Jamie Soto

2.9k total citations · 1 hit paper
27 papers, 1.8k citations indexed

About

Jamie Soto is a scholar working on Molecular Biology, Physiology and Cardiology and Cardiovascular Medicine. According to data from OpenAlex, Jamie Soto has authored 27 papers receiving a total of 1.8k indexed citations (citations by other indexed papers that have themselves been cited), including 19 papers in Molecular Biology, 11 papers in Physiology and 4 papers in Cardiology and Cardiovascular Medicine. Recurrent topics in Jamie Soto's work include Mitochondrial Function and Pathology (9 papers), Adipose Tissue and Metabolism (9 papers) and ATP Synthase and ATPases Research (6 papers). Jamie Soto is often cited by papers focused on Mitochondrial Function and Pathology (9 papers), Adipose Tissue and Metabolism (9 papers) and ATP Synthase and ATPases Research (6 papers). Jamie Soto collaborates with scholars based in United States, France and Switzerland. Jamie Soto's co-authors include E. Dale Abel, Christian Riehle, Adam R. Wende, Heather Theobald, Heiko Bugger, Edward Zúñiga, Jesse D. Sengillo, John S. Sullivan, Rosalinda B. Wenby and David D. Perlmutter and has published in prestigious journals such as Nature Neuroscience, PLoS ONE and Molecular and Cellular Biology.

In The Last Decade

Jamie Soto

26 papers receiving 1.8k citations

Hit Papers

GLUT1 reductions exacerbate Alzheimer's disease vasculo-n... 2015 2026 2018 2022 2015 100 200 300 400 500
What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

  1. GLUT1 reductions exacerbate Alzheimer's disease vasculo-neuronal dysfunction and degeneration
    2015 516 citations Ethan A. Winkler, Yoichiro Nishida et al. Nature Neuroscience profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Jamie Soto United States 18 856 634 369 247 238 27 1.8k
Angelo Maffei Italy 26 764 0.9× 592 0.9× 682 1.8× 177 0.7× 273 1.1× 36 2.1k
Nathalie Thorin‐Trescases Canada 29 663 0.8× 601 0.9× 867 2.3× 220 0.9× 217 0.9× 79 2.2k
Roberta Poulet Italy 15 559 0.7× 446 0.7× 437 1.2× 119 0.5× 194 0.8× 17 1.4k
Lora C. Bailey‐Downs United States 14 566 0.7× 371 0.6× 167 0.5× 188 0.8× 145 0.6× 26 1.4k
Xunde Xian China 22 539 0.6× 487 0.8× 209 0.6× 158 0.6× 177 0.7× 60 1.5k
Eitaro Nakashima Japan 24 642 0.8× 523 0.8× 138 0.4× 127 0.5× 216 0.9× 49 1.9k
Xiangdong Xu China 17 843 1.0× 868 1.4× 98 0.3× 255 1.0× 153 0.6× 32 2.2k
Marta Cortés‐Canteli Spain 20 730 0.9× 714 1.1× 138 0.4× 505 2.0× 210 0.9× 33 2.0k
Nobutaka Koibuchi Japan 31 957 1.1× 365 0.6× 695 1.9× 323 1.3× 165 0.7× 58 2.5k
Ying Ann Chiao United States 24 1.2k 1.4× 569 0.9× 707 1.9× 61 0.2× 258 1.1× 49 2.5k

Countries citing papers authored by Jamie Soto

Since Specialization
Citations

This map shows the geographic impact of Jamie Soto's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jamie Soto with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jamie Soto more than expected).

Fields of papers citing papers by Jamie Soto

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jamie Soto. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jamie Soto. The network helps show where Jamie Soto may publish in the future.

