular Biology, Genetics and Physiology. According to data from OpenAlex, John M. Boyle has authored 21 papers receiving a total of 872 indexed citations (citations by other indexed papers that have themselves been cited), including 12 papers in Molecular Biology, 6 papers in Genetics and 5 papers in Physiology. Recurrent topics in John M. Boyle's work include DNA Repair Mechanisms (4 papers), Genomic variations and chromosomal abnormalities (4 papers) and Survey Methodology and Nonresponse (3 papers). John M. Boyle is often cited by papers focused on DNA Repair Mechanisms (4 papers), Genomic variations and chromosomal abnormalities (4 papers) and Survey Methodology and Nonresponse (3 papers). John M. Boyle collaborates with scholars based in United Kingdom and United States. John M. Boyle's co-authors include N. Symonds, Jacob M. Vogan, Dirk Hockemeyer, Michael S. Blaiss, Eli O. Meltzer, M. Jennifer Derebery, Tina Wagner, Kunitoshi Chiba, Robert M. Naclerio and Stuart Stoloff and has published in prestigious journals such as Science, Genes & Development and SHILAP Revista de lepidopterología.

In The Last Decade

John M. Boyle

20 papers receiving 840 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
John M. Boyle United Kingdom 13 420 330 209 159 87 21 872
Chiara Rasi Sweden 11 392 0.9× 126 0.4× 297 1.4× 306 1.9× 89 1.0× 15 1.0k
Mario Cervone Italy 8 214 0.5× 79 0.2× 101 0.5× 84 0.5× 55 0.6× 24 571
Hironori Niizeki Japan 18 132 0.3× 152 0.5× 119 0.6× 101 0.6× 109 1.3× 52 1.0k
Amanda J. Oliver United Kingdom 16 256 0.6× 133 0.4× 24 0.1× 70 0.4× 518 6.0× 33 1.1k
Daniel P. Kestler United States 15 423 1.0× 34 0.1× 29 0.1× 66 0.4× 77 0.9× 38 738
Robert Lawrence United Kingdom 10 192 0.5× 209 0.6× 302 1.4× 236 1.5× 33 0.4× 16 880
Leslee Sprague United States 14 194 0.5× 99 0.3× 58 0.3× 106 0.7× 95 1.1× 19 722
Kate Elizabeth Brown United States 7 260 0.6× 80 0.2× 127 0.6× 70 0.4× 28 0.3× 14 606
Feng-Qian Li United States 11 383 0.9× 155 0.5× 38 0.2× 541 3.4× 95 1.1× 11 865
J.B. Whitney United States 17 596 1.4× 118 0.4× 14 0.1× 440 2.8× 34 0.4× 38 1.0k

Countries citing papers authored by John M. Boyle

Since Specialization
Citations

This map shows the geographic impact of John M. Boyle's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by John M. Boyle with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites John M. Boyle more than expected).

Fields of papers citing papers by John M. Boyle

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by John M. Boyle. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by John M. Boyle. The network helps show where John M. Boyle may publish in the future.

