Christopher J. Stefan
- Molecular Biology top 2%
- Cell Biology top 0.2%
- Epidemiology top 5%
- Physiology top 5%
- Cellular and Molecular Neuroscience top 5%
- Co-authors
- Scott D. EmrAndrew G. ManfordJason A. MacGurnStefan NobelSten OrreniusAndrew F.G. SlaterAnjon AudhyaKendall Blumer
- Topics
- Cellular transport and secretion (42 papers)Endoplasmic Reticulum Stress and Disease (20 papers)Fungal and yeast genetics research (17 papers)
- Partner nations
- United StatesUnited KingdomGermany
In The Last Decade
Christopher J. Stefan
59 papers receiving 5.0k citations
Hit Papers
Peers
Comparison fields: 5 of 109
- Molecular Biology 3.7k
- Cell Biology 2.8k
- Epidemiology 543
- Physiology 470
- Cellular and Molecular Neuroscience 374
Countries citing papers authored by Christopher J. Stefan
This map shows the geographic impact of Christopher J. Stefan's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Christopher J. Stefan with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Christopher J. Stefan more than expected).
Fields of papers citing papers by Christopher J. Stefan
This network shows the impact of papers produced by Christopher J. Stefan. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Christopher J. Stefan. The network helps show where Christopher J. Stefan may publish in the future.
Co-authorship network of co-authors of Christopher J. Stefan
This figure shows the co-authorship network connecting the top 25 collaborators of Christopher J. Stefan. A scholar is included among the top collaborators of Christopher J. Stefan based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Christopher J. Stefan. Christopher J. Stefan is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 31 | |
| 2 | 2 | |
| 3 | 9 | |
| 4 | 9 | |
| 5 | 35 | |
| 6 | 82 | |
| 7 | 65 | |
| 8 | 18 | |
| 9 | 38 | |
| 10 | 390 | |
| 11 | 336 | |
| 12 | 75 | |
| 13 | 38 | |
| 14 | 90 | |
| 15 | 395 | |
| 16 | 65 | |
| 17 | 64 | |
| 18 | 135 | |
| 19 | 120 | |
| 20 | 46 |
About Christopher J. Stefan
Christopher J. Stefan is a scholar working on Cell Biology, Physiology and Molecular Biology, having authored 59 papers that have together received 5.0k indexed citations. Recurring topics across this work include Cellular transport and secretion (42 papers), Endoplasmic Reticulum Stress and Disease (20 papers) and Fungal and yeast genetics research (17 papers). The work is most often cited by research in Cell Biology (2.8k citations), Physiology (374 citations) and Molecular Biology (3.7k citations). Christopher J. Stefan has collaborated with scholars based in United States, United Kingdom and Germany. Frequent co-authors include Scott D. Emr, Andrew G. Manford, Jason A. MacGurn, Stefan Nobel, Sten Orrenius, Andrew F.G. Slater, Anjon Audhya, Kendall Blumer, David J. Katzmann and Markus Babst. Their work appears in journals such as Cell, Proceedings of the National Academy of Sciences and Nucleic Acids Research.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.