Martin R. Pollak

23.2k total citations · 6 hit papers
189 papers, 14.6k citations indexed

About

Martin R. Pollak is a scholar working on Nephrology, Molecular Biology and Genetics. According to data from OpenAlex, Martin R. Pollak has authored 189 papers receiving a total of 14.6k indexed citations (citations by other indexed papers that have themselves been cited), including 151 papers in Nephrology, 84 papers in Molecular Biology and 31 papers in Genetics. Recurrent topics in Martin R. Pollak's work include Renal Diseases and Glomerulopathies (118 papers), Chronic Kidney Disease and Diabetes (59 papers) and Renal and related cancers (35 papers). Martin R. Pollak is often cited by papers focused on Renal Diseases and Glomerulopathies (118 papers), Chronic Kidney Disease and Diabetes (59 papers) and Renal and related cancers (35 papers). Martin R. Pollak collaborates with scholars based in United States, Canada and China. Martin R. Pollak's co-authors include David J. Friedman, Edward M. Brown, Steven Hébert, Giulio Genovese, Christine E. Seidman, Jonathan G. Seidman, Jeffrey B. Kopp, Johannes Schlöndorff, Claudine H. Kos and Olga Kifor and has published in prestigious journals such as Nature, Science and New England Journal of Medicine.

In The Last Decade

Martin R. Pollak

185 papers receiving 14.4k citations

Hit Papers

align trajectories sort by overperformance

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Association of Trypanolytic ApoL1 Variants with Kidney Disease in African Americans

2010 1.3k citations Giulio Genovese, David J. Friedman et al. Science profile →
Mutations in ACTN4, encoding α-actinin-4, cause familial focal segmental glomerulosclerosis

2000 951 citations Martin R. Pollak et al. profile →
Mutations in the human Ca2+-sensing receptor gene cause familial hypocalciuric hypercalcemia and neonatal severe hyperparathyroidism

1993 739 citations Martin R. Pollak, Edward M. Brown et al. profile →
TRPC6 is a glomerular slit diaphragm-associated channel required for normal renal function

2005 647 citations Hikaru Sugimoto, Raghu Kalluri et al. profile →
Stem cell engraftment at the endosteal niche is specified by the calcium-sensing receptor

2005 549 citations Martin R. Pollak, Edward M. Brown et al. profile →
Inaxaplin for Proteinuric Kidney Disease in Persons with Two APOL1 Variants

2023 98 citations David J. Friedman, Martin R. Pollak et al. profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Martin R. Pollak United States	61	9.4k	5.5k	2.3k	2.1k	1.5k	189	14.6k
Jochen Reiser United States	51	9.1k 1.0×	5.3k 1.0×	2.0k 0.9×	1.3k 0.6×	1.5k 1.0×	179	14.1k
Dontscho Kerjaschki Austria	79	6.5k 0.7×	8.4k 1.5×	1.7k 0.7×	981 0.5×	1.5k 1.0×	225	21.3k
Roger C. Wiggins United States	51	4.5k 0.5×	3.5k 0.6×	1.2k 0.5×	790 0.4×	671 0.4×	195	9.7k
George C. Tsokos United States	83	1.3k 0.1×	6.2k 1.1×	1.7k 0.7×	1.1k 0.5×	1.8k 1.2×	603	28.8k
Hermann-Josef Gröne Germany	62	1.7k 0.2×	5.0k 0.9×	878 0.4×	687 0.3×	512 0.3×	170	11.9k
Susan E. Quaggin United States	60	4.4k 0.5×	6.0k 1.1×	1.5k 0.7×	428 0.2×	604 0.4×	156	12.2k
Peter J. Nelson Germany	67	2.0k 0.2×	4.8k 0.9×	1.1k 0.5×	1.1k 0.5×	593 0.4×	252	13.8k
Michael Kashgarian United States	63	2.6k 0.3×	5.1k 0.9×	750 0.3×	620 0.3×	700 0.5×	265	13.5k
Søren K. Moestrup Denmark	85	1.4k 0.2×	7.7k 1.4×	1.2k 0.5×	1.3k 0.6×	1.0k 0.7×	246	20.0k
J. Evan Sadler United States	77	1.4k 0.2×	4.2k 0.8×	2.0k 0.9×	2.6k 1.2×	235 0.2×	190	17.9k

Countries citing papers authored by Martin R. Pollak

Since Specialization

Citations

This map shows the geographic impact of Martin R. Pollak's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Martin R. Pollak with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Martin R. Pollak more than expected).

Fields of papers citing papers by Martin R. Pollak

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Martin R. Pollak. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Martin R. Pollak. The network helps show where Martin R. Pollak may publish in the future.

