Diane C. Fingar

14.8k total citations · 6 hit papers
49 papers, 8.7k citations indexed

About

Diane C. Fingar is a scholar working on Molecular Biology, Immunology and Surgery. According to data from OpenAlex, Diane C. Fingar has authored 49 papers receiving a total of 8.7k indexed citations (citations by other indexed papers that have themselves been cited), including 44 papers in Molecular Biology, 10 papers in Immunology and 4 papers in Surgery. Recurrent topics in Diane C. Fingar's work include PI3K/AKT/mTOR signaling in cancer (30 papers), Protein Kinase Regulation and GTPase Signaling (17 papers) and Metabolism, Diabetes, and Cancer (13 papers). Diane C. Fingar is often cited by papers focused on PI3K/AKT/mTOR signaling in cancer (30 papers), Protein Kinase Regulation and GTPase Signaling (17 papers) and Metabolism, Diabetes, and Cancer (13 papers). Diane C. Fingar collaborates with scholars based in United States, United Kingdom and Germany. Diane C. Fingar's co-authors include John Blenis, Andrew R. Tee, Brian Magnuson, Sofie R. Salama, Ed Harlow, Hugo A. Acosta-Jaquez, Sallie W. Smith, Rey‐Huei Chen, Leon O. Murphy and Celeste Richardson and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Genes & Development.

In The Last Decade

Diane C. Fingar

49 papers receiving 8.5k citations

Hit Papers

Target of rapamycin (TOR): an integrator of nutrient and ... 2002 2026 2010 2018 2004 2002 2011 2002 2003 250 500 750 1000

Peers

Diane C. Fingar
Comparison fields: 5 of 131
  • Molecular Biology 6.2k
  • Physiology 1.2k
  • Immunology 981
  • Cell Biology 949
  • Epidemiology 941
Replace Andrew R. Tee with:
Andrew R. Tee United Kingdom
Stephan Wullschleger Switzerland
Robert A. Saxton United States
Peter M. Finan United Kingdom
Timothy R. Peterson United States
Shomit Sengupta United States
Leon O. Murphy United States
Kenta Hara Japan
Estela Jacinto United States
Robert Latek United States
Andrew R. Tee United Kingdom View profile →
Citations per field, relative to Diane C. Fingar
Diane C. Fingar · 1×
Citations per year, relative to Diane C. Fingar
Diane C. Fingar · 1×

Countries citing papers authored by Diane C. Fingar

Since Specialization
Citations

This map shows the geographic impact of Diane C. Fingar's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Diane C. Fingar with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Diane C. Fingar more than expected).

Fields of papers citing papers by Diane C. Fingar

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Diane C. Fingar. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Diane C. Fingar. The network helps show where Diane C. Fingar may publish in the future.

Co-authorship network of co-authors of Diane C. Fingar

This figure shows the co-authorship network connecting the top 25 collaborators of Diane C. Fingar. A scholar is included among the top collaborators of Diane C. Fingar based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Diane C. Fingar. Diane C. Fingar is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
# Work Indexed citations
1 16
2 6
3 6
4 46
5 16
6 175
7 78
8 169
9 189
10 138
11 16
12 80
13 47
14 198
15
mTOR Controls Cell Cycle Progression through Its Cell Growth Effectors S6K1 and 4E-BP1/Eukaryotic Translation Initiation Factor 4E breakdown →
733
16 395
17
Tuberous sclerosis complex-1 and -2 gene products function together to inhibit mammalian target of rapamycin (mTOR)-mediated downstream signaling breakdown →
647
18
Molecular interpretation of ERK signal duration by immediate early gene products breakdown →
766
19
Mammalian cell size is controlled by mTOR and its downstream targets S6K1 and 4EBP1/eIF4E breakdown →
872
20 91

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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