Brian Malester

3.0k total citations · 1 hit paper
13 papers, 2.4k citations indexed

About

Brian Malester is a scholar working on Molecular Biology, Physiology and Pathology and Forensic Medicine. According to data from OpenAlex, Brian Malester has authored 13 papers receiving a total of 2.4k indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Molecular Biology, 6 papers in Physiology and 5 papers in Pathology and Forensic Medicine. Recurrent topics in Brian Malester's work include Alzheimer's disease research and treatments (6 papers), Ion channel regulation and function (5 papers) and Cardiac Ischemia and Reperfusion (5 papers). Brian Malester is often cited by papers focused on Alzheimer's disease research and treatments (6 papers), Ion channel regulation and function (5 papers) and Cardiac Ischemia and Reperfusion (5 papers). Brian Malester collaborates with scholars based in United States, Switzerland and Netherlands. Brian Malester's co-authors include Karen Duff, John J. LaFrancois, Lorenzo M. Refolo, Miguel A. Pappolla, G S Tint, Tara Bryant‐Thomas, Kumar Sambamurti, Rong Wang, William A. Coetzee and Stephen D. Schmidt and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and The Journal of Physiology.

In The Last Decade

Brian Malester

13 papers receiving 2.4k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Hypercholesterolemia Accelerates the Alzheimer's Amyloid Pathology in a Transgenic Mouse Model

2000 825 citations Lorenzo M. Refolo, Miguel A. Pappolla et al. Neurobiology of Disease profile →

Peers

Brian Malester

PHYSI

MB

SURGE

CMN

NEURO

Lorenzo M. Refolo United States

Suzana Petanceska United States

Qingguang Jiang United States

Wesley Farris United States

Günter Tremp France

Mitsuru Shinohara Japan

Heike S. Grimm Germany

Michela Guglielmotto Italy

Elena Tamagno Italy

Lorenzo M. Refolo United States

Brian Malester

2.3k ×1.6

PHYSI

1.2k ×1.3

MB

772 ×1.2

SURGE

471 ×1.2

CMN

350 ×0.9

NEURO

Citations per year, relative to Brian Malester Brian Malester (= 1×) peers Lorenzo M. Refolo

Countries citing papers authored by Brian Malester

Since Specialization

Citations

This map shows the geographic impact of Brian Malester's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Brian Malester with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Brian Malester more than expected).

Fields of papers citing papers by Brian Malester

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Brian Malester. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Brian Malester. The network helps show where Brian Malester may publish in the future.

Co-authorship network of co-authors of Brian Malester

This figure shows the co-authorship network connecting the top 25 collaborators of Brian Malester. A scholar is included among the top collaborators of Brian Malester based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Brian Malester. Brian Malester is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

Sort: Min cites: Since: Top N: Style:

13 of 13 papers shown

1.

Li, Bao, et al.. (2011). The cardiac KATP channel associates with the glycolytic enzyme complex. The FASEB Journal. 25(S1). 4 indexed citations

2.

Kefaloyianni, Eirini, Bao Li, Brian Malester, et al.. (2011). Cardiac ATP‐sensitive K ⁺ channel associates with the glycolytic enzyme complex. The FASEB Journal. 25(7). 2456–2467. 46 indexed citations

3.

Malester, Brian, et al.. (2007). The regulation of ion channels and transporters by glycolytically derived ATP. Cellular and Molecular Life Sciences. 64(23). 3069–3083. 89 indexed citations

4.

Malester, Brian, Xiaoyong Tong, Andrianos Kontogeorgis, et al.. (2007). Transgenic expression of a dominant negative K _ATP channel subunit in the mouse endothelium: effects on coronary flow and endothelin‐1 secretion. The FASEB Journal. 21(9). 2162–2172. 56 indexed citations

5.

Kang, Guoxin, Oleg G. Chepurny, Brian Malester, et al.. (2006). cAMP sensor Epac as a determinant of ATP‐sensitive potassium channel activity in human pancreatic β cells and rat INS‐1 cells. The Journal of Physiology. 573(3). 595–609. 115 indexed citations

6.

Han, Sandra Y., Shekhar Srivastava, Weixia Liu, et al.. (2005). The Glycolytic Enzymes, Glyceraldehyde-3-phosphate Dehydrogenase, Triose-phosphate Isomerase, and Pyruvate Kinase Are Components of the KATP Channel Macromolecular Complex and Regulate Its Function. Journal of Biological Chemistry. 280(46). 38464–38470. 108 indexed citations

7.

Akassoglou, Katerina, Brian Malester, Jixiang Xu, et al.. (2004). Brain-specific deletion of neuropathy target esterase/ swisscheese results in neurodegeneration. Proceedings of the National Academy of Sciences. 101(14). 5075–5080. 139 indexed citations

8.

Matsuoka, Yasuji, Brian Malester, John J. LaFrancois, et al.. (2001). Inflammatory Responses to Amyloidosis in a Transgenic Mouse Model of Alzheimer’s Disease. American Journal Of Pathology. 158(4). 1345–1354. 251 indexed citations

9.

Refolo, Lorenzo M., Miguel A. Pappolla, John J. LaFrancois, et al.. (2001). A Cholesterol-Lowering Drug Reduces β-Amyloid Pathology in a Transgenic Mouse Model of Alzheimer's Disease. Neurobiology of Disease. 8(5). 890–899. 441 indexed citations

10.

Poduslo, Joseph F., et al.. (2001). Permeability of Proteins at the Blood–Brain Barrier in the Normal Adult Mouse and Double Transgenic Mouse Model of Alzheimer's Disease. Neurobiology of Disease. 8(4). 555–567. 97 indexed citations

11.

Refolo, Lorenzo M., Miguel A. Pappolla, Brian Malester, et al.. (2000). Hypercholesterolemia Accelerates the Alzheimer's Amyloid Pathology in a Transgenic Mouse Model. Neurobiology of Disease. 7(4). 321–331. 825 indexed citations breakdown →

12.

Duff, Karen, Heather Knight, Lorenzo M. Refolo, et al.. (2000). Characterization of Pathology in Transgenic Mice Over-Expressing Human Genomic and cDNA Tau Transgenes. Neurobiology of Disease. 7(2). 87–98. 237 indexed citations

13.

Refolo, Lorenzo M., Miguel A. Pappolla, Brian Malester, et al.. (2000). Hypercholesterolemia Accelerates the Alzheimer's Amyloid Pathology in a Transgenic Mouse Model. Neurobiology of Disease. 7(6). 690–691. 18 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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