Patrizia Casaccia‐Bonnefil

6.7k total citations · 1 hit paper
56 papers, 5.6k citations indexed

About

Patrizia Casaccia‐Bonnefil is a scholar working on Molecular Biology, Developmental Neuroscience and Cancer Research. According to data from OpenAlex, Patrizia Casaccia‐Bonnefil has authored 56 papers receiving a total of 5.6k indexed citations (citations by other indexed papers that have themselves been cited), including 42 papers in Molecular Biology, 28 papers in Developmental Neuroscience and 20 papers in Cancer Research. Recurrent topics in Patrizia Casaccia‐Bonnefil's work include Neurogenesis and neuroplasticity mechanisms (27 papers), Epigenetics and DNA Methylation (23 papers) and MicroRNA in disease regulation (13 papers). Patrizia Casaccia‐Bonnefil is often cited by papers focused on Neurogenesis and neuroplasticity mechanisms (27 papers), Epigenetics and DNA Methylation (23 papers) and MicroRNA in disease regulation (13 papers). Patrizia Casaccia‐Bonnefil collaborates with scholars based in United States, United Kingdom and Canada. Patrizia Casaccia‐Bonnefil's co-authors include Moses V. Chao, Jiadong Li, Bruce Carter, Siming Shen, Rick T. Dobrowsky, Sung Ok Yoon, Bruce Carter, Andrew Koff, Ye He and Chenghua Gu and has published in prestigious journals such as Nature, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.

In The Last Decade

Patrizia Casaccia‐Bonnefil

56 papers receiving 5.5k citations

Hit Papers

Death of oligodendrocytes mediated by the interaction of ... 1996 2026 2006 2016 1996 200 400 600
What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

  1. Death of oligodendrocytes mediated by the interaction of nerve growth factor with its receptor p75
    1996 679 citations Patrizia Casaccia‐Bonnefil, Moses V. Chao et al. profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Patrizia Casaccia‐Bonnefil United States 40 3.4k 2.1k 1.9k 946 693 56 5.6k
Takuya Shimazaki Japan 32 2.7k 0.8× 2.0k 0.9× 1.4k 0.7× 466 0.5× 706 1.0× 61 5.1k
Dies Meijer Netherlands 43 3.1k 0.9× 1.7k 0.8× 2.6k 1.4× 495 0.5× 541 0.8× 74 6.7k
Rashmi Bansal United States 45 3.0k 0.9× 2.8k 1.3× 1.5k 0.8× 622 0.7× 788 1.1× 95 5.6k
Chun‐Li Zhang United States 35 4.9k 1.4× 1.6k 0.8× 1.5k 0.8× 670 0.7× 486 0.7× 64 6.7k
Hirohide Takebayashi Japan 38 3.0k 0.9× 3.1k 1.4× 1.9k 1.0× 939 1.0× 1.1k 1.5× 115 6.3k
Eric D. Laywell United States 27 2.1k 0.6× 2.3k 1.1× 1.4k 0.7× 570 0.6× 676 1.0× 42 4.6k
Anne Baron‐Van Evercooren France 42 2.8k 0.8× 3.9k 1.8× 2.9k 1.5× 762 0.8× 1.3k 1.8× 99 7.0k
Markus H. Schwab Germany 31 2.4k 0.7× 1.8k 0.8× 2.3k 1.2× 298 0.3× 735 1.1× 48 5.0k
Susan K. Goderie United States 26 2.8k 0.8× 2.4k 1.1× 1.5k 0.8× 635 0.7× 691 1.0× 35 5.0k
Benjamin Deneen United States 37 2.8k 0.8× 1.7k 0.8× 1.3k 0.7× 947 1.0× 1.6k 2.4× 86 5.5k

Countries citing papers authored by Patrizia Casaccia‐Bonnefil

Since Specialization
Citations

This map shows the geographic impact of Patrizia Casaccia‐Bonnefil's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Patrizia Casaccia‐Bonnefil with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Patrizia Casaccia‐Bonnefil more than expected).

Fields of papers citing papers by Patrizia Casaccia‐Bonnefil

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Patrizia Casaccia‐Bonnefil. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Patrizia Casaccia‐Bonnefil. The network helps show where Patrizia Casaccia‐Bonnefil may publish in the future.

