Marcus Krüger

15.3k total citations · 4 hit papers
189 papers, 10.7k citations indexed

About

Marcus Krüger is a scholar working on Molecular Biology, Cell Biology and Physiology. According to data from OpenAlex, Marcus Krüger has authored 189 papers receiving a total of 10.7k indexed citations (citations by other indexed papers that have themselves been cited), including 133 papers in Molecular Biology, 40 papers in Cell Biology and 20 papers in Physiology. Recurrent topics in Marcus Krüger's work include Muscle Physiology and Disorders (21 papers), Mitochondrial Function and Pathology (19 papers) and Ubiquitin and proteasome pathways (13 papers). Marcus Krüger is often cited by papers focused on Muscle Physiology and Disorders (21 papers), Mitochondrial Function and Pathology (19 papers) and Ubiquitin and proteasome pathways (13 papers). Marcus Krüger collaborates with scholars based in Germany, United States and Italy. Marcus Krüger's co-authors include Thomas Braun, Hendrik Nolte, Soraya Hölper, Matthias Mann, Thomas Boettger, Didier Y. R. Stainier, Zacharias Kontarakis, Andrea Rossi, Claudia Gerri and Sara Zanivan and has published in prestigious journals such as Nature, Science and Cell.

In The Last Decade

Marcus Krüger

183 papers receiving 10.6k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Genetic compensation induced by deleterious mutations but not gene knockdowns

2015 890 citations Hendrik Nolte, Soraya Hölper et al. profile →
The Failing Heart Relies on Ketone Bodies as a Fuel

2016 549 citations Grégory Aubert, Ola J. Martin et al. Circulation profile →
Acquisition of the contractile phenotype by murine arterial smooth muscle cells depends on the Mir143/145 gene cluster

2009 528 citations Marcus Krüger, Thomas Braun et al. Journal of Clinical Investigation profile →
SILAC Mouse for Quantitative Proteomics Uncovers Kindlin-3 as an Essential Factor for Red Blood Cell Function

2008 514 citations Marcus Krüger, Markus Moser et al. profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Marcus Krüger Germany	53	7.2k	1.6k	1.6k	1.4k	1.0k	189	10.7k
Timothy Haystead United States	58	8.5k 1.2×	1.9k 1.2×	779 0.5×	979 0.7×	397 0.4×	170	11.7k
Richard B. Pearson Australia	61	9.6k 1.3×	1.5k 0.9×	1.3k 0.8×	922 0.7×	425 0.4×	136	12.4k
Stine F. Pedersen Denmark	54	6.1k 0.8×	1.3k 0.8×	1.3k 0.8×	1.2k 0.8×	278 0.3×	187	9.6k
Michael J.O. Wakelam United Kingdom	64	10.5k 1.4×	2.5k 1.6×	1.6k 1.0×	2.0k 1.5×	863 0.9×	241	15.0k
David L. Brautigan United States	70	12.4k 1.7×	3.6k 2.2×	953 0.6×	1.3k 1.0×	407 0.4×	249	16.5k
Ming Zhou China	62	9.7k 1.3×	1.0k 0.6×	2.3k 1.4×	492 0.4×	818 0.8×	346	14.1k
Stefan Krauß Norway	53	9.6k 1.3×	1.8k 1.1×	1.0k 0.6×	2.8k 2.0×	908 0.9×	163	13.6k
Pingsheng Liu China	47	5.0k 0.7×	2.8k 1.7×	605 0.4×	2.0k 1.5×	816 0.8×	129	8.3k
Gunnar Dittmar Germany	38	8.7k 1.2×	1.2k 0.7×	2.5k 1.6×	442 0.3×	922 0.9×	128	11.3k
Nick Morrice United Kingdom	65	10.5k 1.5×	1.8k 1.1×	1.0k 0.7×	902 0.7×	538 0.5×	113	13.1k

Countries citing papers authored by Marcus Krüger

Since Specialization

Citations

This map shows the geographic impact of Marcus Krüger's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Marcus Krüger with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Marcus Krüger more than expected).

Fields of papers citing papers by Marcus Krüger

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Marcus Krüger. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Marcus Krüger. The network helps show where Marcus Krüger may publish in the future.

Co-authorship network of co-authors of Marcus Krüger

This figure shows the co-authorship network connecting the top 25 collaborators of Marcus Krüger. A scholar is included among the top collaborators of Marcus Krüger based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Marcus Krüger. Marcus Krüger is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Tellkamp, Frederik, et al.. (2025). Neural stimulation suppresses mTORC1-mediated protein synthesis in skeletal muscle. Science Advances. 11(14). eadt4955–eadt4955. 3 indexed citations

Mörgelin, Matthias, Uwe Hansen, Branko Zevnik, et al.. (2025). Furin-like cleavage at the C1-C2 linker region of the ⍺3 chain is not required for collagen VI assembly. Matrix Biology. 143. 1–13. 1 indexed citations

