Cemil Özcelik

2.2k total citations
24 papers, 1.1k citations indexed

About

Cemil Özcelik is a scholar working on Cardiology and Cardiovascular Medicine, Molecular Biology and Epidemiology. According to data from OpenAlex, Cemil Özcelik has authored 24 papers receiving a total of 1.1k indexed citations (citations by other indexed papers that have themselves been cited), including 16 papers in Cardiology and Cardiovascular Medicine, 11 papers in Molecular Biology and 3 papers in Epidemiology. Recurrent topics in Cemil Özcelik's work include Cardiomyopathy and Myosin Studies (5 papers), Congenital heart defects research (4 papers) and Cardiovascular Function and Risk Factors (4 papers). Cemil Özcelik is often cited by papers focused on Cardiomyopathy and Myosin Studies (5 papers), Congenital heart defects research (4 papers) and Cardiovascular Function and Risk Factors (4 papers). Cemil Özcelik collaborates with scholars based in Germany, United Kingdom and Spain. Cemil Özcelik's co-authors include Michael Bäder, Santhosh Kumar Ghadge, Silke Mühlstedt, Wilhelm Haverkamp, Daniel A. Morris, Andreas Perrot, Rainer Dietz, Florian Blaschke, Maximilian Posch and H. Eichstädt and has published in prestigious journals such as PLoS ONE, The FASEB Journal and Biophysical Journal.

In The Last Decade

Cemil Özcelik

24 papers receiving 1.1k citations

Peers

Cemil Özcelik
Paul J C Barenbrug Netherlands
Luke Pater United States
Rosaly Correa United States
Tamer Rezk United Kingdom
Cemil Özcelik
Citations per year, relative to Cemil Özcelik Cemil Özcelik (= 1×) peers Lionel Leroux

Countries citing papers authored by Cemil Özcelik

Since Specialization
Citations

This map shows the geographic impact of Cemil Özcelik's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Cemil Özcelik with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Cemil Özcelik more than expected).

Fields of papers citing papers by Cemil Özcelik

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Cemil Özcelik. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Cemil Özcelik. The network helps show where Cemil Özcelik may publish in the future.

