Thomas Voit

12.7k total citations · 1 hit paper
137 papers, 7.9k citations indexed

About

Thomas Voit is a scholar working on Molecular Biology, Physiology and Cardiology and Cardiovascular Medicine. According to data from OpenAlex, Thomas Voit has authored 137 papers receiving a total of 7.9k indexed citations (citations by other indexed papers that have themselves been cited), including 102 papers in Molecular Biology, 26 papers in Physiology and 23 papers in Cardiology and Cardiovascular Medicine. Recurrent topics in Thomas Voit's work include Muscle Physiology and Disorders (77 papers), Cardiomyopathy and Myosin Studies (19 papers) and Genetic Neurodegenerative Diseases (15 papers). Thomas Voit is often cited by papers focused on Muscle Physiology and Disorders (77 papers), Cardiomyopathy and Myosin Studies (19 papers) and Genetic Neurodegenerative Diseases (15 papers). Thomas Voit collaborates with scholars based in Germany, France and United Kingdom. Thomas Voit's co-authors include Volker Straub, Francesco Muntoni, Francesco Muntoni, Ralf Herrmann, S. Brown, Norma B. Romero, Caroline A. Sewry, Martin Brockington, Pascale Guicheney and Eugenio Mercuri and has published in prestigious journals such as Science, Proceedings of the National Academy of Sciences and The Lancet.

In The Last Decade

Thomas Voit

134 papers receiving 7.7k citations

Hit Papers

Muscular Dystrophy and Neuronal Migration Disorder Caused... 2001 2026 2009 2017 2001 100 200 300 400 500
What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

  1. Muscular Dystrophy and Neuronal Migration Disorder Caused by Mutations in a Glycosyltransferase, POMGnT1
    2001 536 citations Volker Straub, Beril Talim et al. profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Thomas Voit Germany 50 5.8k 1.4k 1.3k 1.3k 1.2k 137 7.9k
Melissa J. Spencer United States 47 5.3k 0.9× 1.0k 0.7× 1.4k 1.1× 826 0.7× 1.5k 1.3× 100 6.5k
Kevin M. Flanigan United States 45 5.6k 1.0× 1.2k 0.8× 844 0.6× 1.3k 1.1× 573 0.5× 189 7.3k
Luciano Merlini Italy 54 8.3k 1.4× 1.6k 1.1× 1.1k 0.8× 1.5k 1.2× 1.7k 1.4× 250 11.0k
Klaus Addicks Germany 50 3.7k 0.6× 1.2k 0.8× 1.1k 0.8× 645 0.5× 941 0.8× 199 8.4k
Kathryn R. Wagner United States 44 5.6k 1.0× 812 0.6× 1.3k 1.0× 1.2k 1.0× 782 0.7× 123 7.4k
Kate Bushby United Kingdom 58 8.0k 1.4× 1.9k 1.3× 1.2k 0.9× 2.4k 1.9× 1.3k 1.1× 183 9.7k
Enzo Ricci Italy 49 5.4k 0.9× 1.2k 0.8× 611 0.5× 1.3k 1.1× 517 0.4× 169 7.1k
Mayana Zatz Brazil 49 6.2k 1.1× 1.9k 1.3× 1.0k 0.8× 1.4k 1.1× 1.2k 1.0× 344 9.5k
Hart G.W. Lidov United States 38 4.3k 0.7× 1.8k 1.3× 1.4k 1.1× 654 0.5× 692 0.6× 104 7.0k
Kanneboyina Nagaraju United States 52 5.1k 0.9× 727 0.5× 2.0k 1.5× 789 0.6× 783 0.7× 150 7.9k

Countries citing papers authored by Thomas Voit

Since Specialization
Citations

This map shows the geographic impact of Thomas Voit's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Thomas Voit with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Thomas Voit more than expected).

Fields of papers citing papers by Thomas Voit

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Thomas Voit. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Thomas Voit. The network helps show where Thomas Voit may publish in the future.

