Robert Maile

3.0k total citations
69 papers, 1.5k citations indexed

About

Robert Maile is a scholar working on Immunology, Molecular Biology and Epidemiology. According to data from OpenAlex, Robert Maile has authored 69 papers receiving a total of 1.5k indexed citations (citations by other indexed papers that have themselves been cited), including 30 papers in Immunology, 24 papers in Molecular Biology and 23 papers in Epidemiology. Recurrent topics in Robert Maile's work include Burn Injury Management and Outcomes (18 papers), Immune Response and Inflammation (14 papers) and T-cell and B-cell Immunology (11 papers). Robert Maile is often cited by papers focused on Burn Injury Management and Outcomes (18 papers), Immune Response and Inflammation (14 papers) and T-cell and B-cell Immunology (11 papers). Robert Maile collaborates with scholars based in United States, France and United Kingdom. Robert Maile's co-authors include Jeffrey A. Frelinger, Bruce A. Cairns, Edward J. Collins, Anthony A. Meyer, Laurel B. Kartchner, Shaomin Tian, Lance R. Thurlow, Anthony R. Richardson, Gauri S. Joshi and Matthew C. Wolfgang and has published in prestigious journals such as Nature Communications, The Journal of Immunology and PLoS ONE.

In The Last Decade

Robert Maile

64 papers receiving 1.5k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Robert Maile United States 22 637 538 355 197 164 69 1.5k
Lena Yao United States 16 547 0.9× 356 0.7× 229 0.6× 150 0.8× 249 1.5× 26 1.8k
William F. Carson United States 23 1.2k 1.8× 708 1.3× 345 1.0× 91 0.5× 183 1.1× 34 2.1k
Ajitha Thanabalasuriar Canada 17 1.0k 1.6× 628 1.2× 200 0.6× 244 1.2× 81 0.5× 24 1.9k
Charles T. Spencer United States 15 827 1.3× 370 0.7× 385 1.1× 350 1.8× 127 0.8× 34 1.7k
Anne Krüger Germany 19 397 0.6× 333 0.6× 164 0.5× 102 0.5× 69 0.4× 44 1.3k
Hisanori Domon Japan 27 626 1.0× 440 0.8× 347 1.0× 88 0.4× 77 0.5× 77 2.0k
Loreto Abusleme United States 22 800 1.3× 819 1.5× 210 0.6× 172 0.9× 115 0.7× 32 3.5k
Jennifer Brazil United States 22 695 1.1× 678 1.3× 173 0.5× 108 0.5× 159 1.0× 44 1.8k
Christof Wagner Germany 25 646 1.0× 655 1.2× 121 0.3× 162 0.8× 102 0.6× 41 1.6k
Sinéad M. Smith Ireland 26 679 1.1× 604 1.1× 350 1.0× 293 1.5× 238 1.5× 60 2.3k

Countries citing papers authored by Robert Maile

Since Specialization
Citations

This map shows the geographic impact of Robert Maile's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Robert Maile with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Robert Maile more than expected).

Fields of papers citing papers by Robert Maile

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Robert Maile. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Robert Maile. The network helps show where Robert Maile may publish in the future.

