Chiara Costanzi
- Psychiatry and Mental health top 10%
- Physiology
- Cellular and Molecular Neuroscience top 10%
- Molecular Biology
- Neurology
- Co-authors
- Alessandro PadovaniBarbara BorroniLuigi CaimiSilvana ArchettiMario GrassiMónica Di LucaCarlo CaltagironeSimone Franzoni
- Topics
- Alzheimer's disease research and treatments (5 papers)Dementia and Cognitive Impairment Research (4 papers)Mitochondrial Function and Pathology (3 papers)
- Partner nations
- ItalyUnited StatesBelarus
In The Last Decade
Chiara Costanzi
17 papers receiving 542 citations
Peers
Comparison fields: 5 of 72
- Psychiatry and Mental health 181
- Physiology 177
- Cellular and Molecular Neuroscience 156
- Molecular Biology 102
- Neurology 84
Countries citing papers authored by Chiara Costanzi
This map shows the geographic impact of Chiara Costanzi's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Chiara Costanzi with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Chiara Costanzi more than expected).
Fields of papers citing papers by Chiara Costanzi
This network shows the impact of papers produced by Chiara Costanzi. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Chiara Costanzi. The network helps show where Chiara Costanzi may publish in the future.
Co-authorship network of co-authors of Chiara Costanzi
This figure shows the co-authorship network connecting the top 25 collaborators of Chiara Costanzi. A scholar is included among the top collaborators of Chiara Costanzi based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Chiara Costanzi. Chiara Costanzi is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 4 | |
| 2 | 15 | |
| 3 | 0 | |
| 4 | 16 | |
| 5 | 24 | |
| 6 | 27 | |
| 7 | 43 | |
| 8 | 55 | |
| 9 | 0 | |
| 10 | 58 | |
| 11 | 33 | |
| 12 | 20 | |
| 13 | 30 | |
| 14 | 26 | |
| 15 | 33 | |
| 16 | 46 | |
| 17 | 68 | |
| 18 | 45 | |
| 19 | The molecular genetics of colon cancer. | 17 |
About Chiara Costanzi
Chiara Costanzi is a scholar working on Psychiatry and Mental health, Cellular and Molecular Neuroscience and Internal Medicine, having authored 19 papers that have together received 560 indexed citations. Recurring topics across this work include Alzheimer's disease research and treatments (5 papers), Dementia and Cognitive Impairment Research (4 papers) and Mitochondrial Function and Pathology (3 papers). The work is most often cited by research in Biological Psychiatry (41 citations), Psychiatry and Mental health (181 citations) and Cellular and Molecular Neuroscience (156 citations). Chiara Costanzi has collaborated with scholars based in Italy, United States and Belarus. Frequent co-authors include Alessandro Padovani, Barbara Borroni, Luigi Caimi, Silvana Archetti, Mario Grassi, Mónica Di Luca, Carlo Caltagirone, Simone Franzoni, Nicola Gilberti and C. Agosti. Their work appears in journals such as Annals of Neurology, Neurobiology of Aging and Journal of the Neurological Sciences.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.