David C. Lyden
Impact in
- Molecular Biology top 0.02%
- Extracellular vesicles in disease
- Angiogenesis and VEGF in Cancer
- Oncology top 0.2%
- Cancer Cells and Metastasis
Papers in
-
- Extracellular vesicles in disease 39
- Angiogenesis and VEGF in Cancer 19
- PI3K/AKT/mTOR signaling in cancer 3
- Oncology 26
- Cancer Cells and Metastasis 17
- Co-authors
- Shahin Rafii (25 shared papers)Bethan Psaila (8 shared papers)Héctor Peinado (9 shared papers)Rosandra Natasha Kaplan (8 shared papers)Beate Heissig (8 shared papers)Haiying Zhang (12 shared papers)Robert Benezra (6 shared papers)Koichi Hattori (7 shared papers)
- Journals
- Cancer Cell (11 papers)Nature (10 papers)Nature Medicine (8 papers)Nature Cell Biology (7 papers)Nature Communications (7 papers)
- Partner nations
- United StatesPortugalJapan
In The Last Decade
David C. Lyden
162 papers receiving 44.0k citations
David C. Lyden's Hit Papers
Peers
Comparison fields: 5 of 125
- Molecular Biology 18.9k
- Oncology 5.0k
- Genetics 1.7k
- Cancer Research 1.7k
- Hematology 1.4k
Countries citing papers authored by David C. Lyden
This map shows the geographic impact of David C. Lyden's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by David C. Lyden with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites David C. Lyden more than expected).
Fields of papers citing papers by David C. Lyden
This network shows the impact of papers produced by David C. Lyden. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by David C. Lyden. The network helps show where David C. Lyden may publish in the future.
Co-authors
The 25 scholars most cited alongside David C. Lyden, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
Showing the 20 most-cited of 167 papers — load more, or switch the sort, to bring in the rest.
| # | Work | ||
|---|---|---|---|
| 1 | Tumour exosome integrins determine organotropic metastasis Hit paper breakdown → | 2015 | 4267 |
| 2 | Melanoma exosomes educate bone marrow progenitor cells toward a pro-metastatic phenotype through MET Hit paper breakdown → | 2012 | 3182 |
| 3 | VEGFR1-positive haematopoietic bone marrow progenitors initiate the pre-metastatic niche Hit paper breakdown → | 2005 | 2693 |
| 4 | Pancreatic cancer exosomes initiate pre-metastatic niche formation in the liver Hit paper breakdown → | 2015 | 2282 |
| 5 | Extracellular Vesicles in Cancer: Cell-to-Cell Mediators of Metastasis Hit paper breakdown → | 2016 | 1652 |
| 6 | Impaired recruitment of bone-marrow–derived endothelial and hematopoietic precursor cells blocks tumor angiogenesis and growth Hit paper breakdown → | 2001 | 1592 |
| 7 | Pre-metastatic niches: organ-specific homes for metastases Hit paper breakdown → | 2017 | 1529 |
| 8 | Recruitment of Stem and Progenitor Cells from the Bone Marrow Niche Requires MMP-9 Mediated Release of Kit-Ligand Hit paper breakdown → | 2002 | 1481 |
| 9 | Double-stranded DNA in exosomes: a novel biomarker in cancer detection Hit paper breakdown → | 2014 | 1410 |
| 10 | Identification of distinct nanoparticles and subsets of extracellular vesicles by asymmetric flow field-flow fractionation Hit paper breakdown → | 2018 | 1400 |
| 11 | Therapeutic stem and progenitor cell transplantation for organ vascularization and regeneration Hit paper breakdown → | 2003 | 1361 |
| 12 | Tumor Response to Radiotherapy Regulated by Endothelial Cell Apoptosis Hit paper breakdown → | 2003 | 1346 |
| 13 | The metastatic niche: adapting the foreign soil Hit paper breakdown → | 2009 | 1067 |
| 14 | Exosome-Mediated Metastasis: Communication from a Distance Hit paper breakdown → | 2019 | 1039 |
| 15 | Extracellular Vesicle and Particle Biomarkers Define Multiple Human Cancers Hit paper breakdown → | 2020 | 1008 |
| 16 | The perivascular niche regulates breast tumour dormancy Hit paper breakdown → | 2013 | 931 |
| 17 | 1999 | 774 | |
| 18 | CD133 expression is not restricted to stem cells, and both CD133+ and CD133– metastatic colon cancer cells initiate tumors Hit paper breakdown → | 2008 | 771 |
| 19 | Inductive angiocrine signals from sinusoidal endothelium are required for liver regeneration Hit paper breakdown → | 2010 | 686 |
| 20 | Chemokine-mediated interaction of hematopoietic progenitors with the bone marrow vascular niche is required for thrombopoiesis Hit paper breakdown → | 2003 | 642 |
About David C. Lyden
David C. Lyden is a scholar working on Molecular Biology, Oncology, Genetics, Hematology and Cardiology and Cardiovascular Medicine, having authored 167 papers that have together received 44.9k indexed citations. Recurring topics across this work include Extracellular vesicles in disease (39 papers), Angiogenesis and VEGF in Cancer (19 papers), Cancer Cells and Metastasis (17 papers), Glioma Diagnosis and Treatment (12 papers), Viral Infections and Immunology Research (6 papers), Spaceflight effects on biology (5 papers), Hematopoietic Stem Cell Transplantation (4 papers) and PI3K/AKT/mTOR signaling in cancer (3 papers). The work is most often cited by research in Molecular Biology (18.9k citations), Oncology (5.0k citations), Genetics (1.7k citations), Cancer Research (1.7k citations) and Hematology (1.4k citations). David C. Lyden has collaborated with scholars based in United States, Portugal and Japan. Frequent co-authors include Shahin Rafii, Bethan Psaila, Héctor Peinado, Rosandra Natasha Kaplan, Beate Heissig, Haiying Zhang, Robert Benezra, Koichi Hattori, Sérgio Dias and Candia M. Kenific. Their work appears in journals such as Cancer Cell, Nature, Nature Medicine, Nature Cell Biology and Nature Communications.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.