Co-authorship network of co-authors of Jamie Soto

This figure shows the co-authorship network connecting the top 25 collaborators of Jamie Soto. A scholar is included among the top collaborators of Jamie Soto based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jamie Soto. Jamie Soto is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Sun, Xutong, Manivannan Yegambaram, Qing Lü, et al.. (2025). Mitochondrial fission produces a Warburg effect via the oxidative inhibition of prolyl hydroxylase domain-2. Redox Biology. 81. 103529–103529. 4 indexed citations
2.
Yegambaram, Manivannan, Qing Lü, Jamie Soto, et al.. (2025). Restoration of pp60Src Re-Establishes Electron Transport Chain Complex I Activity in Pulmonary Hypertensive Endothelial Cells. International Journal of Molecular Sciences. 26(8). 3815–3815. 1 indexed citations
3.
Datar, Sanjeev A., Xutong Sun, Wenhui Gong, et al.. (2025). Mechanotransductive stabilization of HIF-1α is inhibited by mitochondrial antioxidant therapy in the setting of pulmonary overcirculation. Scientific Reports. 15(1). 16320–16320. 1 indexed citations
4.
Yegambaram, Manivannan, Qing Lü, Alejandro Garcia Flores, et al.. (2025). c-Myc promotes metabolic reprogramming in pulmonary hypertension via the stimulation of glutaminolysis and the reductive tricarboxylic acid cycle. Redox Biology. 85. 103765–103765.
5.
Yegambaram, Manivannan, Xutong Sun, Qing Lü, et al.. (2023). Mitochondrial hyperfusion induces metabolic remodeling in lung endothelial cells by modifying the activities of electron transport chain complexes I and III. Free Radical Biology and Medicine. 210. 183–194. 11 indexed citations
6.
Yegambaram, Manivannan, Xutong Sun, Alejandro Garcia Flores, et al.. (2023). Novel Relationship between Mitofusin 2-Mediated Mitochondrial Hyperfusion, Metabolic Remodeling, and Glycolysis in Pulmonary Arterial Endothelial Cells. International Journal of Molecular Sciences. 24(24). 17533–17533. 7 indexed citations
7.
Swarup, Aditi, Ivy S. Samuels, Jamie Soto, et al.. (2018). Modulation of GLUT1 expression in the RPE impacts outer segment renewal and results in photoreceptor degeneration. Investigative Ophthalmology & Visual Science. 59(9). 4967–4967. 1 indexed citations
8.
Park, Sung Jun, Oksana Gavrilova, Alexandra L. Brown, et al.. (2017). DNA-PK Promotes the Mitochondrial, Metabolic, and Physical Decline that Occurs During Aging. Cell Metabolism. 25(5). 1135–1146.e7. 93 indexed citations
9.
Ilkun, Olesya, Nicole Wilde, Joseph Tuinei, et al.. (2015). Antioxidant treatment normalizes mitochondrial energetics and myocardial insulin sensitivity independently of changes in systemic metabolic homeostasis in a mouse model of the metabolic syndrome. Journal of Molecular and Cellular Cardiology. 85. 104–116. 26 indexed citations
10.
Winkler, Ethan A., Yoichiro Nishida, Abhay P. Sagare, et al.. (2015). GLUT1 reductions exacerbate Alzheimer's disease vasculo-neuronal dysfunction and degeneration. Nature Neuroscience. 18(4). 521–530. 516 indexed citations breakdown →
11.
Jaishy, Bharat P., Quan‐Jiang Zhang, Heaseung Sophia Chung, et al.. (2014). Lipid-induced NOX2 activation inhibits autophagic flux by impairing lysosomal enzyme activity. Journal of Lipid Research. 56(3). 546–561. 104 indexed citations
12.
Pereira, Renata O., Adam R. Wende, Curtis D. Olsen, et al.. (2014). GLUT1 deficiency in cardiomyocytes does not accelerate the transition from compensated hypertrophy to heart failure. Journal of Molecular and Cellular Cardiology. 72. 95–103. 40 indexed citations
13.
14.
Pereira, Renata O., Adam R. Wende, Curtis D. Olsen, et al.. (2013). Inducible Overexpression of GLUT1 Prevents Mitochondrial Dysfunction and Attenuates Structural Remodeling in Pressure Overload but Does Not Prevent Left Ventricular Dysfunction. Journal of the American Heart Association. 2(5). e000301–e000301. 79 indexed citations
15.
Zhu, Yi, Jamie Soto, Christian Riehle, et al.. (2013). Regulation of fatty acid metabolism by mTOR in adult murine hearts occurs independently of changes in PGC-1α. American Journal of Physiology-Heart and Circulatory Physiology. 305(1). H41–H51. 33 indexed citations
16.
Bugger, Heiko, Christian Riehle, Bharat P. Jaishy, et al.. (2012). Genetic loss of insulin receptors worsens cardiac efficiency in diabetes. Journal of Molecular and Cellular Cardiology. 52(5). 1019–1026. 59 indexed citations
17.
Sloan, Crystal, Joseph Tuinei, Jonathan Frandsen, et al.. (2011). Central Leptin Signaling Is Required to Normalize Myocardial Fatty Acid Oxidation Rates in Caloric-Restricted ob/ob Mice. Diabetes. 60(5). 1424–1434. 75 indexed citations
18.
Liu, Siming, Terumasa Okada, Anke Aßmann, et al.. (2009). Insulin Signaling Regulates Mitochondrial Function in Pancreatic β-Cells. PLoS ONE. 4(11). e7983–e7983. 50 indexed citations
19.
Amiott, Elizabeth, Paul C. Lott, Jamie Soto, et al.. (2008). Mitochondrial fusion and function in Charcot–Marie–Tooth type 2A patient fibroblasts with mitofusin 2 mutations. Experimental Neurology. 211(1). 115–127. 84 indexed citations
20.
Kim, Jaetaek, Adam R. Wende, Sandra Sena, et al.. (2008). Insulin-Like Growth Factor I Receptor Signaling Is Required for Exercise-Induced Cardiac Hypertrophy. Molecular Endocrinology. 22(11). 2531–2543. 172 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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