David Sontag

943 total citations
14 papers, 750 citations indexed

About

David Sontag is a scholar working on Molecular Biology, Surgery and Cardiology and Cardiovascular Medicine. According to data from OpenAlex, David Sontag has authored 14 papers receiving a total of 750 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Molecular Biology, 5 papers in Surgery and 5 papers in Cardiology and Cardiovascular Medicine. Recurrent topics in David Sontag's work include Fibroblast Growth Factor Research (5 papers), Cardiac electrophysiology and arrhythmias (4 papers) and Cholangiocarcinoma and Gallbladder Cancer Studies (3 papers). David Sontag is often cited by papers focused on Fibroblast Growth Factor Research (5 papers), Cardiac electrophysiology and arrhythmias (4 papers) and Cholangiocarcinoma and Gallbladder Cancer Studies (3 papers). David Sontag collaborates with scholars based in Canada, Qatar and China. David Sontag's co-authors include William C. Cole, Peter A. Cattini, Odile Clément‐Chomienne, Michael P. Walsh, Elissavet Kardami, Ernesto A. Aiello, Farah Sheikh, Robert R. Fandrich, Kuniaki Ishii and Paul M. Kerr and has published in prestigious journals such as Circulation Research, Journal of Pharmacology and Experimental Therapeutics and American Journal of Physiology-Heart and Circulatory Physiology.

In The Last Decade

David Sontag

14 papers receiving 735 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
David Sontag Canada	10	359	355	338	189	102	14	750
Xian‐Liang Tang United States	12	399 1.1×	532 1.5×	288 0.9×	224 1.2×	136 1.3×	30	959
Sergej Belosjorow Germany	10	310 0.9×	335 0.9×	371 1.1×	143 0.8×	79 0.8×	10	800
Víctor Hernando Spain	15	404 1.1×	511 1.4×	277 0.8×	279 1.5×	96 0.9×	16	913
André P. Heinen Germany	14	343 1.0×	479 1.3×	135 0.4×	182 1.0×	59 0.6×	17	879
Alberto Cabestrero Spain	10	777 2.2×	412 1.2×	197 0.6×	199 1.1×	103 1.0×	14	1.0k
Takamichi Uchiyama Japan	12	209 0.6×	238 0.7×	103 0.3×	85 0.4×	70 0.7×	20	514
S KASSECKERT Germany	14	252 0.7×	188 0.5×	159 0.5×	73 0.4×	87 0.9×	17	539
Alexej Duschin Germany	8	292 0.8×	255 0.7×	243 0.7×	128 0.7×	170 1.7×	8	653
Marcos Poncelas Spain	12	243 0.7×	205 0.6×	156 0.5×	106 0.6×	83 0.8×	14	509
H. M. Piper Germany	16	432 1.2×	171 0.5×	275 0.8×	53 0.3×	161 1.6×	23	722

Countries citing papers authored by David Sontag

Since Specialization

Citations

This map shows the geographic impact of David Sontag's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by David Sontag with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites David Sontag more than expected).

Fields of papers citing papers by David Sontag

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by David Sontag. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by David Sontag. The network helps show where David Sontag may publish in the future.

Co-authorship network of co-authors of David Sontag

This figure shows the co-authorship network connecting the top 25 collaborators of David Sontag. A scholar is included among the top collaborators of David Sontag based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with David Sontag. David Sontag is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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14 of 14 papers shown

Yang, Jiaqi, David Sontag, Frank J. Burczynski, et al.. (2022). Comparison of growth features and cancer stem cell prevalence in intrahepatic and extrahepatic cholangiocarcinoma cell lines. Clinical and Experimental Hepatology. 8(1). 60–69. 2 indexed citations

Yang, Jiaqi, David Sontag, Yuewen Gong, & Gerald Y. Minuk. (2020). Enhanced gemcitabine cytotoxicity with knockdown of multidrug resistance protein genes in human cholangiocarcinoma cell lines. Journal of Gastroenterology and Hepatology. 36(4). 1103–1109. 6 indexed citations

Yang, Jiaqi, David Sontag, Yuewen Gong, & Gerald Y. Minuk. (2020). Alterations in chemokine receptor CCR5 activity influence tumor cell biology in human cholangiocarcinoma cell lines. Annals of Hepatology. 21. 100265–100265. 1 indexed citations

Sontag, David, et al.. (2020). The effects of hyperthermia on human hepatocellular carcinoma stem and mature cancer cells. Annals of Hepatology. 19(3). 265–268. 3 indexed citations

Yang, Jiaqi, Yuewen Gong, David Sontag, Ian R. Corbin, & Gerald Y. Minuk. (2018). Effects of low-density lipoprotein docosahexaenoic acid nanoparticles on cancer stem cells isolated from human hepatoma cell lines. Molecular Biology Reports. 45(5). 1023–1036. 14 indexed citations

Wang, Jie, David Sontag, & Peter A. Cattini. (2014). Heart-specific expression of FGF-16 and a potential role in postnatal cardioprotection. Cytokine & Growth Factor Reviews. 26(1). 59–66. 17 indexed citations

Sontag, David, Jie Wang, Elissavet Kardami, & Peter A. Cattini. (2013). FGF-2 and FGF-16 Protect Isolated Perfused Mouse Hearts from Acute Doxorubicin-Induced Contractile Dysfunction. Cardiovascular Toxicology. 13(3). 244–253. 20 indexed citations

Lu, Shun, David Sontag, Karen A. Detillieux, & Peter A. Cattini. (2008). FGF-16 is released from neonatal cardiac myocytes and alters growth-related signaling: a possible role in postnatal development. American Journal of Physiology-Cell Physiology. 294(5). C1242–C1249. 33 indexed citations

Sontag, David & Peter A. Cattini. (2003). Cloning and bacterial expression of postnatal mouse heart FGF-16. Molecular and Cellular Biochemistry. 242(1-2). 65–70. 14 indexed citations

10.

Kerr, Paul M., Odile Clément‐Chomienne, Kevin S. Thorneloe, et al.. (2001). Heteromultimeric Kv1.2-Kv1.5 Channels Underlie 4-Aminopyridine-Sensitive Delayed Rectifier K⁺Current of Rabbit Vascular Myocytes. Circulation Research. 89(11). 1038–1044. 77 indexed citations

11.

Sheikh, Farah, David Sontag, Robert R. Fandrich, Elissavet Kardami, & Peter A. Cattini. (2001). Overexpression of FGF-2 increases cardiac myocyte viability after injury in isolated mouse hearts. American Journal of Physiology-Heart and Circulatory Physiology. 280(3). H1039–H1050. 59 indexed citations

12.

Aiello, Ernesto A., Odile Clément‐Chomienne, David Sontag, Michael P. Walsh, & William C. Cole. (1996). Protein kinase C inhibits delayed rectifier K+ current in rabbit vascular smooth muscle cells. American Journal of Physiology-Heart and Circulatory Physiology. 271(1). H109–H119. 66 indexed citations

13.

Pierce, Grant N., William C. Cole, Kan‐Zhi Liu, et al.. (1993). Modulation of cardiac performance by amiloride and several selected derivatives of amiloride.. Journal of Pharmacology and Experimental Therapeutics. 265(3). 1280–1291. 58 indexed citations

14.

Cole, William C., et al.. (1991). ATP-regulated K+ channels protect the myocardium against ischemia/reperfusion damage.. Circulation Research. 69(3). 571–581. 380 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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