Martin R. Pollak
- Nephrology top 0.01%
- Molecular Biology top 1%
- Genetics top 0.5%
- Genetics top 0.2%
- Pathology and Forensic Medicine top 0.5%
- Co-authors
- David J. FriedmanEdward M. BrownSteven HébertGiulio GenoveseChristine E. SeidmanJonathan G. SeidmanJeffrey B. KoppJohannes Schlöndorff
- Topics
- Renal Diseases and Glomerulopathies (118 papers)Chronic Kidney Disease and Diabetes (59 papers)Renal and related cancers (35 papers)
- Cited by
- NephrologyGeneticsTransplantation
- Partner nations
- United StatesCanadaChina
In The Last Decade
Martin R. Pollak
185 papers receiving 14.4k citations
Hit Papers
Peers
Comparison fields: 5 of 152
- Nephrology 9.4k
- Molecular Biology 5.5k
- Genetics 2.3k
- Genetics 2.1k
- Pathology and Forensic Medicine 1.5k
Countries citing papers authored by Martin R. Pollak
This map shows the geographic impact of Martin R. Pollak's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Martin R. Pollak with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Martin R. Pollak more than expected).
Fields of papers citing papers by Martin R. Pollak
This network shows the impact of papers produced by Martin R. Pollak. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Martin R. Pollak. The network helps show where Martin R. Pollak may publish in the future.
Co-authorship network of co-authors of Martin R. Pollak
This figure shows the co-authorship network connecting the top 25 collaborators of Martin R. Pollak. A scholar is included among the top collaborators of Martin R. Pollak based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Martin R. Pollak. Martin R. Pollak is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 1 | |
| 2 | 12 | |
| 3 | 3 | |
| 4 | 14 | |
| 5 | 22 | |
| 6 | 35 | |
| 7 | 40 | |
| 8 | 41 | |
| 9 | 43 | |
| 10 | 11 | |
| 11 | 110 | |
| 12 | 84 | |
| 13 | 94 | |
| 14 | Association of Trypanolytic ApoL1 Variants with Kidney Disease in African Americansbreakdown → | 1329 |
| 15 | 22 | |
| 16 | 119 | |
| 17 | 104 | |
| 18 | 159 | |
| 19 | 143 | |
| 20 | 197 |
About Martin R. Pollak
Martin R. Pollak is a scholar working on Nephrology, Genetics and Gastroenterology, having authored 189 papers that have together received 14.6k indexed citations. Recurring topics across this work include Renal Diseases and Glomerulopathies (118 papers), Chronic Kidney Disease and Diabetes (59 papers) and Renal and related cancers (35 papers). The work is most often cited by research in Nephrology (9.4k citations), Genetics (2.1k citations) and Transplantation (349 citations). Martin R. Pollak has collaborated with scholars based in United States, Canada and China. Frequent co-authors include David J. Friedman, Edward M. Brown, Steven Hébert, Giulio Genovese, Christine E. Seidman, Jonathan G. Seidman, Jeffrey B. Kopp, Johannes Schlöndorff, Claudine H. Kos and Olga Kifor. Their work appears in journals such as Nature, Science and New England Journal of Medicine.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.