Björn Kruspig
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- Cancer, Hypoxia, and Metabolism 4
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- Mitochondrial Function and Pathology 3
- Cell death mechanisms and regulation 2
- CRISPR and Genetic Engineering 2
- ATP Synthase and ATPases Research 2
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- Cancer-related Molecular Pathways 2
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- Endoplasmic Reticulum Stress and Disease 2
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- Neuroblastoma Research and Treatments 2
- Co-authors
- Vladimir GogvadzeBoris ZhivotovskyDaniel J. MurphySten OrreniusSeth B. CoffeltJulia B. CorderoYachuan YuAlessandro Scopelliti
- Journals
- Mitochondrion (2 papers)Biochimica et Biophysica Acta (BBA) - Molecular Cell Research (2 papers)Cellular and Molecular Life Sciences (2 papers)
- Partner nations
- SwedenUnited KingdomRussia
In The Last Decade
Björn Kruspig
13 papers receiving 383 citations
Peers
Comparison fields: 5 of 74
- Cancer Research 117
- Aging 10
- Molecular Biology 212
- Immunology 62
- Oncology 79
Countries citing papers authored by Björn Kruspig
This map shows the geographic impact of Björn Kruspig's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Björn Kruspig with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Björn Kruspig more than expected).
Fields of papers citing papers by Björn Kruspig
This network shows the impact of papers produced by Björn Kruspig. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Björn Kruspig. The network helps show where Björn Kruspig may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Björn Kruspig, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2020 | 2 | |
| 2 | 2019 | 4 | |
| 3 | 2018 | 70 | |
| 4 | 2018 | 72 | |
| 5 | CRISPR/Cas9-derived models of ovarian high grade serous carcinoma targeting Brca1, Pten and Nf1, and correlation with platinum sensitivity | 2017 | 2 |
| 6 | 2017 | 66 | |
| 7 | 2016 | 24 | |
| 8 | 2016 | 17 | |
| 9 | 2014 | 15 | |
| 10 | 2012 | 43 | |
| 11 | 2012 | 41 | |
| 12 | 2012 | 17 | |
| 13 | 2012 | 13 |
About Björn Kruspig
Björn Kruspig is a scholar working on Cancer Research, Reproductive Medicine, Cell Biology, Biochemistry and Oncology, having authored 13 papers that have together received 386 indexed citations. Recurring topics across this work include Cancer, Hypoxia, and Metabolism (4 papers), Mitochondrial Function and Pathology (3 papers), Cell death mechanisms and regulation (2 papers), CRISPR and Genetic Engineering (2 papers), ATP Synthase and ATPases Research (2 papers), Endoplasmic Reticulum Stress and Disease (2 papers), Neuroblastoma Research and Treatments (2 papers) and Cancer-related Molecular Pathways (2 papers). The work is most often cited by research in Cancer Research (117 citations), Aging (10 citations), Molecular Biology (212 citations), Immunology (62 citations) and Oncology (79 citations). Björn Kruspig has collaborated with scholars based in Sweden, United Kingdom and Russia. Frequent co-authors include Vladimir Gogvadze, Boris Zhivotovsky, Daniel J. Murphy, Sten Orrenius, Seth B. Coffelt, Julia B. Cordero, Yachuan Yu, Alessandro Scopelliti, Oliver D.K. Maddocks and Marcos Vidal. Their work appears in journals such as Mitochondrion, Biochimica et Biophysica Acta (BBA) - Molecular Cell Research, Cellular and Molecular Life Sciences, Cell Metabolism and Scientific Reports.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.