Bruce X. Wong

1.6k total citations · 1 hit paper
19 papers, 1.4k citations indexed

About

Bruce X. Wong is a scholar working on Physiology, Nutrition and Dietetics and Molecular Biology. According to data from OpenAlex, Bruce X. Wong has authored 19 papers receiving a total of 1.4k indexed citations (citations by other indexed papers that have themselves been cited), including 12 papers in Physiology, 9 papers in Nutrition and Dietetics and 5 papers in Molecular Biology. Recurrent topics in Bruce X. Wong's work include Alzheimer's disease research and treatments (12 papers), Trace Elements in Health (8 papers) and Parkinson's Disease Mechanisms and Treatments (4 papers). Bruce X. Wong is often cited by papers focused on Alzheimer's disease research and treatments (12 papers), Trace Elements in Health (8 papers) and Parkinson's Disease Mechanisms and Treatments (4 papers). Bruce X. Wong collaborates with scholars based in Australia, United Kingdom and France. Bruce X. Wong's co-authors include James A. Duce, Ashley I. Bush, Paul A. Adlard, Scott Ayton, Peng Lei, David I. Finkelstein, Andrew Tsatsanis, Blaine R. Roberts, Robert A. Cherny and Linh Q. Lam and has published in prestigious journals such as Journal of Biological Chemistry, Nature Medicine and PLoS ONE.

In The Last Decade

Bruce X. Wong

19 papers receiving 1.3k citations

Hit Papers

Tau deficiency induces parkinsonism with dementia by impa... 2012 2026 2016 2021 2012 100 200 300 400

Peers

Bruce X. Wong
Bruce X. Wong
Citations per year, relative to Bruce X. Wong Bruce X. Wong (= 1×) peers Pamela J. Urrutia

Countries citing papers authored by Bruce X. Wong

Since Specialization
Citations

This map shows the geographic impact of Bruce X. Wong's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Bruce X. Wong with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Bruce X. Wong more than expected).

Fields of papers citing papers by Bruce X. Wong

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Bruce X. Wong. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Bruce X. Wong. The network helps show where Bruce X. Wong may publish in the future.

Co-authorship network of co-authors of Bruce X. Wong

This figure shows the co-authorship network connecting the top 25 collaborators of Bruce X. Wong. A scholar is included among the top collaborators of Bruce X. Wong based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Bruce X. Wong. Bruce X. Wong is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