Co-authorship network of co-authors of Cemil Özcelik

This figure shows the co-authorship network connecting the top 25 collaborators of Cemil Özcelik. A scholar is included among the top collaborators of Cemil Özcelik based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Cemil Özcelik. Cemil Özcelik is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Nielsen, Tanja, Marcel Grunert, Andreas Perrot, et al.. (2020). Identification of MYOM2 as a candidate gene in hypertrophic cardiomyopathy and Tetralogy of Fallot, and its functional evaluation in the Drosophila heart. Disease Models & Mechanisms. 13(12). 22 indexed citations
2.
Özcelik, Cemil, et al.. (2018). Implementation of a near-zero fluoroscopy approach in interventional electrophysiology: impact of operator experience. Journal of Interventional Cardiac Electrophysiology. 51(3). 215–220. 6 indexed citations
3.
Streckfuß‐Bömeke, Katrin, Malte Tiburcy, Xiaojing Luo, et al.. (2017). Severe DCM phenotype of patient harboring RBM20 mutation S635A can be modeled by patient-specific induced pluripotent stem cell-derived cardiomyocytes. Journal of Molecular and Cellular Cardiology. 113. 9–21. 63 indexed citations
4.
Buttgereit, Jens, Julia Shanks, Dan Li, et al.. (2016). C-type natriuretic peptide and natriuretic peptide receptor B signalling inhibits cardiac sympathetic neurotransmission and autonomic function. Cardiovascular Research. 112(3). 637–644. 31 indexed citations
5.
Mühlstedt, Silke, Santhosh Kumar Ghadge, Johan Duchêne, et al.. (2016). Cardiomyocyte-derived CXCL12 is not involved in cardiogenesis but plays a crucial role in myocardial infarction. Journal of Molecular Medicine. 94(9). 1005–1014. 16 indexed citations
6.
Wutzler, Alexander, Andreas Perrot, Martin Huemer, et al.. (2013). Variations in the human soluble epoxide hydrolase gene and recurrence of atrial fibrillation after catheter ablation. International Journal of Cardiology. 168(4). 3647–3651. 20 indexed citations
7.
Nyström, Alexander, Jens Buttgereit, Michael Bäder, et al.. (2013). Rat Model for Dominant Dystrophic Epidermolysis Bullosa: Glycine Substitution Reduces Collagen VII Stability and Shows Gene-Dosage Effect. PLoS ONE. 8(5). e64243–e64243. 14 indexed citations
8.
Morris, Daniel A., et al.. (2012). Myocardial systolic and diastolic consequences of left ventricular mechanical dyssynchrony in heart failure with normal left ventricular ejection fraction. European Heart Journal - Cardiovascular Imaging. 13(7). 556–567. 31 indexed citations
9.
Mühlstedt, Silke, Fatimunnisa Qadri, Herbert Schulz, Cemil Özcelik, & Michael Bäder. (2012). 770 A SCREENING FOR NOVEL FACTORS IN THE PATHOPHYSIOLOGY OF MYOCARDIAL INFARCTION REVEALS OSTEOACTIVIN/GPNMB. Journal of Hypertension. 30(Supplement 1). e222–e222. 1 indexed citations
10.
Montag, Judith, Bianca Borchert, Antonio Francino, et al.. (2011). Unequal allelic expression of wild-type and mutated β-myosin in familial hypertrophic cardiomyopathy. Basic Research in Cardiology. 106(6). 1041–1055. 44 indexed citations
11.
Fokstuen, Siv, Paola Melacini, Sabino Iliceto, et al.. (2011). Rapid detection of genetic variants in hypertrophic cardiomyopathy by custom DNA resequencing array in clinical practice. Journal of Medical Genetics. 48(8). 572–576. 34 indexed citations
12.
Morris, Daniel A., et al.. (2011). Right Ventricular Myocardial Systolic and Diastolic Dysfunction in Heart Failure with Normal Left Ventricular Ejection Fraction. Journal of the American Society of Echocardiography. 24(8). 886–897. 71 indexed citations
13.
Morris, Daniel A., et al.. (2011). Left Atrial Systolic and Diastolic Dysfunction in Heart Failure with Normal Left Ventricular Ejection Fraction. Journal of the American Society of Echocardiography. 24(6). 651–662. 124 indexed citations
14.
Ghadge, Santhosh Kumar, Silke Mühlstedt, Cemil Özcelik, & Michael Bäder. (2010). SDF-1α as a therapeutic stem cell homing factor in myocardial infarction. Pharmacology & Therapeutics. 129(1). 97–108. 176 indexed citations
15.
Panek, A., Maximilian Posch, Natália Alenina, et al.. (2009). Connective Tissue Growth Factor Overexpression in Cardiomyocytes Promotes Cardiac Hypertrophy and Protection against Pressure Overload. PLoS ONE. 4(8). e6743–e6743. 107 indexed citations
16.
Köhler, Jan, Antonio Francino, F Navarro-López, et al.. (2009). Cardiomyopathy Mutations Reveal Variable Region of Myosin Converter as Major Element of Cross-Bridge Compliance. Biophysical Journal. 97(3). 806–824. 58 indexed citations
17.
Alonso‐Montes, Cristina, María G. Castro, Julián R. Reguero, et al.. (2008). Mitochondrial Transcription Factors TFA, TFB1 and TFB2: A Search for DNA Variants/Haplotypes and the Risk of Cardiac Hypertrophy. Disease Markers. 25(3). 131–139. 9 indexed citations
18.
Perrot, Andreas, Volker Ruppert, Hartmut Schmidt, et al.. (2008). Identification of mutational hot spots in LMNA encoding lamin A/C in patients with familial dilated cardiomyopathy. Basic Research in Cardiology. 104(1). 90–99. 61 indexed citations
19.
Posch, Maximilian, Andreas Perrot, Katharina Schmitt, et al.. (2007). Mutations in GATA4, NKX2.5, CRELD1, and BMP4 are infrequently found in patients with congenital cardiac septal defects. American Journal of Medical Genetics Part A. 146A(2). 251–253. 75 indexed citations
20.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact

Rankless by CCL
2026