Christopher Nardone
Impact in
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- Iron Metabolism and Disorders
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- Hemoglobinopathies and Related Disorders
Papers in
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- Ubiquitin and proteasome pathways 5
- Protein Degradation and Inhibitors 2
- Mitochondrial Function and Pathology 1
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- Endoplasmic Reticulum Stress and Disease 2
- Microtubule and mitosis dynamics 1
- Co-authors
- Stephen J. Elledge (6 shared papers)Xin Gu (4 shared papers)Nolan Kamitaki (2 shared papers)Michael E. Greenberg (2 shared papers)Richard T. Timms (2 shared papers)Yumei Leng (2 shared papers)Marianne Wessling‐Resnick (1 shared paper)Bruno G. Nicolau (1 shared paper)
- Journals
- Molecular Cell (3 papers)Science (2 papers)Nature Structural & Molecular Biology (2 papers)Nature (1 paper)Cureus (1 paper)
- Partner nations
- United StatesUnited KingdomGermany
In The Last Decade
Christopher Nardone
9 papers receiving 278 citations
Peers
Comparison fields: 5 of 65
- Hematology 34
- Genetics 25
- Molecular Biology 164
- Nutrition and Dietetics 25
- Cell Biology 23
Countries citing papers authored by Christopher Nardone
This map shows the geographic impact of Christopher Nardone's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Christopher Nardone with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Christopher Nardone more than expected).
Fields of papers citing papers by Christopher Nardone
This network shows the impact of papers produced by Christopher Nardone. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Christopher Nardone. The network helps show where Christopher Nardone may publish in the future.
Co-authors
The 25 scholars most cited alongside Christopher Nardone, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2017 | 115 | |
| 2 | 2023 | 92 | |
| 3 | 2023 | 30 | |
| 4 | 2023 | 28 | |
| 5 | 2025 | 8 | |
| 6 | 2025 | 4 | |
| 7 | 2025 | 2 | |
| 8 | 2025 | 1 | |
| 9 | 2020 | 1 |
About Christopher Nardone
Christopher Nardone is a scholar working on Molecular Biology, Cell Biology, Surgery, Genetics and Pathology and Forensic Medicine, having authored 9 papers that have together received 281 indexed citations. Recurring topics across this work include Ubiquitin and proteasome pathways (5 papers), Endoplasmic Reticulum Stress and Disease (2 papers), Protein Degradation and Inhibitors (2 papers), Folate and B Vitamins Research (1 paper), Cancer Mechanisms and Therapy (1 paper), Mitochondrial Function and Pathology (1 paper), Hemoglobinopathies and Related Disorders (1 paper) and Microtubule and mitosis dynamics (1 paper). The work is most often cited by research in Hematology (34 citations), Genetics (25 citations), Molecular Biology (164 citations), Nutrition and Dietetics (25 citations) and Cell Biology (23 citations). Christopher Nardone has collaborated with scholars based in United States, United Kingdom and Germany. Frequent co-authors include Stephen J. Elledge, Xin Gu, Nolan Kamitaki, Michael E. Greenberg, Richard T. Timms, Yumei Leng, Marianne Wessling‐Resnick, Bruno G. Nicolau, Young Ah Seo and Murui Han. Their work appears in journals such as Molecular Cell, Science, Nature Structural & Molecular Biology, Nature and Cureus.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.