Víctor Hernando

1.5k total citations
16 papers, 913 citations indexed

About

Víctor Hernando is a scholar working on Pathology and Forensic Medicine, Cardiology and Cardiovascular Medicine and Emergency Medicine. According to data from OpenAlex, Víctor Hernando has authored 16 papers receiving a total of 913 indexed citations (citations by other indexed papers that have themselves been cited), including 12 papers in Pathology and Forensic Medicine, 8 papers in Cardiology and Cardiovascular Medicine and 8 papers in Emergency Medicine. Recurrent topics in Víctor Hernando's work include Cardiac Ischemia and Reperfusion (12 papers), Cardiac Arrest and Resuscitation (8 papers) and Cardiac electrophysiology and arrhythmias (6 papers). Víctor Hernando is often cited by papers focused on Cardiac Ischemia and Reperfusion (12 papers), Cardiac Arrest and Resuscitation (8 papers) and Cardiac electrophysiology and arrhythmias (6 papers). Víctor Hernando collaborates with scholars based in Spain, Chile and Palestinian Territory. Víctor Hernando's co-authors include Javier Inserte, David García‐Dorado, David García‐Dorado, Ignasi Barba, Marcos Poncelas, J INSERTE, Jordi Soler‐Soler, Marisol Ruiz‐Meana, Carmem Luíza Sartório and José A. Barrabés and has published in prestigious journals such as Circulation, Circulation Research and Cardiovascular Research.

In The Last Decade

Víctor Hernando

16 papers receiving 900 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Víctor Hernando Spain	15	511	404	279	277	199	16	913
J INSERTE Spain	9	434 0.8×	351 0.9×	196 0.7×	269 1.0×	71 0.4×	13	711
Marcos Poncelas Spain	12	205 0.4×	243 0.6×	106 0.4×	156 0.6×	103 0.5×	14	509
K Kishi Japan	8	416 0.8×	496 1.2×	146 0.5×	123 0.4×	38 0.2×	11	887
Alberto Cabestrero Spain	10	412 0.8×	777 1.9×	199 0.7×	197 0.7×	31 0.2×	14	1.0k
Maryline Abrial France	14	185 0.4×	497 1.2×	73 0.3×	164 0.6×	139 0.7×	17	741
Christof Weinbrenner Germany	14	537 1.1×	333 0.8×	285 1.0×	329 1.2×	25 0.1×	22	926
Sophie Tamareille France	15	310 0.6×	302 0.7×	120 0.4×	220 0.8×	25 0.1×	20	694
Vishnu Undyala United States	11	216 0.4×	245 0.6×	75 0.3×	113 0.4×	130 0.7×	15	669
David Sontag Canada	10	355 0.7×	359 0.9×	189 0.7×	338 1.2×	26 0.1×	14	750
Siavash Beikoghli Kalkhoran United Kingdom	12	322 0.6×	610 1.5×	68 0.2×	190 0.7×	53 0.3×	18	898

Countries citing papers authored by Víctor Hernando

Since Specialization

Citations

This map shows the geographic impact of Víctor Hernando's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Víctor Hernando with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Víctor Hernando more than expected).

Fields of papers citing papers by Víctor Hernando

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Víctor Hernando. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Víctor Hernando. The network helps show where Víctor Hernando may publish in the future.

Co-authorship network of co-authors of Víctor Hernando

This figure shows the co-authorship network connecting the top 25 collaborators of Víctor Hernando. A scholar is included among the top collaborators of Víctor Hernando based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Víctor Hernando. Víctor Hernando is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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16 of 16 papers shown

Poncelas, Marcos, et al.. (2017). Delayed, oral pharmacological inhibition of calpains attenuates adverse post-infarction remodelling. Cardiovascular Research. 113(8). 950–961. 27 indexed citations

Inserte, Javier, et al.. (2016). Studies on the role of apoptosis after transient myocardial ischemia: genetic deletion of the executioner caspases-3 and -7 does not limit infarct size and ventricular remodeling. Basic Research in Cardiology. 111(2). 18–18. 40 indexed citations