Co-authorship network of co-authors of Robert Maile

This figure shows the co-authorship network connecting the top 25 collaborators of Robert Maile. A scholar is included among the top collaborators of Robert Maile based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Robert Maile. Robert Maile is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Kim, Han Sang, Jaimar C. Rincon, Philip A. Efron, & Robert Maile. (2025). DAMP-driven trained immunity: metabolic and epigenetic reprogramming in critical illness and chronic inflammation. Frontiers in Immunology. 16. 1669054–1669054.
2.
Herring, Laura E., Gilbert R. Upchurch, Ashish K. Sharma, et al.. (2025). Temporal changes in the protein cargo of extracellular vesicles and resultant immune reprogramming after severe burn injury in humans and mice. Frontiers in Immunology. 16. 1596598–1596598. 1 indexed citations
3.
Wallet, Shannon M., et al.. (2024). Current updates in precision and personalized medicine in sepsis and trauma. Surgery. 176(2). 541–543.
4.
Bellotti, Paolo, Gang Su, Robert Maile, et al.. (2024). Resolvin D2 / GPR18 signaling enhances monocytic myeloid‐derived suppressor cell function to mitigate abdominal aortic aneurysm formation. The FASEB Journal. 38(18). e70067–e70067. 3 indexed citations
5.
Park, Gwoncheol, Lauren S. Kelly, Kolenkode B. Kannan, et al.. (2024). Persistence and Sexual Dimorphism of Gut Dysbiosis and Pathobiome after Sepsis and Trauma. Annals of Surgery. 280(3). 491–503. 3 indexed citations
6.
7.
Duin, David van, Paula D. Strassle, Lauren DiBiase, et al.. (2016). Timeline of health care–associated infections and pathogens after burn injuries. American Journal of Infection Control. 44(12). 1511–1516. 76 indexed citations
8.
Kartchner, Laurel B., April E. Mendoza, Jeffrey A. Frelinger, et al.. (2014). Flagellin Treatment Prevents Increased Susceptibility to Systemic Bacterial Infection after Injury by Inhibiting Anti-Inflammatory IL-10+ IL-12- Neutrophil Polarization. PLoS ONE. 9(1). e85623–e85623. 55 indexed citations
9.
Jones, Samuel, Haibo Zhou, Shiara Ortiz‐Pujols, et al.. (2013). Bronchoscopy-Derived Correlates of Lung Injury following Inhalational Injuries: A Prospective Observational Study. PLoS ONE. 8(5). e64250–e64250. 30 indexed citations
10.
Mendoza, April E., Anthony Charles, Laurel B. Kartchner, et al.. (2012). Radiation Combined With Thermal Injury Induces Immature Myeloid Cells. Shock. 38(5). 532–542. 18 indexed citations
11.
Tian, Shaomin, Robert Maile, Edward J. Collins, & Jeffrey A. Frelinger. (2007). CD8+ T Cell Activation Is Governed by TCR-Peptide/MHC Affinity, Not Dissociation Rate. The Journal of Immunology. 179(5). 2952–2960. 99 indexed citations
12.
Maile, Robert, et al.. (2006). Lymphopenia-Induced Homeostatic Proliferation of CD8+ T Cells Is a Mechanism for Effective Allogeneic Skin Graft Rejection following Burn Injury. The Journal of Immunology. 176(11). 6717–6726. 21 indexed citations
13.
Wardrop, Richard M., Ting Gui, Robert Maile, et al.. (2006). Transgene expression levels and kinetics determine risk of humoral immune response modeled in factor IX knockout and missense mutant mice. Gene Therapy. 14(5). 429–440. 34 indexed citations
14.
Maile, Robert, Lucinda Hensley, Paula Kavathas, et al.. (2003). Interplay between TCR Affinity and Necessity of Coreceptor Ligation: High-Affinity Peptide-MHC/TCR Interaction Overcomes Lack of CD8 Engagement. The Journal of Immunology. 171(9). 4493–4503. 73 indexed citations
15.
Wang, Bo, et al.. (2002). Peptidic Termini Play a Significant Role in TCR Recognition. The Journal of Immunology. 169(6). 3137–3145. 18 indexed citations
16.
Maile, Robert, Bo Wang, Wesley Schooler, et al.. (2001). Antigen-Specific Modulation of an Immune Response by In Vivo Administration of Soluble MHC Class I Tetramers. The Journal of Immunology. 167(7). 3708–3714. 67 indexed citations
17.
Cairns, Bruce A., Robert Maile, Ian Buchanan, et al.. (2001). CD8+ T cells express a T-helper 1–like phenotype after burn injury. Surgery. 130(2). 210–216. 12 indexed citations
18.
Wang, Bo, et al.. (2000). Naive CD8+ T Cells Do Not Require Costimulation for Proliferation and Differentiation into Cytotoxic Effector Cells. The Journal of Immunology. 164(3). 1216–1222. 95 indexed citations
19.
20.
Maile, Robert, et al.. (1998). Effective formation by thyroid epithelial cells of MHC Class II-peptide complexes derived from endogenous antigen. Immunology. 95. 2 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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