David Prevette

6.8k total citations · 1 hit paper
61 papers, 5.9k citations indexed

About

David Prevette is a scholar working on Cellular and Molecular Neuroscience, Molecular Biology and Developmental Neuroscience. According to data from OpenAlex, David Prevette has authored 61 papers receiving a total of 5.9k indexed citations (citations by other indexed papers that have themselves been cited), including 38 papers in Cellular and Molecular Neuroscience, 33 papers in Molecular Biology and 28 papers in Developmental Neuroscience. Recurrent topics in David Prevette's work include Nerve injury and regeneration (33 papers), Neurogenesis and neuroplasticity mechanisms (28 papers) and Spinal Cord Injury Research (11 papers). David Prevette is often cited by papers focused on Nerve injury and regeneration (33 papers), Neurogenesis and neuroplasticity mechanisms (28 papers) and Spinal Cord Injury Research (11 papers). David Prevette collaborates with scholars based in United States, Hong Kong and Japan. David Prevette's co-authors include Ronald W. Oppenheim, Lucien J. Houenou, Qin-Wei Yin, Linxi Li, Sharon Vinsant, Yan Qiao, RW Oppenheim, James E. Johnson, Siwei Wang and Albert Lo and has published in prestigious journals such as Nature, Science and Neuron.

In The Last Decade

David Prevette

61 papers receiving 5.7k citations

Hit Papers

align trajectories sort by overperformance

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Developing motor neurons rescued from programmed and axotomy-induced cell death by GDNF

1995 587 citations Ronald W. Oppenheim, Lucien J. Houenou et al. profile →

Author Peers

Peers are selected by citation overlap in the author's most active subfields. citations · hero ref

Author						Papers	Cites
David Prevette	3.4k	2.8k	1.8k	1000	837	61	5.9k
Dies Meijer	2.6k 0.8×	3.1k 1.1×	1.7k 0.9×	703 0.7×	553 0.7×	74	6.7k
Cynthia Wetmore	2.6k 0.8×	2.5k 0.9×	1.6k 0.9×	522 0.5×	1.1k 1.3×	86	5.6k
Patrick M. Wood	4.9k 1.5×	2.1k 0.8×	2.8k 1.6×	636 0.6×	858 1.0×	94	7.0k
Bettina Holtmann	2.0k 0.6×	1.9k 0.7×	1.2k 0.7×	460 0.5×	492 0.6×	44	4.5k
Robert G. Kalb	4.2k 1.2×	4.0k 1.5×	1.0k 0.6×	1.6k 1.6×	466 0.6×	112	8.4k
Paul N. Hoffman	3.1k 0.9×	2.4k 0.9×	1.4k 0.8×	1.2k 1.2×	435 0.5×	53	7.0k
Jeffery L. Twiss	3.0k 0.9×	4.5k 1.6×	1.1k 0.6×	458 0.5×	486 0.6×	98	6.9k
Anne Baron‐Van Evercooren	2.9k 0.9×	2.8k 1.0×	3.9k 2.2×	345 0.3×	675 0.8×	99	7.0k
Mary Hynes	3.3k 1.0×	5.3k 1.9×	1.2k 0.7×	1.4k 1.4×	244 0.3×	45	8.8k
Pantelis Tsoulfas	2.6k 0.8×	2.1k 0.7×	1.6k 0.9×	263 0.3×	512 0.6×	62	4.7k

Countries citing papers authored by David Prevette

Since Specialization

Citations

This map shows the geographic impact of David Prevette's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by David Prevette with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites David Prevette more than expected).

Fields of papers citing papers by David Prevette

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by David Prevette. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by David Prevette. The network helps show where David Prevette may publish in the future.

Co-authorship network of co-authors of David Prevette

This figure shows the co-authorship network connecting the top 25 collaborators of David Prevette. A scholar is included among the top collaborators of David Prevette based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with David Prevette. David Prevette is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Texidó, Laura, Jordi Calderó, Lídia Piedrafita, et al.. (2010). Increased intramuscular nerve branching and inhibition of programmed cell death of chick embryo motoneurons by immunoglobulins from patients with motoneuron disease. Journal of Neuroimmunology. 229(1-2). 157–168. 3 indexed citations

Oppenheim, Ronald W., Jordi Calderó, Josep E. Esquerda, et al.. (2008). The rescue of developing avian motoneurons from programmed cell death by a selective inhibitor of the fetal muscle‐specific nicotinic acetylcholine receptor. Developmental Neurobiology. 68(7). 972–980. 14 indexed citations

Oppenheim, Ronald W., Klas Blomgren, Douglas W. Ethell, et al.. (2008). Developing Postmitotic Mammalian NeuronsIn VivoLacking Apaf-1 Undergo Programmed Cell Death by a Caspase-Independent, Nonapoptotic Pathway Involving Autophagy. Journal of Neuroscience. 28(6). 1490–1497. 28 indexed citations

