H. M. Piper

850 total citations
23 papers, 722 citations indexed

About

H. M. Piper is a scholar working on Molecular Biology, Cardiology and Cardiovascular Medicine and Pathology and Forensic Medicine. According to data from OpenAlex, H. M. Piper has authored 23 papers receiving a total of 722 indexed citations (citations by other indexed papers that have themselves been cited), including 16 papers in Molecular Biology, 12 papers in Cardiology and Cardiovascular Medicine and 7 papers in Pathology and Forensic Medicine. Recurrent topics in H. M. Piper's work include Cardiac Ischemia and Reperfusion (7 papers), Nitric Oxide and Endothelin Effects (6 papers) and Cardiac electrophysiology and arrhythmias (6 papers). H. M. Piper is often cited by papers focused on Cardiac Ischemia and Reperfusion (7 papers), Nitric Oxide and Endothelin Effects (6 papers) and Cardiac electrophysiology and arrhythmias (6 papers). H. M. Piper collaborates with scholars based in Germany, Israel and Spain. H. M. Piper's co-authors include K.‐D. Schlüter, Y Ladilov, Thomas Noll, Albrecht Hempel, Silvia Haffner, H Maxeiner, Gerhild Euler, Britta Siegmund, Jacqueline Heger and A. Muhs and has published in prestigious journals such as The FASEB Journal, Cardiovascular Research and American Journal of Physiology-Heart and Circulatory Physiology.

In The Last Decade

H. M. Piper

23 papers receiving 698 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
H. M. Piper Germany 16 432 275 171 161 133 23 722
Y Ladilov Germany 15 386 0.9× 231 0.8× 249 1.5× 81 0.5× 66 0.5× 17 736
Polina Gross United States 12 836 1.9× 255 0.9× 109 0.6× 161 1.0× 146 1.1× 20 1.1k
Keli Hu United States 19 587 1.4× 399 1.5× 444 2.6× 97 0.6× 135 1.0× 33 1.2k
David Sontag Canada 10 359 0.8× 338 1.2× 355 2.1× 102 0.6× 91 0.7× 14 750
Tomoko Kamishima Japan 17 471 1.1× 140 0.5× 59 0.3× 119 0.7× 147 1.1× 39 729
Ricky Malhotra United States 13 540 1.3× 368 1.3× 77 0.5× 74 0.5× 129 1.0× 15 871
Setsuya Miyata Japan 10 538 1.2× 484 1.8× 85 0.5× 72 0.4× 41 0.3× 16 914
Attila Kiss Austria 19 397 0.9× 320 1.2× 163 1.0× 198 1.2× 32 0.2× 102 1.0k
Scott M. MacDonnell United States 16 698 1.6× 539 2.0× 76 0.4× 177 1.1× 90 0.7× 28 1.2k
G L Engelmann United States 13 447 1.0× 264 1.0× 49 0.3× 107 0.7× 57 0.4× 19 740

Countries citing papers authored by H. M. Piper

Since Specialization
Citations

This map shows the geographic impact of H. M. Piper's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by H. M. Piper with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites H. M. Piper more than expected).

Fields of papers citing papers by H. M. Piper

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by H. M. Piper. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by H. M. Piper. The network helps show where H. M. Piper may publish in the future.

