Brenda Finucane
- Genetics top 0.5%
- Molecular Biology top 10%
- Cognitive Neuroscience top 2%
- Clinical Psychology top 10%
- Cellular and Molecular Neuroscience top 10%
- Co-authors
- Elisabeth M. DykensRobert M. HodappSarah H. ElseaRebecca E. SlagerChristopher N. VlangosElizabeth Berry‐KravisRandi J. HagermanLiane Abrams
- Topics
- Genetics and Neurodevelopmental Disorders (30 papers)Genomic variations and chromosomal abnormalities (28 papers)Autism Spectrum Disorder Research (23 papers)
- Partner nations
- United StatesCanadaBelgium
In The Last Decade
Brenda Finucane
60 papers receiving 2.5k citations
Peers
Comparison fields: 5 of 95
- Genetics 1.8k
- Molecular Biology 1.2k
- Cognitive Neuroscience 921
- Clinical Psychology 225
- Cellular and Molecular Neuroscience 206
Countries citing papers authored by Brenda Finucane
This map shows the geographic impact of Brenda Finucane's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Brenda Finucane with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Brenda Finucane more than expected).
Fields of papers citing papers by Brenda Finucane
This network shows the impact of papers produced by Brenda Finucane. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Brenda Finucane. The network helps show where Brenda Finucane may publish in the future.
Co-authorship network of co-authors of Brenda Finucane
This figure shows the co-authorship network connecting the top 25 collaborators of Brenda Finucane. A scholar is included among the top collaborators of Brenda Finucane based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Brenda Finucane. Brenda Finucane is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 2 | |
| 2 | 0 | |
| 3 | 6 | |
| 4 | 1 | |
| 5 | 2 | |
| 6 | 3 | |
| 7 | 6 | |
| 8 | 28 | |
| 9 | 15 | |
| 10 | 5 | |
| 11 | 2 | |
| 12 | 0 | |
| 13 | 56 | |
| 14 | 137 | |
| 15 | 13 | |
| 16 | 233 | |
| 17 | 17 | |
| 18 | 1 | |
| 19 | 25 | |
| 20 | 74 |
About Brenda Finucane
Brenda Finucane is a scholar working on Genetics, Cognitive Neuroscience and Developmental Neuroscience, having authored 65 papers that have together received 2.6k indexed citations. Recurring topics across this work include Genetics and Neurodevelopmental Disorders (30 papers), Genomic variations and chromosomal abnormalities (28 papers) and Autism Spectrum Disorder Research (23 papers). The work is most often cited by research in Genetics (1.8k citations), Cognitive Neuroscience (921 citations) and Developmental Neuroscience (155 citations). Brenda Finucane has collaborated with scholars based in United States, Canada and Belgium. Frequent co-authors include Elisabeth M. Dykens, Robert M. Hodapp, Sarah H. Elsea, Rebecca E. Slager, Christopher N. Vlangos, Elizabeth Berry‐Kravis, Randi J. Hagerman, Liane Abrams, Flora Tassone and Elliott W. Simon. Their work appears in journals such as Proceedings of the National Academy of Sciences, JAMA and Nature Communications.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.