David G. Cook

5.7k total citations · 1 hit paper
78 papers, 4.6k citations indexed

About

David G. Cook is a scholar working on Cellular and Molecular Neuroscience, Epidemiology and Molecular Biology. According to data from OpenAlex, David G. Cook has authored 78 papers receiving a total of 4.6k indexed citations (citations by other indexed papers that have themselves been cited), including 27 papers in Cellular and Molecular Neuroscience, 21 papers in Epidemiology and 20 papers in Molecular Biology. Recurrent topics in David G. Cook's work include Alzheimer's disease research and treatments (20 papers), Traumatic Brain Injury Research (18 papers) and Traumatic Brain Injury and Neurovascular Disturbances (16 papers). David G. Cook is often cited by papers focused on Alzheimer's disease research and treatments (20 papers), Traumatic Brain Injury Research (18 papers) and Traumatic Brain Injury and Neurovascular Disturbances (16 papers). David G. Cook collaborates with scholars based in United States, Australia and United Kingdom. David G. Cook's co-authors include Robert W. Doms, James S. Meabon, Francisco González‐Scarano, Suzanne Craft, James B. Leverenz, J. Jacob Kulstad, Jane Sullivan, G. Stennis Watson, Steven L. Spitalnik and Elaine R. Peskind and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of the American Chemical Society and Journal of Biological Chemistry.

In The Last Decade

David G. Cook

75 papers receiving 4.5k citations

Hit Papers

Lipopolysaccharide-induced blood-brain barrier disruption... 2015 2026 2018 2022 2015 100 200 300 400

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
David G. Cook United States 33 2.0k 1.4k 956 758 603 78 4.6k
Inna I. Kruman United States 28 1.1k 0.5× 2.3k 1.6× 746 0.8× 496 0.7× 197 0.3× 38 4.9k
Manuel Buttini United States 33 2.3k 1.2× 1.9k 1.3× 1.1k 1.1× 1.5k 1.9× 340 0.6× 59 5.4k
Jacques Hugon France 47 2.6k 1.3× 3.0k 2.1× 1.7k 1.8× 1.2k 1.5× 453 0.8× 213 7.8k
Bruno Conti United States 43 1.3k 0.6× 2.3k 1.6× 1.4k 1.5× 1.1k 1.4× 453 0.8× 95 6.4k
Juan Orellana Chile 42 1.1k 0.5× 3.1k 2.1× 918 1.0× 687 0.9× 417 0.7× 153 5.6k
Steven J. Henriksen United States 47 1.1k 0.5× 2.0k 1.4× 3.1k 3.3× 851 1.1× 603 1.0× 119 7.7k
Michelle A. Erickson United States 32 1.5k 0.7× 1.4k 1.0× 552 0.6× 2.1k 2.7× 259 0.4× 67 5.4k
Carlos R. Plata‐Saláman United States 47 1.6k 0.8× 1.2k 0.8× 1.3k 1.4× 1.0k 1.4× 739 1.2× 142 6.7k
Douglas E. Brenneman United States 54 1.6k 0.8× 4.3k 2.9× 5.4k 5.7× 873 1.2× 363 0.6× 192 9.9k
Rosemarie M. Booze United States 42 1.1k 0.6× 2.1k 1.4× 1.9k 1.9× 1.3k 1.8× 267 0.4× 177 6.2k

Countries citing papers authored by David G. Cook

Since Specialization
Citations

This map shows the geographic impact of David G. Cook's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by David G. Cook with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites David G. Cook more than expected).

Fields of papers citing papers by David G. Cook

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by David G. Cook. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by David G. Cook. The network helps show where David G. Cook may publish in the future.