Co-authorship network of co-authors of H. M. Piper

This figure shows the co-authorship network connecting the top 25 collaborators of H. M. Piper. A scholar is included among the top collaborators of H. M. Piper based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with H. M. Piper. H. M. Piper is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
2.
Schlüter, K.‐D., et al.. (2008). Enhanced SERCA2A expression improves contractile performance of ventricular cardiomyocytes of rat under adrenergic stimulation. Pflügers Archiv - European Journal of Physiology. 457(2). 485–491. 15 indexed citations
3.
Piper, H. M., D. Garcia-Dorado, & Elizabeth A. Martinson. (2007). A New Look for CVR in 2008. Cardiovascular Research. 77(1). 1–1. 2 indexed citations
4.
Anwar, Maryam, et al.. (2006). Repression of anti-apoptotic genes via AP-1 as a mechanism of apoptosis induction in ventricular cardiomyocytes. Pflügers Archiv - European Journal of Physiology. 454(1). 53–61. 10 indexed citations
5.
Kuhlmann, Christoph, Astrid Most, Claudia Schaefer, et al.. (2005). Endothelin‐1‐induced proliferation of human endothelial cells depends on activation of K+ channels and Ca2+ influx. Acta Physiologica Scandinavica. 183(2). 161–169. 24 indexed citations
6.
Schäfer, Mathias, et al.. (2003). Signaling of hypoxia‐induced autonomous proliferation of endothelial cells. The FASEB Journal. 17(3). 1–23. 23 indexed citations
7.
Inserte, Javier, et al.. (2000). Influence of simulated ischemia on apoptosis induction by oxidative stress in adult cardiomyocytes of rats. American Journal of Physiology-Heart and Circulatory Physiology. 278(1). H94–H99. 21 indexed citations
8.
Noll, Thomas, et al.. (2000). ATP induces dephosphorylation of myosin light chain in endothelial cells. American Journal of Physiology-Cell Physiology. 279(3). C717–C723. 12 indexed citations
9.
Ladilov, Y, et al.. (1999). Cardioprotective effects of KB-R7943: a novel inhibitor of the reverse mode of Na+/Ca2+exchanger. American Journal of Physiology-Heart and Circulatory Physiology. 276(6). H1868–H1876. 110 indexed citations
10.
Schlüter, K.‐D. & H. M. Piper. (1999). Regulation of growth in the adult cardiomyocytes. The FASEB Journal. 13(9001). S17–22. 48 indexed citations
11.
Muhs, A., Thomas Noll, & H. M. Piper. (1997). Vinculin phosphorylation and barrier failure of coronary endothelial monolayers under energy depletion. American Journal of Physiology-Heart and Circulatory Physiology. 273(2). H608–H617. 15 indexed citations
12.
Hölschermann, Hans, Thomas Noll, Albrecht Hempel, & H. M. Piper. (1997). Dual role of cGMP in modulation of macromolecule permeability of aortic endothelial cells. American Journal of Physiology-Heart and Circulatory Physiology. 272(1). H91–H98. 29 indexed citations
13.
Hempel, Albrecht, Thomas Noll, A. Muhs, & H. M. Piper. (1996). Functional antagonism between cAMP and cGMP on permeability of coronary endothelial monolayers. American Journal of Physiology-Heart and Circulatory Physiology. 270(4). H1264–H1271. 38 indexed citations
14.
Piper, H. M., C. Balser, Y Ladilov, et al.. (1996). The role of Na+/H+ exchange in ischemia-reperfusion. Basic Research in Cardiology. 91(3). 191–202. 95 indexed citations
15.
Noll, Thomas, Albrecht Hempel, & H. M. Piper. (1996). Neuropeptide Y reduces macromolecule permeability of coronary endothelial monolayers. American Journal of Physiology-Heart and Circulatory Physiology. 271(5). H1878–H1883. 19 indexed citations
16.
Pinson, A., K.‐D. Schlüter, Xi Zhou, et al.. (1993). Alpha- and Beta-Adrenergic Stimulation of Protein Synthesis in Cultured Adult Ventricular Cardiomyocytes. Journal of Molecular and Cellular Cardiology. 25(4). 477–490. 75 indexed citations
17.
Piper, H. M. & Roberto Ferrari. (1993). Basic research in cardiology focussed issue on ?energy and cation control in ischemia-reperfusion?. Basic Research in Cardiology. 88(5). 379–379. 2 indexed citations
18.
Millar, B. Cherie, H. M. Piper, Michael Weber, et al.. (1991). Positive and negative contractile effects of neuropeptide Y on ventricular cardiomyocytes. American Journal of Physiology-Heart and Circulatory Physiology. 261(6). H1727–H1733. 46 indexed citations
19.
Hütter, Joachim, H. M. Piper, & P. G. Spieckermann. (1985). An index for estimation of oxygen consumption in rat heart by hemodynamic parameters. American Journal of Physiology-Heart and Circulatory Physiology. 249(4). H729–H734. 20 indexed citations
20.
Schwartz, Peter J., H. M. Piper, & P. G. Spieckermann. (1983). Anoxia of cultured adult cardiac myocytes: Morphological changes☆. Journal of Molecular and Cellular Cardiology. 15. 2–2. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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