Ferdek, Paweł E., Marta Targosz‐Korecka, Marcus Krüger, et al.. (2024). Upregulation of utrophin improves the phenotype of Duchenne muscular dystrophy hiPSC-derived CMs. Molecular Therapy — Nucleic Acids. 35(3). 102247–102247. 5 indexed citations

Nolte, Hendrik, Elisabeth Vogelsang, Michiko Takeda, et al.. (2024). Differential regulation of the proteome and phosphoproteome along the dorso-ventral axis of the early Drosophila embryo. eLife. 13. 2 indexed citations

Kopacz, Aleksandra, et al.. (2023). Casein kinase 2 activity is a host restriction factor for AAV transduction. Molecular Therapy. 32(1). 84–102. 6 indexed citations

Leibing, Thomas, Mirco Friedrich, Hendrik Nolte, et al.. (2022). Targeting of Scavenger Receptors Stabilin-1 and Stabilin-2 Ameliorates Atherosclerosis by a Plasma Proteome Switch Mediating Monocyte/Macrophage Suppression. Circulation. 146(23). 1783–1799. 41 indexed citations

Pepin, Mark E., Markus Grosch, Sandra Clauder‐Münster, et al.. (2022). Deep phenotyping of two preclinical mouse models and a cohort of RBM20 mutation carriers reveals no sex-dependent disease severity in RBM20 cardiomyopathy. American Journal of Physiology-Heart and Circulatory Physiology. 323(6). H1296–H1310. 7 indexed citations

Santiago, Ulises, Hendrik Nolte, Rose M. Gathungu, et al.. (2022). A heterotypic assembly mechanism regulates CHIP E3 ligase activity. The EMBO Journal. 41(15). e109566–e109566. 10 indexed citations

Leone, Marina, Fulvia Ferrazzi, Janica L. Wiederstein, et al.. (2021). IQGAP3, a YAP Target, Is Required for Proper Cell-Cycle Progression and Genome Stability. Molecular Cancer Research. 19(10). 1712–1726. 14 indexed citations

10.

Klapproth, Sarah, Karsten Richter, Clara Türk, et al.. (2021). Low kindlin-3 levels in osteoclasts of kindlin-3 hypomorphic mice result in osteopetrosis due to leaky sealing zones. Journal of Cell Science. 134(22). 3 indexed citations

11.

Gerbracht, Jennifer V., Volker Boehm, Thiago Britto‐Borges, et al.. (2020). CASC3 promotes transcriptome-wide activation of nonsense-mediated decay by the exon junction complex. Nucleic Acids Research. 48(15). 8626–8644. 36 indexed citations

12.

Krüger, Marcus, John R. Daum, Johan A. Slotman, et al.. (2019). CDK1-mediated phosphorylation at H2B serine 6 is required for mitotic chromosome segregation. The Journal of Cell Biology. 218(4). 1164–1181. 25 indexed citations

13.

Klapproth, Sarah, Thomas Bromberger, Clara Türk, Marcus Krüger, & Markus Moser. (2019). A kindlin-3–leupaxin–paxillin signaling pathway regulates podosome stability. The Journal of Cell Biology. 218(10). 3436–3454. 25 indexed citations

14.

Lang, Franziska, Hendrik Nolte, Soraya Hölper, et al.. (2017). Dynamic changes in the skeletal muscle proteome during denervation-induced atrophy. Disease Models & Mechanisms. 10(7). 881–896. 66 indexed citations

15.

Aubert, Grégory, Ola J. Martin, Julie L. Horton, et al.. (2016). The Failing Heart Relies on Ketone Bodies as a Fuel. Circulation. 133(8). 698–705. 549 indexed citations breakdown →

16.

Hölper, Soraya, Hendrik Nolte, Eva Bober, Thomas Braun, & Marcus Krüger. (2014). Dissection of metabolic pathways in the Db/Db mouse model by integrative proteome and acetylome analysis. Molecular BioSystems. 11(3). 908–922. 18 indexed citations

17.

Zaglia, Tania, Giulia Milan, Aaron Ruhs, et al.. (2014). Atrogin-1 deficiency promotes cardiomyopathy and premature death via impaired autophagy. Journal of Clinical Investigation. 124(6). 2410–2424. 117 indexed citations

18.

Hamdani, Nazha, Judith Krysiak, Michael M. Kreußer, et al.. (2013). Crucial Role for Ca ²⁺ /Calmodulin-Dependent Protein Kinase-II in Regulating Diastolic Stress of Normal and Failing Hearts via Titin Phosphorylation. Circulation Research. 112(4). 664–674. 143 indexed citations

19.

Pohjoismäki, Jaakko, et al.. (2013). Postnatal cardiomyocyte growth and mitochondrial reorganization cause multiple changes in the proteome of human cardiomyocytes. Molecular BioSystems. 9(6). 1210–1219. 33 indexed citations

20.

Heidler, Juliana, Christian Kukat, Isabelle Salwig, et al.. (2011). Nitric Oxide-associated Protein 1 (NOA1) Is Necessary for Oxygen-dependent Regulation of Mitochondrial Respiratory Complexes. Journal of Biological Chemistry. 286(37). 32086–32093. 25 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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