Inserte, Javier, Víctor Hernando, Marisol Ruiz‐Meana, et al.. (2014). Delayed phospholamban phosphorylation in post-conditioned heart favours Ca2+ normalization and contributes to protection. Cardiovascular Research. 103(4). 542–553. 26 indexed citations

Ruiz‐Meana, Marisol, Estefanía Núñez, Elisabet Miró‐Casas, et al.. (2014). Ischemic preconditioning protects cardiomyocyte mitochondria through mechanisms independent of cytosol. Journal of Molecular and Cellular Cardiology. 68. 79–88. 45 indexed citations

Inserte, Javier, et al.. (2013). Activation of cGMP/Protein Kinase G Pathway in Postconditioned Myocardium Depends on Reduced Oxidative Stress and Preserved Endothelial Nitric Oxide Synthase Coupling. Journal of the American Heart Association. 2(1). e005975–e005975. 55 indexed citations

Inserte, Javier, Víctor Hernando, & David García‐Dorado. (2012). Contribution of calpains to myocardial ischaemia/reperfusion injury. Cardiovascular Research. 96(1). 23–31. 120 indexed citations

Inserte, Javier, Ignasi Barba, Marcos Poncelas, et al.. (2011). cGMP/PKG pathway mediates myocardial postconditioning protection in rat hearts by delaying normalization of intracellular acidosis during reperfusion. Journal of Molecular and Cellular Cardiology. 50(5). 903–909. 59 indexed citations

Ruiz‐Meana, Marisol, Javier Inserte, Celia Fernández‐Sanz, et al.. (2011). The role of mitochondrial permeability transition in reperfusion-induced cardiomyocyte death depends on the duration of ischemia. Basic Research in Cardiology. 106(6). 1259–1268. 51 indexed citations

Hernando, Víctor, et al.. (2010). Calpain translocation and activation as pharmacological targets during myocardial ischemia/reperfusion. Journal of Molecular and Cellular Cardiology. 49(2). 271–279. 88 indexed citations

10.

Barrabés, José A., Javier Inserte, Maribel Mirabet, et al.. (2010). Antagonism of P2Y12 or GPIIb/IIIa receptors reduces platelet-mediated myocardial injury after ischaemia and reperfusion in isolated rat hearts. Thrombosis and Haemostasis. 104(7). 128–135. 46 indexed citations

11.

INSERTE, J, José A. Barrabés, Víctor Hernando, & David García‐Dorado. (2009). Orphan targets for reperfusion injury. Cardiovascular Research. 83(2). 169–178. 39 indexed citations

12.

Inserte, Javier, Ignasi Barba, Víctor Hernando, & David García‐Dorado. (2008). Delayed recovery of intracellular acidosis during reperfusion prevents calpain activation and determines protection in postconditioned myocardium. Cardiovascular Research. 81(1). 116–122. 89 indexed citations

13.

Barrabés, José A., Javier Inserte, Maribel Mirabet, et al.. (2008). Abstract 1712: The P2Y ₁₂ Antagonist Cangrelor and the GP IIb/IIIa Blocker Abciximab Reduce Platelet-Mediated Myocardial Injury After Ischemia and Reperfusion. Circulation. 118(suppl_18). 1 indexed citations

14.

INSERTE, J, Ignasi Barba, Víctor Hernando, et al.. (2007). Effect of acidic reperfusion on prolongation of intracellular acidosis and myocardial salvage. Cardiovascular Research. 77(4). 782–790. 83 indexed citations

15.

INSERTE, J, et al.. (2006). Ischemic preconditioning prevents calpain-mediated impairment of Na+/K+-ATPase activity during early reperfusion☆. Cardiovascular Research. 70(2). 364–373. 49 indexed citations

16.

Inserte, Javier, David García‐Dorado, Víctor Hernando, & Jordi Soler‐Soler. (2005). Calpain-Mediated Impairment of Na ⁺ /K ⁺ –ATPase Activity During Early Reperfusion Contributes to Cell Death After Myocardial Ischemia. Circulation Research. 97(5). 465–473. 95 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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