Co-authorship network of co-authors of John M. Boyle

This figure shows the co-authorship network connecting the top 25 collaborators of John M. Boyle. A scholar is included among the top collaborators of John M. Boyle based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with John M. Boyle. John M. Boyle is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Lu, Robert, Richard Ho, Shirin S Jenkins, et al.. (2025). Active telomere elongation by a subclass of cancer-associated POT1 mutations. Genes & Development. 39(7-8). 445–462.
2.
Boyle, John M., et al.. (2022). Pronounced Declines in Meperidine in the US: Is the End Imminent?. SHILAP Revista de lepidopterología. 10(6). 154–154. 4 indexed citations
3.
Boyle, John M., Samuel G. Regalado, Jacob M. Vogan, et al.. (2020). Telomere length set point regulation in human pluripotent stem cells critically depends on the shelterin protein TPP1. Molecular Biology of the Cell. 31(23). 2583–2596. 12 indexed citations
4.
Mena, Elijah L., Robert A. Saxton, Achim Werner, et al.. (2018). Dimerization quality control ensures neuronal development and survival. Science. 362(6411). 59 indexed citations
5.
Boyle, John M., et al.. (2017). Characteristics of the Population of Internet Panel Members. Survey Practice. 10(4). 1–8. 8 indexed citations
6.
Chiba, Kunitoshi, et al.. (2015). Cancer-associated TERT promoter mutations abrogate telomerase silencing. eLife. 4. 198 indexed citations
7.
Meltzer, Eli O., Michael S. Blaiss, Robert M. Naclerio, et al.. (2012). Burden of allergic rhinitis: Allergies in America, Latin America, and Asia-Pacific adult surveys. Allergy and Asthma Proceedings. 33(5). 113–141. 201 indexed citations
8.
Boyle, John M., et al.. (2010). Segmented or Overlapping Dual Frame Samples in Telephone Surveys. Survey Practice. 3(6). 1–9. 5 indexed citations
9.
Boyle, John M., et al.. (2009). Cell Phone Mainly Households: Coverage and Reach for Telephone Surveys Using RDD Landline Samples. Survey Practice. 2(9). 1–10. 6 indexed citations
10.
Blaiss, Michael S., Eli O. Meltzer, M. Jennifer Derebery, & John M. Boyle. (2007). Patient and healthcare-provider perspectives on the burden of allergic rhinitis. Allergy and Asthma Proceedings. 28(3). 4–10. 36 indexed citations
12.
Stein, Torsten, Diane Crighton, John M. Boyle, Jennifer M. Varley, & Robert J. White. (2002). RNA polymerase III transcription can be derepressed by oncogenes or mutations that compromise p53 function in tumours and Li-Fraumeni syndrome. Oncogene. 21(19). 2961–2970. 29 indexed citations
13.
Williams, Kaye J., et al.. (1997). Cell cycle arrest defect in Li – Fraumeni Syndrome: a mechanism of cancer predisposition?. Oncogene. 14(3). 277–282. 28 indexed citations
14.
Pappas, George J., Mihael H. Polymeropoulos, John M. Boyle, & Jeffrey M. Trent. (1995). Regional assignment by hybrid mapping of 36 expressed sequence tags (ESTs) on human chromosome 6. Genomics. 25(1). 124–129. 14 indexed citations
15.
Santibanez‐Koref, Mauro, et al.. (1994). Physical Mapping of 43 STSs to Human Chromosome 6. Genomics. 20(2). 301–304. 8 indexed citations
16.
Menasce, Lia P., et al.. (1994). Deletion of a common region on the long arm of chromosome 6 in acute lymphoblastic leukaemia. Genes Chromosomes and Cancer. 10(1). 26–29. 37 indexed citations
17.
Menasce, Lia P., et al.. (1994). Common region of deletion on the long arm of chromosome 6 in non‐Hodgkin's lymphoma and acute lymphoblastic leukaemia. Genes Chromosomes and Cancer. 10(4). 286–288. 60 indexed citations
18.
McGown, Gail, et al.. (1993). Thirteen dinucleotide repeat polymorphisms on chromosome 6. Human Molecular Genetics. 2(12). 2196–2196. 14 indexed citations
19.
Boyle, John M.. (1969). Radiation-sensitive mutants of T4D II. T4y: Genetic characterization. Mutation Research/Fundamental and Molecular Mechanisms of Mutagenesis. 8(3). 441–449. 27 indexed citations
20.
Boyle, John M. & N. Symonds. (1969). Radiation-sensitive mutants of T4D I. T4y: A new radiation-sensitive mutant: Effect of the mutation on radiation survival, growth and recombination. Mutation Research/Fundamental and Molecular Mechanisms of Mutagenesis. 8(3). 431–439. 118 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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