Clemens Duerrschmid
- Endocrine and Autonomic Systems top 10%
- Physiology top 10%
- Adipose Tissue and Metabolism 2
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- Cardiac Fibrosis and Remodeling 3
- Viral Infections and Immunology Research 1
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- Biochemical Analysis and Sensing Techniques 2
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- Protein Kinase Regulation and GTPase Signaling 1
- Signaling Pathways in Disease 1
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- Phagocytosis and Immune Regulation 1
- Immune Response and Inflammation 1
- Co-authors
- Sandra B. HaudekMark L. EntmanJoAnn TrialAtul R. ChopraDianna M. MilewiczV. Reid SuttonDavid A. RendonM. Waleed Gaber
- Partner nations
- United States
In The Last Decade
Clemens Duerrschmid
7 papers receiving 602 citations
Hit Papers
Peers
Comparison fields: 5 of 82
- Endocrine and Autonomic Systems 71
- Physiology 265
- Cardiology and Cardiovascular Medicine 148
- Endocrinology, Diabetes and Metabolism 75
- Epidemiology 155
Countries citing papers authored by Clemens Duerrschmid
This map shows the geographic impact of Clemens Duerrschmid's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Clemens Duerrschmid with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Clemens Duerrschmid more than expected).
Fields of papers citing papers by Clemens Duerrschmid
This network shows the impact of papers produced by Clemens Duerrschmid. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Clemens Duerrschmid. The network helps show where Clemens Duerrschmid may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Clemens Duerrschmid, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2023 | 13 | |
| 2 | 2021 | 35 | |
| 3 | Asprosin, a Fasting-Induced Glucogenic Protein Hormonebreakdown → | 2016 | 382 |
| 4 | 2016 | 12 | |
| 5 | 2014 | 49 | |
| 6 | 2013 | 37 | |
| 7 | 2013 | 84 |
About Clemens Duerrschmid
Clemens Duerrschmid is a scholar working on Endocrine and Autonomic Systems, Cardiology and Cardiovascular Medicine, Nutrition and Dietetics, Immunology and Physiology, having authored 7 papers that have together received 612 indexed citations. Recurring topics across this work include Cardiac Fibrosis and Remodeling (3 papers), Biochemical Analysis and Sensing Techniques (2 papers), Adipose Tissue and Metabolism (2 papers), Protein Kinase Regulation and GTPase Signaling (1 paper), Viral Infections and Immunology Research (1 paper), Signaling Pathways in Disease (1 paper), Phagocytosis and Immune Regulation (1 paper) and Immune Response and Inflammation (1 paper). The work is most often cited by research in Endocrine and Autonomic Systems (71 citations), Physiology (265 citations), Cardiology and Cardiovascular Medicine (148 citations), Endocrinology, Diabetes and Metabolism (75 citations) and Epidemiology (155 citations). Clemens Duerrschmid has collaborated with scholars based in United States. Frequent co-authors include Sandra B. Haudek, Mark L. Entman, JoAnn Trial, Atul R. Chopra, Dianna M. Milewicz, V. Reid Sutton, David A. Rendon, M. Waleed Gaber, Scott A. LeMaire and Maria del Solar. Their work appears in journals such as Science Advances, eLife, Cell, Journal of Molecular and Cellular Cardiology and Circulation Heart Failure.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.