Taylor, Anna, David J. Gifondorwa, Jason M. Newbern, et al.. (2007). Astrocyte and Muscle-Derived Secreted Factors Differentially Regulate Motoneuron Survival. Journal of Neuroscience. 27(3). 634–644. 23 indexed citations

Gifondorwa, David J., Mac Robinson, Crystal D. Hayes, et al.. (2007). Exogenous Delivery of Heat Shock Protein 70 Increases Lifespan in a Mouse Model of Amyotrophic Lateral Sclerosis. Journal of Neuroscience. 27(48). 13173–13180. 107 indexed citations

Buss, Robert R., Thomas W. Gould, Jianjun Ma, et al.. (2006). Neuromuscular Development in the Absence of Programmed Cell Death: Phenotypic Alteration of Motoneurons and Muscle. Journal of Neuroscience. 26(52). 13413–13427. 41 indexed citations

Oppenheim, Ronald W., Jordi Calderó, Josep E. Esquerda, et al.. (2003). Rescue of developing spinal motoneurons from programmed cell death by the GABAA agonist muscimol acts by blockade of neuromuscular activity and increased intramuscular nerve branching. Molecular and Cellular Neuroscience. 22(3). 331–343. 30 indexed citations

Prevette, David, et al.. (2002). Developmental expression of nicotinic receptors in the chick and human spinal cord. The Journal of Comparative Neurology. 455(1). 86–99. 20 indexed citations

Yuan, Qiuju, Wutian Wu, Kwok‐Fai So, et al.. (2000). Effects of neurotrophic factors on motoneuron survival following axonal injury in newborn rats. Neuroreport. 11(10). 2237–2241. 72 indexed citations

10.

Gould, Thomas W., et al.. (1999). The Spatial–Temporal Gradient of Naturally Occurring Motoneuron Death Reflects the Time of Prior Exit from the Cell Cycle and Position within the Lateral Motor Column. Developmental Biology. 216(2). 611–621. 20 indexed citations

11.

Grieshammer, Uta, Mark Lewandoski, David Prevette, Ronald W. Oppenheim, & Gail R. Martin. (1998). Muscle-Specific Cell Ablation Conditional upon Cre-Mediated DNA Recombination in Transgenic Mice Leads to Massive Spinal and Cranial Motoneuron Loss. Developmental Biology. 197(2). 234–247. 93 indexed citations

12.

Campagna, Jason, David Prevette, Ronald W. Oppenheim, & John L. Bixby. (1997). Target Contact Regulates Expression of Synaptotagmin Genes in Spinal Motor Neuronsin Vivo. Molecular and Cellular Neuroscience. 8(6). 377–388. 21 indexed citations

13.

Houenou, Lucien J., Ronald W. Oppenheim, Linxi Li, Albert Lo, & David Prevette. (1996). Regulation of spinal motoneuron survival by GDNF during development and following injury. Cell and Tissue Research. 286(2). 219–223. 56 indexed citations

14.

Milligan, Carolanne E., David Prevette, Hiroyuki Yaginuma, et al.. (1995). Peptide inhibitors of the ice protease family arrest programmed cell death of motoneurons in vivo and in vitro. Neuron. 15(2). 385–393. 282 indexed citations

15.

Oppenheim, R, et al.. (1993). Biological studies of a putative avian muscle‐derived neurotrophic factor that prevents naturally occurring motoneuron death in vivo. Journal of Neurobiology. 24(8). 1065–1079. 103 indexed citations

16.

Fournier‐Thibault, Claire, David Prevette, Ronald W. Oppenheim, & Josiane Fontaine-Pérus. (1993). Interactions between spinal cord stimulation and activity blockade in the regulation of synaptogenesis and motoneuron survival in the chick embryo. Journal of Neurobiology. 24(9). 1142–1156. 15 indexed citations

17.

Oppenheim, Ronald W., David Prevette, Michael Tytell, & Shunsaku Homma. (1990). Naturally occurring and induced neuronal death in the chick embryo in vivo requires protein and RNA synthesis: Evidence for the role of cell death genes. Developmental Biology. 138(1). 104–113. 296 indexed citations

18.

McManaman, James L., R Oppenheim, David Prevette, & Philippe Marchetti. (1990). Rescue of motoneurons from cell death by a purified skeletal muscle polypeptide: Effects of the ChAT development factor, CDF. Neuron. 4(6). 891–898. 68 indexed citations

19.

Oppenheim, Ronald W., Toby B. Cole, & David Prevette. (1989). Early regional variations in motoneuron numbers arise by differential proliferation in the chick embryo spinal cord. Developmental Biology. 133(2). 468–474. 76 indexed citations

20.

Maderdrut, Jerome L., Ronald W. Oppenheim, & David Prevette. (1988). Enhancement of naturally occurring cell death in the sympathetic and parasympathetic ganglia of the chicken embryo following blockade of ganglionic transmission. Brain Research. 444(1). 189–194. 69 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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