Co-authorship network of co-authors of Martin R. Pollak

This figure shows the co-authorship network connecting the top 25 collaborators of Martin R. Pollak. A scholar is included among the top collaborators of Martin R. Pollak based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Martin R. Pollak. Martin R. Pollak is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Pollak, Martin R. & David J. Friedman. (2025). APOL1-associated kidney disease: modulators of the genotype-phenotype relationship. Current Opinion in Nephrology & Hypertension. 34(3). 191–198. 2 indexed citations

Simeone, Christopher A., Michelle T. McNulty, Yask Gupta, et al.. (2024). The APOL1 p.N264K variant is co-inherited with the G2 kidney disease risk variant through a proximity recombination event. G3 Genes Genomes Genetics. 15(2). 2 indexed citations

Lang, Konrad, Claire Leroy, Mengmeng Chen, et al.. (2022). Nephrotic Syndrome Gene TBC1D8B Is Required for Endosomal Maturation and Nephrin Endocytosis in Drosophila. Journal of the American Society of Nephrology. 33(12). 2174–2193. 12 indexed citations

Nadkarni, Girish N., Kezhen Fei, Geneviève Galarneau, et al.. (2021). APOL1 renal risk variants are associated with obesity and body composition in African ancestry adults: An observational genotype-phenotype association study.. PubMed. 100(45). e27785–e27785. 3 indexed citations

Feng, Di, Mukesh Kumar, Jan Muntel, et al.. (2020). Phosphorylation of ACTN4 Leads to Podocyte Vulnerability and Proteinuric Glomerulosclerosis. Journal of the American Society of Nephrology. 31(7). 1479–1495. 22 indexed citations

Shah, Shrijal S., et al.. (2019). Apolipoprotein L1 (APOL1) risk variant toxicity depends on the haplotype background. Kidney International. 96(6). 1303–1307. 40 indexed citations

Fan, Yi, Weiqing Liu, Ruiye Bi, et al.. (2018). Interrelated role of Klotho and calcium-sensing receptor in parathyroid hormone synthesis and parathyroid hyperplasia. Proceedings of the National Academy of Sciences. 115(16). E3749–E3758. 41 indexed citations

Feng, Di, Jacob Notbohm, Shijie He, et al.. (2018). Disease-causing mutation in α-actinin-4 promotes podocyte detachment through maladaptation to periodic stretch. Proceedings of the National Academy of Sciences. 115(7). 1517–1522. 43 indexed citations

Feng, Di, Julia Steinke, Ramaswamy Krishnan, Gabriel Birrane, & Martin R. Pollak. (2016). Functional Validation of an Alpha-Actinin-4 Mutation as a Potential Cause of an Aggressive Presentation of Adolescent Focal Segmental Glomerulosclerosis: Implications for Genetic Testing. PLoS ONE. 11(12). e0167467–e0167467. 11 indexed citations

10.

Ito, Kaoru, Alexander G. Bick, Jason Flannick, et al.. (2013). Increased Burden of Cardiovascular Disease in Carriers of APOL1 Genetic Variants. Circulation Research. 114(5). 845–850. 110 indexed citations

11.

Sun, Hua, Johannes Schlöndorff, Henry N. Higgs, & Martin R. Pollak. (2013). Inverted Formin 2 Regulates Actin Dynamics by Antagonizing Rho/Diaphanous-related Formin Signaling. Journal of the American Society of Nephrology. 24(6). 917–929. 44 indexed citations

12.

Barua, Moumita, et al.. (2013). Exome sequencing and in vitro studies identified podocalyxin as a candidate gene for focal and segmental glomerulosclerosis. Kidney International. 85(1). 124–133. 36 indexed citations

13.

Quinn, Stephen, A. Thomsen, Jian Pang, et al.. (2012). Interactions between calcium and phosphorus in the regulation of the production of fibroblast growth factor 23 in vivo. American Journal of Physiology-Endocrinology and Metabolism. 304(3). E310–E320. 84 indexed citations

14.

Kumar, Vineeta, Tom A. Williams, Reza Abdi, et al.. (2012). The APOL1 Genotype of African American Kidney Transplant Recipients Does Not Impact 5-Year Allograft Survival. American Journal of Transplantation. 12(7). 1924–1928. 133 indexed citations

15.

Friedman, David J. & Martin R. Pollak. (2011). Genetics of kidney failure and the evolving story of APOL1. Journal of Clinical Investigation. 121(9). 3367–3374. 94 indexed citations

16.

Genovese, Giulio, David J. Friedman, Michael D. Ross, et al.. (2010). Association of Trypanolytic ApoL1 Variants with Kidney Disease in African Americans. Science. 329(5993). 841–845. 1329 indexed citations breakdown →

17.

Rockmore, Daniel N., et al.. (2008). A SNP Streak Model for the Identification of Genetic Regions Identical-by-descent. Statistical Applications in Genetics and Molecular Biology. 7(1). Article16–Article16. 22 indexed citations

18.

Weins, Astrid, Johannes Schlöndorff, Fumihiko Nakamura, et al.. (2007). Disease-associated mutant α-actinin-4 reveals a mechanism for regulating its F-actin-binding affinity. Proceedings of the National Academy of Sciences. 104(41). 16080–16085. 119 indexed citations

19.

Dandapani, Savita, Hikaru Sugimoto, Benjamin D. Matthews, et al.. (2006). α-Actinin-4 Is Required for Normal Podocyte Adhesion. Journal of Biological Chemistry. 282(1). 467–477. 104 indexed citations

20.

Pollak, Martin R., Yah-Huei Chou, Stephen J. Marx, et al.. (1994). Familial hypocalciuric hypercalcemia and neonatal severe hyperparathyroidism. Effects of mutant gene dosage on phenotype.. Journal of Clinical Investigation. 93(3). 1108–1112. 197 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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