Co-authorship network of co-authors of Thomas Voit

This figure shows the co-authorship network connecting the top 25 collaborators of Thomas Voit. A scholar is included among the top collaborators of Thomas Voit based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Thomas Voit. Thomas Voit is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Schierbauer, Janis, Harald Sourij, Paul Zimmermann, et al.. (2025). Performance of three simultaneously used rtCGM systems around a physically active weekend camp in adults with type 1 diabetes: A prospective lab and real‐world study. Diabetes Obesity and Metabolism. 28(3). 1867–1873.
2.
Hogrel, Jean‐Yves, V. Decostre, Isabelle Ledoux, et al.. (2020). Normalized grip strength is a sensitive outcome measure through all stages of Duchenne muscular dystrophy. Journal of Neurology. 267(7). 2022–2028. 16 indexed citations
3.
Catapano, Francesco, Joana Domingos, Valeria Ricotti, et al.. (2020). Novel Free-Circulating and Extracellular Vesicle-Derived MiRNAs Dysregulated in Duchenne Muscular Dystrophy. Epigenomics. 12(21). 1899–1915. 5 indexed citations
4.
Schwartz, Christine, Martina Fischer, Kamel Mamchaoui, et al.. (2017). Lamins and nesprin-1 mediate inside-out mechanical coupling in muscle cell precursors through FHOD1. Scientific Reports. 7(1). 1253–1253. 38 indexed citations
5.
Domenger, Claire, Virginie François, Adrien Léger, et al.. (2017). RNA-Seq Analysis of an Antisense Sequence Optimized for Exon Skipping in Duchenne Patients Reveals No Off-Target Effect. Molecular Therapy — Nucleic Acids. 10. 277–291. 7 indexed citations
6.
Capelle, Carine I. van, Jan C. van der Meijden, Johanna M. P. van den Hout, et al.. (2016). Childhood Pompe disease: clinical spectrum and genotype in 31 patients. Orphanet Journal of Rare Diseases. 11(1). 65–65. 47 indexed citations
7.
Ricotti, Valeria, Francesco Muntoni, & Thomas Voit. (2015). Challenges of clinical trial design for DMD. Neuromuscular Disorders. 25(12). 932–935. 10 indexed citations
8.
9.
Jeanson-Leh, Laurence, Fatima Amor, Caroline Le Guiner, et al.. (2014). Serum Profiling Identifies Novel Muscle miRNA and Cardiomyopathy-Related miRNA Biomarkers in Golden Retriever Muscular Dystrophy Dogs and Duchenne Muscular Dystrophy Patients. American Journal Of Pathology. 184(11). 2885–2898. 66 indexed citations
10.
Amthor, Helge, Raymond Macharia, Roberto Navarrete, et al.. (2007). Lack of myostatin results in excessive muscle growth but impaired force generation. Proceedings of the National Academy of Sciences. 104(6). 1835–1840. 328 indexed citations
11.
Merlini, Luciano, Christian Dohna‐Schwake, Maria Jędrzejowska, et al.. (2007). G.P.2.15 EUROSMART: European Spinal Muscular Atrophy Randomised Placebo-Controlled Trial of acetyl-L-carnitine in spinal muscular atrophy. Neuromuscular Disorders. 17(9-10). 780–781. 2 indexed citations
12.
Piao, Xianhua, R. Sean Hill, Adria Bodell, et al.. (2004). G Protein-Coupled Receptor-Dependent Development of Human Frontal Cortex. Science. 303(5666). 2033–2036. 405 indexed citations
13.
Hunter, Michael, Rafaëlle Bernard, Elizabeth Freitas, et al.. (2003). Mutation screening of the N-myc downstream-regulated gene 1 (NDRG1) in patients with Charcot-Marie-Tooth Disease. Human Mutation. 22(2). 129–135. 41 indexed citations
14.
Blake, Donna Jo, Martin Brockington, Luciano Merlini, et al.. (2002). 85th ENMC International Workshop on Congenital Muscular Dystrophy - 6th International CMD Workshop - 1st Workshop of the Myo-Cluster Project 'GENRE' - 27-28th October 2000, Naarden, The Netherlands (vol 12, pg 69, 2002). UCL Discovery (University College London). 2 indexed citations
15.
Ferreiro, Ana, Nicole Monnier, Norma B. Romero, et al.. (2002). A recessive form of central core disease, transiently presenting as multi‐minicore disease, is associated with a homozygous mutation in the ryanodine receptor type 1 gene. Annals of Neurology. 51(6). 750–759. 137 indexed citations
16.
Klepper, Jörg & Thomas Voit. (2002). Facilitated glucose transporter protein type 1 (GLUT1) deficiency syndrome: impaired glucose transport into brain – a review. European Journal of Pediatrics. 161(6). 295–304. 121 indexed citations
17.
Goebel, Hans H., Lev G. Goldfarb, R. Schober, et al.. (2001). Reducing Body Myopathy with Cytoplasmic Bodies and Rigid Spine Syndrome: A Mixed Congenital Myopathy. Neuropediatrics. 32(4). 196–205. 18 indexed citations
18.
Brockington, Martin, Derek J. Blake, Paola Prandini, et al.. (2001). Mutations in the Fukutin-Related Protein Gene (FKRP) Cause a Form of Congenital Muscular Dystrophy with Secondary Laminin α2 Deficiency and Abnormal Glycosylation of α-Dystroglycan. The American Journal of Human Genetics. 69(6). 1198–1209. 452 indexed citations
19.
Bittner, Reginald E., Sigrid Shorny, Wolfgang Sperl, et al.. (1995). Sarcolemmal expression of dystrophin C-terminus but reduced expression of 6q-dystrophin-related protein in two DMD patients with large deletions of the dystrophin gene. Neuromuscular Disorders. 5(2). 81–92. 3 indexed citations
20.
Wessalowski, Rüdiger, et al.. (1993). Multiple brain abscesses caused by Salmonella enteritidis in a neonate. The Pediatric Infectious Disease Journal. 12(8). 683–687. 32 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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