Bastian G. Brinkmann
- Developmental Neuroscience top 0.2%
- Neurogenesis and neuroplasticity mechanisms 5
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- Nerve injury and regeneration 5
- Hereditary Neurological Disorders 4
- Axon Guidance and Neuronal Signaling 2
- Neurology top 1%
- Neuroinflammation and Neurodegeneration Mechanisms 2
- Neurological diseases and metabolism 1
- Cell Biology top 5%
- Neurology top 5%
- Neuroinflammation and Neurodegeneration Mechanisms 2
- Neurological diseases and metabolism 1
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- Cancer Cells and Metastasis 1
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- Signaling Pathways in Disease 1
- Co-authors
- Michael W. SeredaKlaus‐Armin NaveMarkus H. SchwabDies MeijerCary LaiCarmen BirchmeierTobias M. FischerG. Michailov
- Partner nations
- GermanyUnited StatesUnited Kingdom
In The Last Decade
Bastian G. Brinkmann
10 papers receiving 2.8k citations
Hit Papers
Peers
Comparison fields: 5 of 101
- Developmental Neuroscience 1.1k
- Cellular and Molecular Neuroscience 1.3k
- Neurology 575
- Cell Biology 295
- Neurology 271
Countries citing papers authored by Bastian G. Brinkmann
This map shows the geographic impact of Bastian G. Brinkmann's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Bastian G. Brinkmann with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Bastian G. Brinkmann more than expected).
Fields of papers citing papers by Bastian G. Brinkmann
This network shows the impact of papers produced by Bastian G. Brinkmann. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Bastian G. Brinkmann. The network helps show where Bastian G. Brinkmann may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Bastian G. Brinkmann, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2017 | 7 | |
| 2 | 2016 | 114 | |
| 3 | 2012 | 53 | |
| 4 | 2012 | 219 | |
| 5 | Glycolytic oligodendrocytes maintain myelin and long-term axonal integritybreakdown → | 2012 | 1085 |
| 6 | 2009 | 45 | |
| 7 | 2009 | 71 | |
| 8 | 2008 | 291 | |
| 9 | 2007 | 186 | |
| 10 | Axonal Neuregulin-1 Regulates Myelin Sheath Thicknessbreakdown → | 2004 | 730 |
About Bastian G. Brinkmann
Bastian G. Brinkmann is a scholar working on Developmental Neuroscience, Cellular and Molecular Neuroscience and Neurology, having authored 10 papers that have together received 2.8k indexed citations. Recurring topics across this work include Nerve injury and regeneration (5 papers), Neurogenesis and neuroplasticity mechanisms (5 papers), Hereditary Neurological Disorders (4 papers), Axon Guidance and Neuronal Signaling (2 papers), Neuroinflammation and Neurodegeneration Mechanisms (2 papers), Cancer Cells and Metastasis (1 paper), Neurological diseases and metabolism (1 paper) and Signaling Pathways in Disease (1 paper). The work is most often cited by research in Developmental Neuroscience (1.1k citations), Cellular and Molecular Neuroscience (1.3k citations) and Neurology (575 citations). Bastian G. Brinkmann has collaborated with scholars based in Germany, United States and United Kingdom. Frequent co-authors include Michael W. Sereda, Klaus‐Armin Nave, Markus H. Schwab, Dies Meijer, Cary Lai, Carmen Birchmeier, Tobias M. Fischer, G. Michailov, Lorna W. Role and Bernhard A. Haug. Their work appears in journals such as Nature, Science and Neuron.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.