C. E. Lumsden

1.3k citations
21 papers · 821 indexed · h-index 14

Impact in

Papers in

    • Neuroinflammation and Neurodegeneration Mechanisms 2
    • Neurological Disorders and Treatments 2
    • Neurogenesis and neuroplasticity mechanisms 3

C. E. Lumsden

21 papers receiving 709 citations

Peers

C. E. Lumsden
Comparison fields: 5 of 88
  • Developmental Neuroscience 75
  • Pathology and Forensic Medicine 267
  • Neurology 103
  • Neurology 111
  • Cellular and Molecular Neuroscience 108
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Citations per field
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Citations per year

Countries citing papers authored by C. E. Lumsden

Since Specialization
Citations

This map shows the geographic impact of C. E. Lumsden's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by C. E. Lumsden with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites C. E. Lumsden more than expected).

Fields of papers citing papers by C. E. Lumsden

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by C. E. Lumsden. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by C. E. Lumsden. The network helps show where C. E. Lumsden may publish in the future.

Co-authorship network

The 12 scholars most cited alongside C. E. Lumsden, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with C. E. Lumsden Line = papers co-authored together C. E. Lumsden links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown
#Work
1 197913
2 197517
3 197513
4
Binding of normal human IgG to myelin sheaths, glia and neurons.
197593
5 19718
6 1971143
7 197064
8 1969118
9 196819
10
Fractionation of encephalitogenic polypeptides from bovine spinal cord by gel filtration in phenol--acetic acid--water.
196714
11 196617
12 196651
13 196224
14 19619
15 19583
16 19567
17 195213
18 195125
19 195185
20 195159

About C. E. Lumsden

C. E. Lumsden is a scholar working on Neurology, Developmental Neuroscience, Pathology and Forensic Medicine, Sensory Systems and Biophysics, having authored 21 papers that have together received 821 indexed citations. Recurring topics across this work include Glycosylation and Glycoproteins Research (4 papers), Neurogenesis and neuroplasticity mechanisms (3 papers), Prion Diseases and Protein Misfolding (2 papers), Multiple Sclerosis Research Studies (2 papers), Neuroinflammation and Neurodegeneration Mechanisms (2 papers), Biochemical effects in animals (2 papers), Neurological Disorders and Treatments (2 papers) and Tumors and Oncological Cases (2 papers). The work is most often cited by research in Developmental Neuroscience (75 citations), Pathology and Forensic Medicine (267 citations), Neurology (103 citations), Neurology (111 citations) and Cellular and Molecular Neuroscience (108 citations). C. E. Lumsden has collaborated with scholars based in United Kingdom, United States and Australia. Frequent co-authors include S. R. Aparicio, C. M. Pomerat, D. M. Robertson, Johan A. Aarli, O. Tönder, P. R. Carnegie, J. P. Dickinson, Laurence M. Howard, James S. Scott and Samuel Aparício. Their work appears in journals such as Brain Research, Nature, The Journal of Pathology, Journal of Neurochemistry and The Anatomical Record.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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