Changli Wei

7.7k total citations · 1 hit paper
61 papers, 4.2k citations indexed

About

Changli Wei is a scholar working on Nephrology, Molecular Biology and Pathology and Forensic Medicine. According to data from OpenAlex, Changli Wei has authored 61 papers receiving a total of 4.2k indexed citations (citations by other indexed papers that have themselves been cited), including 44 papers in Nephrology, 11 papers in Molecular Biology and 10 papers in Pathology and Forensic Medicine. Recurrent topics in Changli Wei's work include Renal Diseases and Glomerulopathies (42 papers), Chronic Kidney Disease and Diabetes (20 papers) and Autoimmune Bullous Skin Diseases (7 papers). Changli Wei is often cited by papers focused on Renal Diseases and Glomerulopathies (42 papers), Chronic Kidney Disease and Diabetes (20 papers) and Autoimmune Bullous Skin Diseases (7 papers). Changli Wei collaborates with scholars based in United States, Germany and China. Changli Wei's co-authors include Jochen Reiser, Mehmet M. Altintas, Clemens Möller, Maria Pia Rastaldi, Peter Mündel, Raghu Kalluri, Matthias Kretzler, Sanja Sever, Christian Faul and George W. Burke and has published in prestigious journals such as New England Journal of Medicine, Journal of Biological Chemistry and Journal of Clinical Investigation.

In The Last Decade

Changli Wei

59 papers receiving 4.2k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

TRPC6 is a glomerular slit diaphragm-associated channel required for normal renal function

2005 647 citations Jochen Reiser, Krishna R. Polu et al. Nature Genetics profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Changli Wei United States	27	2.9k	1.2k	663	651	580	61	4.2k
Maria Pia Rastaldi Italy	43	4.1k 1.4×	2.6k 2.1×	871 1.3×	1.4k 2.1×	480 0.8×	120	7.3k
Alkis Pierides Cyprus	30	1.2k 0.4×	1.7k 1.3×	538 0.8×	405 0.6×	264 0.5×	88	3.8k
Richard J. Coward United Kingdom	31	2.3k 0.8×	1.4k 1.1×	365 0.6×	416 0.6×	204 0.4×	72	4.0k
Fujio Shimizu Japan	35	1.9k 0.7×	1.4k 1.1×	311 0.5×	569 0.9×	283 0.5×	118	3.7k
Andrey V. Cybulsky Canada	36	1.8k 0.6×	1.3k 1.1×	455 0.7×	1.0k 1.6×	230 0.4×	114	3.9k
Lan Ni United Kingdom	25	1.7k 0.6×	1.3k 1.1×	278 0.4×	294 0.5×	197 0.3×	43	2.9k
Catherine Meyer‐Schwesinger Germany	37	2.2k 0.8×	1.3k 1.1×	678 1.0×	1.3k 2.0×	267 0.5×	93	4.6k
Weichun He China	33	1.3k 0.5×	2.0k 1.6×	459 0.7×	375 0.6×	145 0.3×	55	3.9k
Keiju Hiromura Japan	29	1.2k 0.4×	1.1k 0.9×	259 0.4×	866 1.3×	231 0.4×	123	3.4k
Marcus J. Moeller Germany	42	2.9k 1.0×	2.3k 1.9×	392 0.6×	484 0.7×	213 0.4×	97	5.0k

Countries citing papers authored by Changli Wei

Since Specialization

Citations

This map shows the geographic impact of Changli Wei's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Changli Wei with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Changli Wei more than expected).

Fields of papers citing papers by Changli Wei

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Changli Wei. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Changli Wei. The network helps show where Changli Wei may publish in the future.

Co-authorship network of co-authors of Changli Wei

This figure shows the co-authorship network connecting the top 25 collaborators of Changli Wei. A scholar is included among the top collaborators of Changli Wei based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Changli Wei. Changli Wei is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Chu, Jian, Changli Wei, Melody Cobleigh, et al.. (2024). Soluble urokinase plasminogen activator receptor and cardiotoxicity in doxorubicin-treated breast cancer patients: a prospective exploratory study. Cardio-Oncology. 10(1). 3–3. 2 indexed citations

Yu, Ziqi, et al.. (2024). Iron metabolism and ferroptosis in nonalcoholic fatty liver disease: what is our next step?. American Journal of Physiology-Endocrinology and Metabolism. 326(6). E767–E775. 10 indexed citations

Zhu, Ke, Kamalika Mukherjee, Changli Wei, et al.. (2023). The D2D3 form of uPAR acts as an immunotoxin and may cause diabetes and kidney disease. Science Translational Medicine. 15(714). eabq6492–eabq6492. 1 indexed citations

