Behzad Moghadaszadeh

3.0k total citations · 1 hit paper
29 papers, 2.1k citations indexed

About

Behzad Moghadaszadeh is a scholar working on Molecular Biology, Cell Biology and Cardiology and Cardiovascular Medicine. According to data from OpenAlex, Behzad Moghadaszadeh has authored 29 papers receiving a total of 2.1k indexed citations (citations by other indexed papers that have themselves been cited), including 23 papers in Molecular Biology, 6 papers in Cell Biology and 4 papers in Cardiology and Cardiovascular Medicine. Recurrent topics in Behzad Moghadaszadeh's work include Muscle Physiology and Disorders (16 papers), Nuclear Structure and Function (5 papers) and Cellular transport and secretion (4 papers). Behzad Moghadaszadeh is often cited by papers focused on Muscle Physiology and Disorders (16 papers), Nuclear Structure and Function (5 papers) and Cellular transport and secretion (4 papers). Behzad Moghadaszadeh collaborates with scholars based in United States, France and United Kingdom. Behzad Moghadaszadeh's co-authors include Alan H. Beggs, Pascale Guicheney, Luciano Merlini, Francesco Muntoni, Haluk Topaloğlu, Nathalie Petit, Martin Brockington, B. Estournet, Norma B. Romero and Emily C. Troiano and has published in prestigious journals such as Cell, Proceedings of the National Academy of Sciences and Nature Genetics.

In The Last Decade

Behzad Moghadaszadeh

28 papers receiving 2.0k citations

Hit Papers

align trajectories sort by overperformance

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Directed evolution of a family of AAV capsid variants enabling potent muscle-directed gene delivery across species

2021 292 citations Mohammadsharif Tabebordbar, Kim A. Lagerborg et al. Cell profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Behzad Moghadaszadeh United States	21	1.5k	515	321	289	274	29	2.1k
Tomasa Barrientos United States	17	1.2k 0.8×	249 0.5×	223 0.7×	146 0.5×	243 0.9×	20	2.0k
Juliette Hadchouel France	29	2.3k 1.6×	226 0.4×	433 1.3×	337 1.2×	116 0.4×	55	2.7k
Reimar Abraham Italy	10	2.1k 1.4×	199 0.4×	97 0.3×	281 1.0×	200 0.7×	16	3.0k
Martin Brockington United Kingdom	34	3.7k 2.5×	761 1.5×	138 0.4×	362 1.3×	460 1.7×	55	4.1k
David Tuil France	19	1.1k 0.8×	222 0.4×	84 0.3×	192 0.7×	127 0.5×	34	1.5k
Leslie E. Stolz United States	14	1.6k 1.1×	195 0.4×	72 0.2×	352 1.2×	320 1.2×	21	2.1k
Ekaterina Salimova Australia	21	1.1k 0.8×	469 0.9×	118 0.4×	74 0.3×	56 0.2×	42	1.8k
Yan‐Shan Dai United States	20	2.0k 1.4×	838 1.6×	40 0.1×	271 0.9×	136 0.5×	37	2.6k
Tsuyoshi Sakoda Japan	23	1.3k 0.9×	621 1.2×	52 0.2×	172 0.6×	268 1.0×	54	2.6k
Michiko Hayasaka Japan	14	1.6k 1.1×	106 0.2×	169 0.5×	421 1.5×	115 0.4×	22	2.0k

Countries citing papers authored by Behzad Moghadaszadeh

Since Specialization

Citations

This map shows the geographic impact of Behzad Moghadaszadeh's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Behzad Moghadaszadeh with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Behzad Moghadaszadeh more than expected).

Fields of papers citing papers by Behzad Moghadaszadeh

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Behzad Moghadaszadeh. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Behzad Moghadaszadeh. The network helps show where Behzad Moghadaszadeh may publish in the future.

Co-authorship network of co-authors of Behzad Moghadaszadeh

This figure shows the co-authorship network connecting the top 25 collaborators of Behzad Moghadaszadeh. A scholar is included among the top collaborators of Behzad Moghadaszadeh based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Behzad Moghadaszadeh. Behzad Moghadaszadeh is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Hickey, Emily, et al.. (2025). Loss of adenylosuccinate synthetase 1 in mice recapitulates features of ADSS1 myopathy. Human Molecular Genetics. 35(1).

