David R. Kaplan

2.8k total citations · 1 hit paper
24 papers, 2.5k citations indexed

About

David R. Kaplan is a scholar working on Molecular Biology, Oncology and Genetics. According to data from OpenAlex, David R. Kaplan has authored 24 papers receiving a total of 2.5k indexed citations (citations by other indexed papers that have themselves been cited), including 18 papers in Molecular Biology, 4 papers in Oncology and 4 papers in Genetics. Recurrent topics in David R. Kaplan's work include Protein Kinase Regulation and GTPase Signaling (6 papers), Receptor Mechanisms and Signaling (5 papers) and PI3K/AKT/mTOR signaling in cancer (4 papers). David R. Kaplan is often cited by papers focused on Protein Kinase Regulation and GTPase Signaling (6 papers), Receptor Mechanisms and Signaling (5 papers) and PI3K/AKT/mTOR signaling in cancer (4 papers). David R. Kaplan collaborates with scholars based in United States, Canada and Poland. David R. Kaplan's co-authors include Deborah K. Morrison, Lewis T. Williams, Thomas M. Roberts, Jaime A. Escobedo, Frank McCormick, Gail A. Wong, U R Rapp, Ulf R. Rapp, W. Michael Kavanaugh and Christoph W. Turck and has published in prestigious journals such as Cell, Proceedings of the National Academy of Sciences and The Journal of Experimental Medicine.

In The Last Decade

David R. Kaplan

24 papers receiving 2.4k citations

Hit Papers

PDGF β-receptor stimulates tyrosine phosphorylation of GA... 1990 2026 2002 2014 1990 100 200 300 400 500

Peers

David R. Kaplan
Ingrid Verlaan Netherlands
Liza Zokas United States
Barbara Marte United States
Steven Swendeman United States
Michael D. Waterfield United Kingdom
Joseph P. Gardner United States
Julie L. Wilsbacher United States
Nancy Olashaw United States
David R. Kaplan
Citations per year, relative to David R. Kaplan David R. Kaplan (= 1×) peers Maria Rozakis-Adcock

Countries citing papers authored by David R. Kaplan

Since Specialization
Citations

This map shows the geographic impact of David R. Kaplan's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by David R. Kaplan with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites David R. Kaplan more than expected).

Fields of papers citing papers by David R. Kaplan

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by David R. Kaplan. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by David R. Kaplan. The network helps show where David R. Kaplan may publish in the future.