Polat, Onur, Ke Zhu, Manuel Noben, et al.. (2023). The small GTPase regulatory protein Rac1 drives podocyte injury independent of cationic channel protein TRPC5. Kidney International. 103(6). 1056–1062. 14 indexed citations

Hayek, Salim S., Ayman Samman Tahhan, Yi‐An Ko, et al.. (2022). Soluble Urokinase Plasminogen Activator Receptor Levels and Outcomes in Patients with Heart Failure. Journal of Cardiac Failure. 29(2). 158–167. 25 indexed citations

Hladunewich, Michelle, D C Cattran, Sanjeev Sethi, et al.. (2021). Efficacy of Rituximab in Treatment-Resistant Focal Segmental Glomerulosclerosis With Elevated Soluble Urokinase-Type Plasminogen Activator Receptor and Activation of Podocyte β3 Integrin. Kidney International Reports. 7(1). 68–77. 10 indexed citations

Hayek, Salim S., Douglas Landsittel, Changli Wei, et al.. (2019). Soluble Urokinase Plasminogen Activator Receptor and Decline in Kidney Function in Autosomal Dominant Polycystic Kidney Disease. Journal of the American Society of Nephrology. 30(7). 1305–1313. 23 indexed citations

Ren, Guohui, Nicholas J. Tardi, Fumihiko Matsuda, et al.. (2017). Podocytes exhibit a specialized protein quality control employing derlin-2 in kidney disease. American Journal of Physiology-Renal Physiology. 314(3). F471–F482. 11 indexed citations

Hayek, Salim S., Yi-An Ko, Mosaab Awad, et al.. (2017). Cardiovascular Disease Biomarkers and suPAR in Predicting Decline in Renal Function: A Prospective Cohort Study. Kidney International Reports. 2(3). 425–432. 21 indexed citations

10.

Hahm, Eunsil, Changli Wei, Isabel Cuesta Fernández, et al.. (2016). Bone marrow-derived immature myeloid cells are a main source of circulating suPAR contributing to proteinuric kidney disease. Nature Medicine. 23(1). 100–106. 101 indexed citations

11.

Staeck, Oliver, Torsten Slowinski, Ina Lieker, et al.. (2015). Recurrent Primary Focal Segmental Glomerulosclerosis Managed With Intensified Plasma Exchange and Concomitant Monitoring of Soluble Urokinase-Type Plasminogen Activator Receptor–Mediated Podocyte β3-integrin Activation. Transplantation. 99(12). 2593–2597. 30 indexed citations

12.

Garin, Eduardo H., Jochen Reiser, Gabriel Cara‐Fuentes, et al.. (2014). Case series: CTLA4-IgG1 therapy in minimal change disease and focal segmental glomerulosclerosis. Pediatric Nephrology. 30(3). 469–477. 64 indexed citations

13.

Alachkar, Nada, Changli Wei, Lois J. Arend, et al.. (2013). Podocyte Effacement Closely Links to suPAR Levels at Time of Posttransplantation Focal Segmental Glomerulosclerosis Occurrence and Improves With Therapy. Transplantation. 96(7). 649–656. 51 indexed citations

14.

Reiser, Jochen, Changli Wei, & James A. Tumlin. (2012). Soluble urokinase receptor and focal segmental glomerulosclerosis. Current Opinion in Nephrology & Hypertension. 21(4). 428–432. 26 indexed citations

15.

Daftarian, Pirouz, Changli Wei, Alan King, et al.. (2011). In vivo Electroporation and Non-protein Based Screening Assays to Identify Antibodies Against Native Protein Conformations. Hybridoma. 30(5). 409–418. 6 indexed citations

16.

Wei, Changli, Clemens Möller, Mehmet M. Altintas, et al.. (2007). Modification of kidney barrier function by the urokinase receptor. Nature Medicine. 14(1). 55–63. 419 indexed citations

17.

Wei, Changli, Mehmet M. Altintas, Jing Li, et al.. (2006). Induction of TRPC6 Channel in Acquired Forms of Proteinuric Kidney Disease. Journal of the American Society of Nephrology. 18(1). 29–36. 243 indexed citations

18.

Reiser, Jochen, Krishna R. Polu, Clemens Möller, et al.. (2005). TRPC6 is a glomerular slit diaphragm-associated channel required for normal renal function. Nature Genetics. 37(7). 739–744. 647 indexed citations breakdown →

19.

Wei, Changli. (2004). Temporal expression of hepatic inducible nitric oxide synthase in liver cirrhosis. World Journal of Gastroenterology. 11(3). 362–362. 14 indexed citations

20.

Wei, Changli, et al.. (2002). Differential expression and localization of nitric oxide synthases in cirrhotic livers of bile duct-ligated rats. Nitric Oxide. 7(2). 91–102. 39 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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