Anfeng Mu

445 total citations
13 papers, 196 citations indexed

About

Anfeng Mu is a scholar working on Molecular Biology, Pediatrics, Perinatology and Child Health and Oncology. According to data from OpenAlex, Anfeng Mu has authored 13 papers receiving a total of 196 indexed citations (citations by other indexed papers that have themselves been cited), including 11 papers in Molecular Biology, 3 papers in Pediatrics, Perinatology and Child Health and 3 papers in Oncology. Recurrent topics in Anfeng Mu's work include DNA Repair Mechanisms (7 papers), CRISPR and Genetic Engineering (3 papers) and Mitochondrial Function and Pathology (3 papers). Anfeng Mu is often cited by papers focused on DNA Repair Mechanisms (7 papers), CRISPR and Genetic Engineering (3 papers) and Mitochondrial Function and Pathology (3 papers). Anfeng Mu collaborates with scholars based in Japan, Sweden and Canada. Anfeng Mu's co-authors include Shigeru Taketani, Sakihito Kitajima, Minoru Takata, Takaaki Takeda, Yasushi Adachi, Yusuke Okamoto, Asuka Hira, Ngoc‐Lan Nguyen, Masato T. Kanemaki and Keitaro Matsuo and has published in prestigious journals such as Blood, Scientific Reports and FEBS Letters.

In The Last Decade

Anfeng Mu

13 papers receiving 194 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Anfeng Mu Japan 7 134 38 33 31 30 13 196
Amaia Arruabarrena-Aristorena Spain 6 209 1.6× 34 0.9× 14 0.4× 71 2.3× 26 0.9× 7 276
Antonio Morales‐Hernández United States 10 127 0.9× 32 0.8× 8 0.2× 40 1.3× 11 0.4× 16 253
Qingting Hu United States 11 146 1.1× 103 2.7× 25 0.8× 56 1.8× 26 0.9× 15 354
Marco Ballarini Italy 6 279 2.1× 48 1.3× 19 0.6× 22 0.7× 7 0.2× 8 328
Gloria E. Mao United States 7 239 1.8× 20 0.5× 36 1.1× 19 0.6× 21 0.7× 9 329
Wolfgang Glaesner United States 7 210 1.6× 34 0.9× 8 0.2× 13 0.4× 9 0.3× 9 414
Laura Barbieri Sweden 9 112 0.8× 111 2.9× 11 0.3× 70 2.3× 17 0.6× 13 332
Mieko Matsuyama United States 6 143 1.1× 18 0.5× 11 0.3× 31 1.0× 16 0.5× 10 203
Martin D. Kafina United States 6 126 0.9× 11 0.3× 13 0.4× 11 0.4× 19 0.6× 8 190
S. Kim United States 9 178 1.3× 25 0.7× 19 0.6× 20 0.6× 20 0.7× 11 289

Countries citing papers authored by Anfeng Mu

Since Specialization
Citations

This map shows the geographic impact of Anfeng Mu's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Anfeng Mu with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Anfeng Mu more than expected).

Fields of papers citing papers by Anfeng Mu

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Anfeng Mu. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Anfeng Mu. The network helps show where Anfeng Mu may publish in the future.

Co-authorship network of co-authors of Anfeng Mu

This figure shows the co-authorship network connecting the top 25 collaborators of Anfeng Mu. A scholar is included among the top collaborators of Anfeng Mu based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Anfeng Mu. Anfeng Mu is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

13 of 13 papers shown
1.
Mu, Anfeng, Yusuke Okamoto, Y Katsuki, & Minoru Takata. (2024). The role of SLFN11 in DNA replication stress response and its implications for the Fanconi anemia pathway. DNA repair. 141. 103733–103733. 1 indexed citations
2.
3.
Katsuki, Y, et al.. (2023). Mouse Slfn8 and Slfn9 genes complement human cells lacking SLFN11 during the replication stress response. Communications Biology. 6(1). 1038–1038. 6 indexed citations
4.
Mu, Anfeng, Asuka Hira, Minako Mori, Yusuke Okamoto, & Minoru Takata. (2023). Fanconi anemia and Aldehyde Degradation Deficiency Syndrome: Metabolism and DNA repair protect the genome and hematopoiesis from endogenous DNA damage. DNA repair. 130. 103546–103546. 5 indexed citations
5.
Mu, Anfeng, et al.. (2023). Effects of the major formaldehyde catalyzer ADH5 on phenotypes of fanconi anemia zebrafish model. Molecular Biology Reports. 50(10). 8385–8395. 3 indexed citations
6.
Mu, Anfeng, Asuka Hira, Akira Niwa, et al.. (2021). Analysis of disease model iPSCs derived from patients with a novel Fanconi anemia–like IBMFS ADH5/ALDH2 deficiency. Blood. 137(15). 2021–2032. 27 indexed citations
7.
Mu, Anfeng, Asuka Hira, Keitaro Matsuo, & Minoru Takata. (2021). [Aldehyde degradation deficiency (ADD) syndrome: discovery of a novel fanconi anemia-like inherited BMF syndrome due to combined ADH5/ALDH2 deficiency].. PubMed. 62(6). 547–553. 5 indexed citations
8.
Okamoto, Yusuke, Masako Abe, Anfeng Mu, et al.. (2020). SLFN11 promotes stalled fork degradation that underlies the phenotype in Fanconi anemia cells. Blood. 137(3). 336–348. 25 indexed citations
9.
Takeda, Takaaki, et al.. (2015). Continuous de novo biosynthesis of haem and its rapid turnover to bilirubin are necessary for cytoprotection against cell damage. Scientific Reports. 5(1). 10488–10488. 41 indexed citations
10.
Liem, Pham Hieu, et al.. (2015). A simple and highly sensitive method of measuring heme oxygenase activity. Biological Chemistry. 396(11). 1265–1268. 4 indexed citations
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13.
Fujita, Ken‐ichi, et al.. (2013). Imaging of heme/hemeproteins in nucleus of the living cells expressing heme‐binding nuclear receptors. FEBS Letters. 587(14). 2131–2136. 8 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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2026