Camille Brochier

1.5k total citations · 1 hit paper
14 papers, 1.2k citations indexed

About

Camille Brochier is a scholar working on Molecular Biology, Oncology and Cellular and Molecular Neuroscience. According to data from OpenAlex, Camille Brochier has authored 14 papers receiving a total of 1.2k indexed citations (citations by other indexed papers that have themselves been cited), including 11 papers in Molecular Biology, 5 papers in Oncology and 4 papers in Cellular and Molecular Neuroscience. Recurrent topics in Camille Brochier's work include Histone Deacetylase Inhibitors Research (5 papers), Signaling Pathways in Disease (2 papers) and Mitochondrial Function and Pathology (2 papers). Camille Brochier is often cited by papers focused on Histone Deacetylase Inhibitors Research (5 papers), Signaling Pathways in Disease (2 papers) and Mitochondrial Function and Pathology (2 papers). Camille Brochier collaborates with scholars based in United States, France and Switzerland. Camille Brochier's co-authors include Brett Langley, Alan P. Kozikowski, Jay H. Kalin, Giulio Vistoli, Kyle V. Butler, Mark A. Rivieccio, Rajiv R. Ratan, Dianna E. Willis, Ambreena Siddiq and Samie R. Jaffrey and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of the American Chemical Society and Journal of Neuroscience.

In The Last Decade

Camille Brochier

13 papers receiving 1.2k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Rational Design and Simple Chemistry Yield a Superior, Neuroprotective HDAC6 Inhibitor, Tubastatin A

2010 620 citations Kyle V. Butler, Jay H. Kalin et al. Journal of the American Chemical Society profile →

Peers

Camille Brochier

ONCOL

CMN

PHARM

Gelin Wang China

A. Joseph Bloom United States

Bruna Barneda‐Zahonero Spain

Eileen J. Kennedy United States

Peter Heining Switzerland

Attila Lehotzky Hungary

Yuriko Tachida Japan

Elizabeth Kida United States

Lynn Bedford United Kingdom

Juan Sironi United States

Gelin Wang China

Camille Brochier

900 ×0.9

150 ×0.4

ONCOL

76 ×0.4

CMN

120 ×0.9

49 ×0.5

PHARM

Citations per year, relative to Camille Brochier Camille Brochier (= 1×) peers Gelin Wang

Countries citing papers authored by Camille Brochier

Since Specialization

Citations

This map shows the geographic impact of Camille Brochier's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Camille Brochier with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Camille Brochier more than expected).

Fields of papers citing papers by Camille Brochier

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Camille Brochier. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Camille Brochier. The network helps show where Camille Brochier may publish in the future.

Co-authorship network of co-authors of Camille Brochier

This figure shows the co-authorship network connecting the top 25 collaborators of Camille Brochier. A scholar is included among the top collaborators of Camille Brochier based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Camille Brochier. Camille Brochier is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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14 of 14 papers shown

Clavel, Marie‐Annick, Ivan Peyron, Tovo David, et al.. (2025). <i>Erratum</i> to: “Consistent clinical factor VIII equivalency is unlikely for non-factor therapies in hemophilic mice”. Haematologica. 110(12). 3182–3183.

Clavel, Marie‐Annick, Ivan Peyron, Tovo David, et al.. (2025). Consistent clinical factor VIII equivalency is unlikely for non-factor therapies in hemophilic mice. Haematologica. 110(9). 2064–2075. 2 indexed citations

Maciel, Thiago Trovati, Slimane Allali, Camille Brochier, et al.. (2024). Inhibition of the Complement Alternative Pathway Attenuates Hemolysis and Preserves Renal Function in a Mouse Model of Sickle Cell Disease. Blood. 144(Supplement 1). 2486–2486. 1 indexed citations

Karakachoff, Matilde, Romain Oger, J. Podevin, et al.. (2021). The Caspase-1/IL-18 Axis of the Inflammasome in Tumor Cells: A Modulator of the Th1/Tc1 Response of Tumor-Infiltrating T Lymphocytes in Colorectal Cancer. Cancers. 13(2). 189–189. 12 indexed citations

Brochier, Camille, et al.. (2015). Poly(ADP-ribose) polymerase 1 is a novel target to promote axonal regeneration. Proceedings of the National Academy of Sciences. 112(49). 15220–15225. 29 indexed citations

Brochier, Camille, et al.. (2015). Thiol-Based Potent and Selective HDAC6 Inhibitors Promote Tubulin Acetylation and T-Regulatory Cell Suppressive Function. ACS Medicinal Chemistry Letters. 6(11). 1156–1161. 35 indexed citations

Aleyasin, Hossein, Saravanan S. Karuppagounder, Amit Kumar, et al.. (2014). Antihelminthic Benzimidazoles Are Novel HIF Activators That Prevent Oxidative Neuronal Death via Binding to Tubulin. Antioxidants and Redox Signaling. 22(2). 121–134. 17 indexed citations

Brochier, Camille, et al.. (2013). Specific Acetylation of p53 by HDAC Inhibition Prevents DNA Damage-Induced Apoptosis in Neurons. Journal of Neuroscience. 33(20). 8621–8632. 78 indexed citations

Brochier, Camille & Brett Langley. (2013). Chromatin Modifications Associated with DNA Double-strand Breaks Repair as Potential Targets for Neurological Diseases. Neurotherapeutics. 10(4). 817–830. 25 indexed citations

10.

Butler, Kyle V., Jay H. Kalin, Camille Brochier, et al.. (2010). Rational Design and Simple Chemistry Yield a Superior, Neuroprotective HDAC6 Inhibitor, Tubastatin A. Journal of the American Chemical Society. 132(31). 10842–10846. 620 indexed citations breakdown →

11.

Rivieccio, Mark A., Camille Brochier, Dianna E. Willis, et al.. (2009). HDAC6 is a target for protection and regeneration following injury in the nervous system. Proceedings of the National Academy of Sciences. 106(46). 19599–19604. 251 indexed citations

12.

Langley, Brett, Camille Brochier, & Mark A. Rivieccio. (2009). Targeting Histone Deacetylases as a Multifaceted Approach to Treat the Diverse Outcomes of Stroke. Stroke. 40(8). 2899–2905. 100 indexed citations

13.

Brochier, Camille, Marie‐Claude Gaillard, Elsa Diguet, et al.. (2008). Quantitative gene expression profiling of mouse brain regions reveals differential transcripts conserved in human and affected in disease models. Physiological Genomics. 33(2). 170–179. 35 indexed citations

14.

Chaldée, Michel de, et al.. (2005). Capucin: A novel striatal marker down-regulated in rodent models of Huntington disease. Genomics. 87(2). 200–207. 15 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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