David B. Beck

6.7k total citations · 2 hit papers
62 papers, 2.5k citations indexed

About

David B. Beck is a scholar working on Rheumatology, Molecular Biology and Immunology. According to data from OpenAlex, David B. Beck has authored 62 papers receiving a total of 2.5k indexed citations (citations by other indexed papers that have themselves been cited), including 36 papers in Rheumatology, 25 papers in Molecular Biology and 15 papers in Immunology. Recurrent topics in David B. Beck's work include Otitis Media and Relapsing Polychondritis (35 papers), Vascular Anomalies and Treatments (12 papers) and Immunodeficiency and Autoimmune Disorders (12 papers). David B. Beck is often cited by papers focused on Otitis Media and Relapsing Polychondritis (35 papers), Vascular Anomalies and Treatments (12 papers) and Immunodeficiency and Autoimmune Disorders (12 papers). David B. Beck collaborates with scholars based in United States, France and Netherlands. David B. Beck's co-authors include Danny Reinberg, Hisanobu Oda, Steven S. Shen, William J. Drury, Philipp Voigt, Daniel L. Kastner, Achim Werner, Benjamin A. García, Barry M. Zee and Jinsook Son and has published in prestigious journals such as Science, Cell and The Lancet.

In The Last Decade

David B. Beck

56 papers receiving 2.5k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Benign and malignant hematologic manifestations in patients with VEXAS syndrome due to somatic mutations in UBA1

2021 139 citations Bhavisha A. Patel, Emma M. Groarke et al. Blood Advances profile →
Estimated Prevalence and Clinical Manifestations of UBA1 Variants Associated With VEXAS Syndrome in a Clinical Population

2023 113 citations David B. Beck, Jung Kim et al. profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
David B. Beck United States	22	1.4k	748	533	278	234	62	2.5k
Alan W. Lau United States	19	1.5k 1.1×	333 0.4×	57 0.1×	638 2.3×	93 0.4×	20	2.1k
Genta Nagae Japan	24	2.6k 1.9×	64 0.1×	225 0.4×	468 1.7×	386 1.6×	48	3.4k
Richard N. Freiman United States	24	1.2k 0.9×	79 0.1×	107 0.2×	303 1.1×	483 2.1×	34	2.1k
Elizabeth A. Eklund United States	33	1.6k 1.2×	94 0.1×	222 0.4×	574 2.1×	158 0.7×	90	2.7k
Masaru Niki Japan	21	2.2k 1.6×	58 0.1×	189 0.4×	840 3.0×	156 0.7×	27	3.4k
Gustavo Pacheco–Rodriguez United States	26	895 0.7×	201 0.3×	74 0.1×	446 1.6×	88 0.4×	64	2.0k
Wendy Dubois United States	21	1.1k 0.8×	29 0.0×	117 0.2×	328 1.2×	99 0.4×	35	1.6k
Nianxiang Zhang United States	25	1.6k 1.2×	32 0.0×	121 0.2×	315 1.1×	176 0.8×	47	2.1k
Jonathan Grim United States	8	1.7k 1.3×	41 0.1×	159 0.3×	766 2.8×	160 0.7×	9	2.1k
Javeed Iqbal United States	28	961 0.7×	58 0.1×	493 0.9×	970 3.5×	74 0.3×	115	2.8k

Countries citing papers authored by David B. Beck

Since Specialization

Citations

This map shows the geographic impact of David B. Beck's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by David B. Beck with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites David B. Beck more than expected).

Fields of papers citing papers by David B. Beck

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by David B. Beck. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by David B. Beck. The network helps show where David B. Beck may publish in the future.

Co-authorship network of co-authors of David B. Beck

This figure shows the co-authorship network connecting the top 25 collaborators of David B. Beck. A scholar is included among the top collaborators of David B. Beck based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with David B. Beck. David B. Beck is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Weeks, Lachelle D., Danielle Hammond, Sinisa Savic, et al.. (2025). Establishing a consensus definition of VEXAS flare for clinical research. Lara D. Veeken. 65(2).

Sakuma, Maki, Manja Meggendorfer, Wolfgang Kern, et al.. (2025). Distinct characteristics of VEXAS-causative UBA1 M41 and recurrent functional non-M41 mutations. Leukemia. 39(12). 2872–2880.