Co-authorship network of co-authors of Patrizia Casaccia‐Bonnefil

This figure shows the co-authorship network connecting the top 25 collaborators of Patrizia Casaccia‐Bonnefil. A scholar is included among the top collaborators of Patrizia Casaccia‐Bonnefil based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Patrizia Casaccia‐Bonnefil. Patrizia Casaccia‐Bonnefil is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Shen, Siming, Juan Sandoval, Victoria A. Swiss, et al.. (2008). Age-dependent epigenetic control of differentiation inhibitors is critical for remyelination efficiency. Nature Neuroscience. 11(9). 1024–1034. 370 indexed citations
2.
He, Ye & Patrizia Casaccia‐Bonnefil. (2008). The Yin and Yang of YY1 in the nervous system. Journal of Neurochemistry. 106(4). 1493–1502. 103 indexed citations
3.
Li, Jiadong, et al.. (2006). A molecular insight of Hes5‐dependent inhibition of myelin gene expression: old partners and new players. The EMBO Journal. 25(20). 4833–4842. 137 indexed citations
4.
Mastronardi, Fabrizio G., D. D. Wood, Mei Jiang, et al.. (2006). Increased Citrullination of Histone H3 in Multiple Sclerosis Brain and Animal Models of Demyelination: A Role for Tumor Necrosis Factor-Induced Peptidylarginine Deiminase 4 Translocation. Journal of Neuroscience. 26(44). 11387–11396. 188 indexed citations
5.
Marin‐Husstege, Mireya, Ye He, Jiadong Li, et al.. (2006). Multiple roles of Id4 in developmental myelination: Predicted outcomes and unexpected findings. Glia. 54(4). 285–296. 74 indexed citations
6.
Gökhan, Şölen, et al.. (2005). Combinatorial Profiles of Oligodendrocyte-Selective Classes of Transcriptional Regulators Differentially Modulate Myelin Basic Protein Gene Expression. Journal of Neuroscience. 25(36). 8311–8321. 102 indexed citations
7.
Cunliffe, Vincent T. & Patrizia Casaccia‐Bonnefil. (2005). Histone deacetylase 1 is essential for oligodendrocyte specification in the zebrafish CNS. Mechanisms of Development. 123(1). 24–30. 87 indexed citations
8.
Marin‐Husstege, Mireya, Michela Muggironi, D. Raban, Robert P. Skoff, & Patrizia Casaccia‐Bonnefil. (2004). Oligodendrocyte Progenitor Proliferation and Maturation Is Differentially Regulated by Male and Female Sex Steroid Hormones. Developmental Neuroscience. 26(2-4). 245–254. 100 indexed citations
9.
Kim, Dong, Xiurong Zhao, Christina Tu, Patrizia Casaccia‐Bonnefil, & Moses V. Chao. (2004). Prevention of apoptotic but not necrotic cell death following neuronal injury by neurotrophins signaling through the tyrosine kinase receptor. Journal of neurosurgery. 100(1). 79–87. 40 indexed citations
10.
Muggironi, Michela, et al.. (2003). Oligodendrocyte process outgrowth in vitro is modulated by epigenetic regulation of cytoskeletal severing proteins. Glia. 44(3). 264–274. 49 indexed citations
11.
Doetsch, Fiona, José Manuel García‐Verdugo, Isabelle Caillé, et al.. (2002). Lack of the Cell-Cycle Inhibitor p27Kip1 Results in Selective Increase of Transit-Amplifying Cells for Adult Neurogenesis. Journal of Neuroscience. 22(6). 2255–2264. 157 indexed citations
12.
Xiang, Zhongmin, Sabina Hrabětová, S Moskowitz, et al.. (2000). Long-term maintenance of mature hippocampal slices in vitro. Journal of Neuroscience Methods. 98(2). 145–154. 71 indexed citations
13.
Casaccia‐Bonnefil, Patrizia. (2000). Cell death in the oligodendrocyte lineage: A molecular perspective of life/death decisions in development and disease. Glia. 29(2). 124–135. 81 indexed citations
14.
Casaccia‐Bonnefil, Patrizia, Chenghua Gu, & Moses V. Chao. (1999). Neurotrophins in Cell Survival/Death Decisions. Advances in experimental medicine and biology. 468. 275–282. 119 indexed citations
15.
Casaccia‐Bonnefil, Patrizia, Rebecca Hardy, Kenneth K. Teng, et al.. (1999). Loss of p27Kip1 function results in increased proliferative capacity of oligodendrocyte progenitors but unaltered timing of differentiation. Development. 126(18). 4027–4037. 110 indexed citations
16.
Casaccia‐Bonnefil, Patrizia, Haeyoung Kong, & Moses V. Chao. (1998). Neurotrophins: the biological paradox of survival factors eliciting apoptosis. Cell Death and Differentiation. 5(5). 357–364. 123 indexed citations
17.
Casaccia‐Bonnefil, Patrizia, et al.. (1997). Oligodendrocyte precursor differentiation is perturbed in the absence of the cyclin-dependent kinase inhibitor p27Kip1. Genes & Development. 11(18). 2335–2346. 227 indexed citations
18.
Casaccia‐Bonnefil, Patrizia, et al.. (1996). Central glial and neuronal populations display differential sensitivity to ceramide-dependent cell death. Journal of Neuroscience Research. 43(3). 382–389. 62 indexed citations
19.
Bergold, Peter J., et al.. (1993). Transsynaptic neuronal loss induced in hippocampal slice cultures by a herpes simplex virus vector expressing the GluR6 subunit of the kainate receptor.. Proceedings of the National Academy of Sciences. 90(13). 6165–6169. 70 indexed citations
20.
Casaccia‐Bonnefil, Patrizia, Eiríkur Benedikz, Hong Shen, et al.. (1993). Localized gene transfer into organotypic hippocampal slice cultures and acute hippocampal slices. Journal of Neuroscience Methods. 50(3). 341–351. 17 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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