Li, Qifei, Jasmine Lin, Shiyu Luo, et al.. (2024). Integrated multi‐omics approach reveals the role of striated muscle preferentially expressed protein kinase in skeletal muscle including its relationship with myospryn complex. Journal of Cachexia Sarcopenia and Muscle. 15(3). 1003–1015. 1 indexed citations

Rybalka, Emma, et al.. (2023). Adenylosuccinic Acid: An Orphan Drug with Untapped Potential. Pharmaceuticals. 16(6). 822–822. 7 indexed citations

Tabebordbar, Mohammadsharif, Kim A. Lagerborg, Alexandra C. Stanton, et al.. (2021). Directed evolution of a family of AAV capsid variants enabling potent muscle-directed gene delivery across species. Cell. 184(19). 4919–4938.e22. 292 indexed citations breakdown →

Guo, Yuxuan, Yangpo Cao, Blake D. Jardin, et al.. (2020). Sarcomeres regulate murine cardiomyocyte maturation through MRTF-SRF signaling. Proceedings of the National Academy of Sciences. 118(2). 56 indexed citations

Shelton, G. Diane, Georgina Child, Sophia Tzannes, et al.. (2015). X-linked myotubular myopathy in Rottweiler dogs is caused by a missense mutation in Exon 11 of the MTM1 gene. Skeletal Muscle. 5(1). 1–1. 55 indexed citations

Agrawal, Pankaj B., Christopher R. Pierson, Mugdha Joshi, et al.. (2014). SPEG Interacts with Myotubularin, and Its Deficiency Causes Centronuclear Myopathy with Dilated Cardiomyopathy. The American Journal of Human Genetics. 95(2). 218–226. 115 indexed citations

Huang, Hai, et al.. (2009). Cell Membrane Expression of Cardiac Sodium Channel Na_v1.5 Is Modulated by α-Actinin-2 Interaction. Biochemistry. 49(1). 166–178. 43 indexed citations

Lawlor, Michael W., et al.. (2009). Mutations of tropomyosin 3 (TPM3) are common and associated with type 1 myofiber hypotrophy in congenital fiber type disproportion. Human Mutation. 31(2). 176–183. 53 indexed citations

10.

Keller, Christian E., Arthur P. Hays, Lewis P. Rowland, et al.. (2006). Adult-Onset Nemaline Myopathy and Monoclonal Gammopathy. Archives of Neurology. 63(1). 132–132. 25 indexed citations

11.

Pierson, Christopher R., Kinga K. Tomczak, Pankaj B. Agrawal, Behzad Moghadaszadeh, & Alan H. Beggs. (2005). X-Linked Myotubular and Centronuclear Myopathies. Journal of Neuropathology & Experimental Neurology. 64(7). 555–564. 77 indexed citations

12.

Blasi, Claudia Di, Behzad Moghadaszadeh, Tiziana Negri, et al.. (2004). Abnormal lysosomal and ubiquitin‐proteasome pathways in 19p13.3 distal myopathy. Annals of Neurology. 56(1). 133–138. 15 indexed citations

13.

Moghadaszadeh, Behzad. (2003). Compensation for dystrophin-deficiency: ADAM12 overexpression in skeletal muscle results in increased 7 integrin, utrophin and associated glycoproteins. Human Molecular Genetics. 12(19). 2467–2479. 54 indexed citations

14.

Kawaguchi, Nobuko, Christina Sundberg, Marie Kveiborg, et al.. (2003). ADAM12 induces actin cytoskeleton and extracellular matrix reorganization during early adipocyte differentiation by regulating β1 integrin function. Journal of Cell Science. 116(19). 3893–3904. 114 indexed citations

15.

Kronqvist, Pauliina, Nobuko Kawaguchi, Reidar Albrechtsen, et al.. (2002). ADAM12 Alleviates the Skeletal Muscle Pathology in mdx Dystrophic Mice. American Journal Of Pathology. 161(5). 1535–1540. 49 indexed citations

16.

Mercuri, Eugenio, Beril Talim, Behzad Moghadaszadeh, et al.. (2002). Clinical and imaging findings in six cases of congenital muscular dystrophy with rigid spine syndrome linked to chromosome 1p (RSMD1). Neuromuscular Disorders. 12(7-8). 631–638. 160 indexed citations

17.

Demir, Ercan, Patrizia Sabatelli, Valérie Allamand, et al.. (2002). Mutations in COL6A3 Cause Severe and Mild Phenotypes of Ullrich Congenital Muscular Dystrophy. The American Journal of Human Genetics. 70(6). 1446–1458. 119 indexed citations

18.

Ferreiro, Ana, Susana Quijano-Roy, Claire Pichereau, et al.. (2002). Mutations of the Selenoprotein N Gene, Which Is Implicated in Rigid Spine Muscular Dystrophy, Cause the Classical Phenotype of Multiminicore Disease: Reassessing the Nosology of Early-Onset Myopathies. The American Journal of Human Genetics. 71(4). 739–749. 234 indexed citations

19.

Vignier, Nicolas, Behzad Moghadaszadeh, Françoise Gary, et al.. (1999). Structure, Genetic Localization, and Identification of the Cardiac and Skeletal Muscle Transcripts of the Human Integrin α7 Gene (ITGA7). Biochemical and Biophysical Research Communications. 260(2). 357–364. 18 indexed citations

20.

Moghadaszadeh, Behzad, Isabelle Desguerre, Haluk Topaloğlu, et al.. (1998). Identification of a New Locus for a Peculiar Form of Congenital Muscular Dystrophy with Early Rigidity of the Spine, on Chromosome 1p35-36. The American Journal of Human Genetics. 62(6). 1439–1445. 62 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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