Co-authorship network of co-authors of David R. Kaplan

This figure shows the co-authorship network connecting the top 25 collaborators of David R. Kaplan. A scholar is included among the top collaborators of David R. Kaplan based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with David R. Kaplan. David R. Kaplan is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
2.
Gallagher, Denis, Guang Yang, Andrée Gauthier-Fisher, et al.. (2013). Transient Maternal IL-6 Mediates Long-Lasting Changes in Neural Stem Cell Pools by Deregulating an Endogenous Self-Renewal Pathway. Cell stem cell. 13(5). 564–576. 73 indexed citations
3.
Quevedo, Celia, David R. Kaplan, & W. Brent Derry. (2007). AKT-1 Regulates DNA-Damage-Induced Germline Apoptosis in C. elegans. Current Biology. 17(3). 286–292. 58 indexed citations
4.
Kaplan, David R.. (2003). Enzymatic Amplification Staining for Cell Surface Antigens. Current Protocols in Cytometry. 23(1). Unit 6.14–Unit 6.14. 11 indexed citations
5.
Wenzel, Jennifer M., Ralf Sanzenbacher, Marc Lewitzky, et al.. (2001). Multiple interactions of the cytosolic polyproline region of the CD95 ligand: hints for the reverse signal transduction capacity of a death factor1. FEBS Letters. 509(2). 255–262. 46 indexed citations
6.
Lucarelli, Enrico, David R. Kaplan, & Carol J. Thiele. (1997). Activation of trk-A but not trk-B signal transduction pathway inhibits growth of Neuroblastoma cells. European Journal of Cancer. 33(12). 2068–2070. 40 indexed citations
7.
Stephens, Robert M., et al.. (1992). 95-kilodalton B-Raf serine/threonine kinase: identification of the protein and its major autophosphorylation site.. Molecular and Cellular Biology. 12(9). 3733–3742. 37 indexed citations
8.
Escobedo, Jaime A., David R. Kaplan, W. Michael Kavanaugh, Christoph W. Turck, & Lewis T. Williams. (1991). A Phosphatidylinositol-3 Kinase Binds to Platelet-Derived Growth Factor Receptors through a Specific Receptor Sequence Containing Phosphotyrosine. Molecular and Cellular Biology. 11(2). 1125–1132. 257 indexed citations
9.
Escobedo, J A, David R. Kaplan, W. Michael Kavanaugh, Christoph W. Turck, & L T Williams. (1991). A phosphatidylinositol-3 kinase binds to platelet-derived growth factor receptors through a specific receptor sequence containing phosphotyrosine.. Molecular and Cellular Biology. 11(2). 1125–1132. 110 indexed citations
10.
Kaplan, David R., Deborah K. Morrison, Gail A. Wong, Frank McCormick, & Lewis T. Williams. (1990). PDGF β-receptor stimulates tyrosine phosphorylation of GAP and association of GAP with a signaling complex. Cell. 61(1). 125–133. 503 indexed citations breakdown →
11.
Morrison, Deborah K., David R. Kaplan, Sue Goo Rhee, & Lewis T. Williams. (1990). Platelet-Derived Growth Factor (PDGF)-Dependent Association of Phospholipase C-γ with the PDGF Receptor Signaling Complex. Molecular and Cellular Biology. 10(5). 2359–2366. 187 indexed citations
12.
Jin, Yi, David R. Kaplan, Morris F. White, et al.. (1990). Stimulation via CD3-Ti but not CD2 induces rapid tyrosine phosphorylation of a 68-kDa protein in the human Jurkat T cell line.. The Journal of Immunology. 144(2). 647–652. 28 indexed citations
13.
Hall, David J., Susan Dana Jones, David R. Kaplan, et al.. (1989). Evidence for a Novel Signal Transduction Pathway Activated by Platelet-Derived Growth Factor and by Double-Stranded RNA. Molecular and Cellular Biology. 9(4). 1705–1713. 3 indexed citations
14.
Kaplan, David R., et al.. (1989). Mechanisms of transformation by polyoma virus middle T antigen. Biochimica et Biophysica Acta (BBA) - Reviews on Cancer. 948(3). 345–364. 50 indexed citations
15.
Morrison, Deborah K., David R. Kaplan, Jaime A. Escobedo, et al.. (1989). Direct activation of the serine/threonine kinase activity of raf-1 through tyrosine phosphorylation by the PDGF β-receptor. Cell. 58(4). 649–657. 473 indexed citations
16.
Hall, David J., Susan Dana Jones, David R. Kaplan, et al.. (1989). Evidence for a novel signal transduction pathway activated by platelet-derived growth factor and by double-stranded RNA.. Molecular and Cellular Biology. 9(4). 1705–1713. 29 indexed citations
17.
Whitman, Malcolm, et al.. (1986). Phosphoinositide kinase activity and transformation.. PubMed. 45(11). 2647–52. 4 indexed citations
18.
Kaplan, David R., B J Bockus, Thomas M. Roberts, et al.. (1985). Large-Scale Production of Polyoma Middle T Antigen by Using Genetically Engineered Tumors. Molecular and Cellular Biology. 5(7). 1795–1799. 6 indexed citations
19.
Franklin, Wilbur A., Stefano Mariotti, David R. Kaplan, & Leslie J. DeGroot. (1982). Immunofluorescence localization of thyroglobulin in metastatic thyroid cancer. Cancer. 50(5). 939–945. 16 indexed citations
20.
Kaplan, David R. & J Quintáns. (1978). Alteration of clonal profile. I. Effect of sublethal irradiation on the responses to phosphorylcholine in BALB/c mice.. The Journal of Experimental Medicine. 148(4). 987–995. 9 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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