Co-authorship network of co-authors of David G. Cook

This figure shows the co-authorship network connecting the top 25 collaborators of David G. Cook. A scholar is included among the top collaborators of David G. Cook based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with David G. Cook. David G. Cook is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Braun, Molly, Marie Xun Wang, Emily D. Klein, et al.. (2024). Macroscopic changes in aquaporin-4 underlie blast traumatic brain injury-related impairment in glymphatic function. Brain. 147(6). 2214–2229. 20 indexed citations
2.
Burke, Jacqueline A., Ira Joshi, Sharon R. Roberts, et al.. (2024). Phase-changing citrate macromolecule combats oxidative pancreatic islet damage, enables islet engraftment and function in the omentum. Science Advances. 10(23). eadk3081–eadk3081. 3 indexed citations
3.
Gasperi, Rita De, Miguel A. Gama Sosa, Gissel M. Perez, et al.. (2023). Metabotropic Glutamate Receptor 2 Expression Is Chronically Elevated in Male Rats With Post-Traumatic Stress Disorder Related Behavioral Traits Following Repetitive Low-Level Blast Exposure. Journal of Neurotrauma. 41(5-6). 714–733. 3 indexed citations
4.
Schindler, Abigail G., Britahny M. Baskin, Barbara Juarez, et al.. (2021). Repetitive blast mild traumatic brain injury increases ethanol sensitivity in male mice and risky drinking behavior in male combat veterans. Alcoholism Clinical and Experimental Research. 45(5). 1051–1064. 17 indexed citations
5.
Alonge, Kimberly M., et al.. (2021). Changes in Brain Matrix Glycan Sulfation Associate With Reactive Gliosis and Motor Coordination in Mice With Head Trauma. Frontiers in Behavioral Neuroscience. 15. 745288–745288. 10 indexed citations
6.
Gasperi, Rita De, Miguel A. Gama Sosa, Gissel M. Perez, et al.. (2021). Laterality and region-specific tau phosphorylation correlate with PTSD-related behavioral traits in rats exposed to repetitive low-level blast. Acta Neuropathologica Communications. 9(1). 33–33. 11 indexed citations
7.
Pagulayan, Kathleen F., Eric C. Petrie, David G. Cook, et al.. (2018). Effect of blast-related mTBI on the working memory system: a resting state fMRI study. Brain Imaging and Behavior. 14(4). 949–960. 11 indexed citations
8.
Rau, Holly, Rebecca C. Hendrickson, Hannah Roggenkamp, et al.. (2017). Fatigue – but not mTBI history, PTSD, or sleep quality – directly contributes to reduced prospective memory performance in Iraq and Afghanistan era Veterans. The Clinical Neuropsychologist. 32(7). 1319–1336. 14 indexed citations
9.
Costa, Maria do Carmo, Svetlana Fischer, Thomas J. McQuade, et al.. (2016). Unbiased screen identifies aripiprazole as a modulator of abundance of the polyglutamine disease protein, ataxin-3. Brain. 139(11). 2891–2908. 33 indexed citations
10.
Pratt, Kara G., Ping Zhu, Hirofumi Watari, David G. Cook, & Jane Sullivan. (2011). A Novel Role for γ-Secretase: Selective Regulation of Spontaneous Neurotransmitter Release from Hippocampal Neurons. Journal of Neuroscience. 31(3). 899–906. 25 indexed citations
11.
Mookherjee, Paramita, Pattie S. Green, G. Stennis Watson, et al.. (2011). GLT-1 Loss Accelerates Cognitive Deficit Onset in an Alzheimer's Disease Animal Model. Journal of Alzheimer s Disease. 26(3). 447–455. 127 indexed citations
12.
Lee, Aven, Ashley Anderson, Philip Poronnik, et al.. (2011). Localisation of novel forms of glutamate transporters and the cystine-glutamate antiporter in the choroid plexus: Implications for CSF glutamate homeostasis. Journal of Chemical Neuroanatomy. 43(1). 64–75. 10 indexed citations
13.
Peskind, Elaine R., Eric C. Petrie, Donna J. Cross, et al.. (2010). Cerebrocerebellar hypometabolism associated with repetitive blast exposure mild traumatic brain injury in 12 Iraq war Veterans with persistent post-concussive symptoms. NeuroImage. 54. S76–S82. 207 indexed citations
14.
Fishel, Mark A., G. Stennis Watson, Thomas J. Montine, et al.. (2005). Hyperinsulinemia Provokes Synchronous Increases in Central Inflammation and β-Amyloid in Normal Adults. Archives of Neurology. 62(10). 1539–44. 179 indexed citations
15.
Kulstad, J. Jacob, Pamela J. McMillan, James B. Leverenz, et al.. (2005). Effects of Chronic Glucocorticoid Administration on Insulin-Degrading Enzyme and Amyloid-Beta Peptide in the Aged Macaque. Journal of Neuropathology & Experimental Neurology. 64(2). 139–146. 77 indexed citations
16.
Craft, Suzanne, Sanjay Asthana, David G. Cook, et al.. (2003). Insulin dose–response effects on memory and plasma amyloid precursor protein in Alzheimer’s disease: interactions with apolipoprotein E genotype. Psychoneuroendocrinology. 28(6). 809–822. 209 indexed citations
17.
Linford, Nancy J., Yaxiong Yang, David G. Cook, & Daniel M. Dorsa. (2001). Neuronal Apoptosis Resulting from High Doses of the Isoflavone Genistein: Role for Calcium and P42/44 Mitogen-Activated Protein Kinase. Journal of Pharmacology and Experimental Therapeutics. 299(1). 67–75. 69 indexed citations
18.
Forman, Mark S., David G. Cook, Susan Leight, Robert W. Doms, & Virginia M.‐Y. Lee. (1997). Differential Effects of the Swedish Mutant Amyloid Precursor Protein on β-Amyloid Accumulation and Secretion in Neurons and Nonneuronal Cells. Journal of Biological Chemistry. 272(51). 32247–32253. 47 indexed citations
19.
Cook, David G., Raymond Scott Turner, Dennis L. Kolson, V.M.-Y. Lee, & Robert W. Doms. (1996). Vaccinia virus serves as an efficient vector for expressing heterologous proteins in human NTera 2 neurons. The Journal of Comparative Neurology. 374(4). 481–492. 7 indexed citations
20.
Nathanson, Neal, David G. Cook, Dennis L. Kolson, & Francisco González‐Scarano. (1994). Pathogenesis of HIV Encephalopathya. Annals of the New York Academy of Sciences. 724(1). 87–106. 17 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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