Richardson, Ashley, Jung Kim, Rebecca I. Torene, et al.. (2025). Clinical Manifestations of VEXAS Syndrome Across a Broad Spectrum of UBA1 Mutation Burden. Arthritis & Rheumatology. 78(1). 223–230. 1 indexed citations

Kuchitsu, Yoshihiko, Christopher S. Law, Brett M. Elicker, et al.. (2025). The common HAQ STING allele prevents clinical penetrance of COPA syndrome. The Journal of Experimental Medicine. 222(4). 4 indexed citations

Bourguiba, R., Valentin Lacombe, David B. Beck, et al.. (2025). Characterizing VEXAS syndrome in women: Findings from an international multicenter study. Journal of Internal Medicine. 298(5). 516–524.

Chen, Simon, et al.. (2025). Rapid clinical deployment of UBA1 testing in patients with VEXAS syndrome. American Journal of Clinical Pathology. 164(3). 360–366.

Bisikirska, Brygida, Rossella Labella, Álvaro Cuesta‐Domínguez, et al.. (2024). Melatonin receptor 1A variants as genetic cause of idiopathic osteoporosis. Science Translational Medicine. 16(769). eadj0085–eadj0085. 3 indexed citations

Cardona, Daniela Ospina, Ignasi Rodríguez‐Pintó, Núria Bonet, et al.. (2024). Description of a novel splice site variant in UBA1 gene causing VEXAS syndrome. Lara D. Veeken. 63(10). 2897–2902. 2 indexed citations

Torreggiani, Sofia, et al.. (2024). Somatic mutations in autoinflammatory and autoimmune disease. Nature Reviews Rheumatology. 20(11). 683–698. 8 indexed citations

10.

Stibůrková, Blanka, Kateřina Pavelcová, Monika Beličková, et al.. (2023). Novel Somatic UBA1 Variant in a Patient With VEXAS Syndrome. Arthritis & Rheumatology. 75(7). 1285–1290. 33 indexed citations

11.

Asmar, Anthony J., Shaun R. Abrams, Jason C. Collins, et al.. (2023). A ubiquitin-based effector-to-inhibitor switch coordinates early brain, craniofacial, and skin development. Nature Communications. 14(1). 4499–4499. 2 indexed citations

12.

Ding, Yanna, Alina Dulau‐Florea, Emma M. Groarke, et al.. (2023). Use of flow cytometric light scattering to recognize the characteristic vacuolated marrow cells in VEXAS syndrome. Blood Advances. 7(20). 6151–6155. 4 indexed citations

13.

Cowen, Edward W., et al.. (2023). VEXAS Syndrome—Diagnostic Clues for the Dermatologist and Gaps in Our Current Understanding: A Narrative Review. SHILAP Revista de lepidopterología. 4(1). 100242–100242. 5 indexed citations

14.

Made, Caspar I. van der, Judith Potjewijd, Huub P J Willems, et al.. (2021). Adult-onset autoinflammation caused by somatic mutations in UBA1: A Dutch case series of patients with VEXAS. Journal of Allergy and Clinical Immunology. 149(1). 432–439.e4. 128 indexed citations

15.

Asmar, Anthony J., David B. Beck, & Achim Werner. (2020). Control of craniofacial and brain development by Cullin3-RING ubiquitin ligases: Lessons from human disease genetics. Experimental Cell Research. 396(2). 112300–112300. 8 indexed citations

16.

Beck, David B. & Ivona Aksentijevich. (2019). Biochemistry of Autoinflammatory Diseases: Catalyzing Monogenic Disease. Frontiers in Immunology. 10. 101–101. 17 indexed citations

17.

Beck, David B., Megan T. Cho, Francisca Millan, et al.. (2016). A recurrent de novo CTBP1 mutation is associated with developmental delay, hypotonia, ataxia, and tooth enamel defects. Neurogenetics. 17(3). 173–178. 25 indexed citations

18.

Serrano, Lourdes, Paloma Martínez‐Redondo, Anna Marazuela-Duque, et al.. (2013). The tumor suppressor SirT2 regulates cell cycle progression and genome stability by modulating the mitotic deposition of H4K20 methylation. Genes & Development. 27(6). 639–653. 224 indexed citations

19.

Oda, Hisanobu, Michael R. Hübner, David B. Beck, et al.. (2010). Regulation of the Histone H4 Monomethylase PR-Set7 by CRL4Cdt2-Mediated PCNA-Dependent Degradation during DNA Damage. Molecular Cell. 40(3). 364–376. 200 indexed citations

20.

Beck, David B., Roberto Bonasio, Syuzo Kaneko, et al.. (2010). Chromatin in the Nuclear Landscape. Cold Spring Harbor Symposia on Quantitative Biology. 75(0). 11–22. 21 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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