19 of 19 papers shown
1.
Jobling, Andrew I., et al.. (2024). Pharmacological inhibition of STING reduces neuroinflammation‐mediated damage post‐traumatic brain injury. British Journal of Pharmacology. 181(17). 3118–3135. 8 indexed citations
2.
Tsatsanis, Andrew, Bruce X. Wong, Ellis Patrick, et al.. (2021). The acute phase protein lactoferrin is a key feature of Alzheimer’s disease and predictor of Aβ burden through induction of APP amyloidogenic processing. Molecular Psychiatry. 26(10). 5516–5531. 36 indexed citations
3.
Gunn, Adam P., Bruce X. Wong, Catriona McLean, et al.. (2020). Increased glutaminyl cyclase activity in brains of Alzheimer’s disease individuals. Journal of Neurochemistry. 156(6). 979–987. 17 indexed citations
4.
Tsatsanis, Andrew, Bruce X. Wong, Adam P. Gunn, et al.. (2020). Amyloidogenic processing of Alzheimer’s disease β-amyloid precursor protein induces cellular iron retention. Molecular Psychiatry. 25(9). 1958–1966. 65 indexed citations
5.
Tsatsanis, Andrew, et al.. (2019). Post Translational Modulation of β-Amyloid Precursor Protein Trafficking to the Cell Surface Alters Neuronal Iron Homeostasis. Neurochemical Research. 44(6). 1367–1374. 36 indexed citations
6.
Belaidi, Abdel Ali, Adam P. Gunn, Bruce X. Wong, et al.. (2018). Marked Age-Related Changes in Brain Iron Homeostasis in Amyloid Protein Precursor Knockout Mice. Neurotherapeutics. 15(4). 1055–1062. 60 indexed citations
7.
Sanchez, M. Isabel G. Lopez, Hayley S. Waugh, Andrew Tsatsanis, et al.. (2017). Amyloid precursor protein drives down-regulation of mitochondrial oxidative phosphorylation independent of amyloid beta. Scientific Reports. 7(1). 9835–9835. 34 indexed citations
8.
Duce, James A., et al.. (2017). Post translational changes to α-synuclein control iron and dopamine trafficking; a concept for neuron vulnerability in Parkinson’s disease. Molecular Neurodegeneration. 12(1). 45–45. 64 indexed citations
9.
Gunn, Adam P., Bruce X. Wong, Timothy Johanssen, et al.. (2016). Amyloid-β Peptide Aβ3pE-42 Induces Lipid Peroxidation, Membrane Permeabilization, and Calcium Influx in Neurons. Journal of Biological Chemistry. 291(12). 6134–6145. 73 indexed citations
10.
Lam, Linh Q., Bruce X. Wong, Tony Frugier, et al.. (2016). Oxidation of Iron under Physiologically Relevant Conditions in Biological Fluids from Healthy and Alzheimer’s Disease Subjects. ACS Chemical Neuroscience. 8(4). 731–736. 3 indexed citations
11.
Wong, Bruce X. & James A. Duce. (2014). The iron regulatory capability of the major protein participants in prevalent neurodegenerative disorders. Frontiers in Pharmacology. 5. 81–81. 38 indexed citations
12.
Wong, Bruce X., Ya Hui Hung, Ashley I. Bush, & James A. Duce. (2014). Metals and cholesterol: two sides of the same coin in Alzheimer’s disease pathology. Frontiers in Aging Neuroscience. 6. 91–91. 34 indexed citations
13.
Wong, Bruce X., Scott Ayton, Linh Q. Lam, et al.. (2014). A comparison of ceruloplasmin to biological polyanions in promoting the oxidation of Fe2+ under physiologically relevant conditions. Biochimica et Biophysica Acta (BBA) - General Subjects. 1840(12). 3299–3310. 26 indexed citations
14.
Wong, Bruce X., et al.. (2014). β-Amyloid Precursor Protein Does Not Possess Ferroxidase Activity but Does Stabilize the Cell Surface Ferrous Iron Exporter Ferroportin. PLoS ONE. 9(12). e114174–e114174. 128 indexed citations
15.
Gu, Ben J., James A. Duce, Valentina A. Valova, et al.. (2012). P2X7 Receptor-mediated Scavenger Activity of Mononuclear Phagocytes toward Non-opsonized Particles and Apoptotic Cells Is Inhibited by Serum Glycoproteins but Remains Active in Cerebrospinal Fluid. Journal of Biological Chemistry. 287(21). 17318–17330. 26 indexed citations
16.
Lei, Peng, Scott Ayton, David I. Finkelstein, et al.. (2012). Tau deficiency induces parkinsonism with dementia by impairing APP-mediated iron export. Nature Medicine. 18(2). 291–295. 472 indexed citations breakdown →
17.
Ayton, Scott, Peng Lei, James A. Duce, et al.. (2012). Ceruloplasmin dysfunction and therapeutic potential for Parkinson disease. Annals of Neurology. 73(4). 554–559. 215 indexed citations
18.
Wong, Bruce X., et al.. (2011). Modulation of Macrophage Fatty Acid Content and Composition by Exposure to Dyslipidemic Serum in Vitro. Lipids. 46(4). 371–380. 11 indexed citations
19.
Wong, Bruce X., et al.. (2011). Dyslipidemic Diabetic Serum Increases Lipid Accumulation and Expression of Stearoyl‐CoA Desaturase in Human Macrophages. Lipids. 46(10). 931–941. 7 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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