Axel T. Brünger
- Cell Biology top 0.01%
- Cellular transport and secretion 100
- Endoplasmic Reticulum Stress and Disease 20
- Molecular Biology top 0.01%
- Protein Structure and Dynamics 93
- Lipid Membrane Structure and Behavior 77
- RNA and protein synthesis mechanisms 28
- DNA and Nucleic Acid Chemistry 19
- Structural Biology top 0.2%
- Physiology top 0.05%
- Cellular and Molecular Neuroscience top 0.2%
- Neuroscience and Neuropharmacology Research 34
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- Enzyme Structure and Function 98
- Co-authors
- Luke M. RicePaul D. AdamsMichaël NilgesMartin KarplusG. Marius CloreJiansheng JiangRandy J. ReadNeesh Pannu
- Partner nations
- United StatesGermanyUnited Kingdom
In The Last Decade
Axel T. Brünger
294 papers receiving 55.3k citations
Hit Papers
Peers
Comparison fields: 5 of 186
- Cell Biology 12.5k
- Molecular Biology 42.9k
- Structural Biology 726
- Physiology 1.5k
- Cellular and Molecular Neuroscience 4.9k
Countries citing papers authored by Axel T. Brünger
This map shows the geographic impact of Axel T. Brünger's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Axel T. Brünger with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Axel T. Brünger more than expected).
Fields of papers citing papers by Axel T. Brünger
This network shows the impact of papers produced by Axel T. Brünger. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Axel T. Brünger. The network helps show where Axel T. Brünger may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Axel T. Brünger, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2025 | 1 | |
| 2 | 2025 | 1 | |
| 3 | 2023 | 7 | |
| 4 | 2023 | 14 | |
| 5 | 2022 | 27 | |
| 6 | 2019 | 1 | |
| 7 | 2017 | 19 | |
| 8 | 2015 | 21 | |
| 9 | 2014 | 25 | |
| 10 | 2013 | 83 | |
| 11 | 2012 | 16 | |
| 12 | 2011 | 133 | |
| 13 | 2010 | 1 | |
| 14 | 2009 | 111 | |
| 15 | 2007 | 52 | |
| 16 | 2007 | 41 | |
| 17 | 2007 | 353 | |
| 18 | 2005 | 11 | |
| 19 | 2004 | 0 | |
| 20 | 2004 | 6 |
About Axel T. Brünger
Axel T. Brünger is a scholar working on Cell Biology, Structural Biology and Molecular Biology, having authored 298 papers that have together received 56.0k indexed citations. Recurring topics across this work include Cellular transport and secretion (100 papers), Enzyme Structure and Function (98 papers), Protein Structure and Dynamics (93 papers), Lipid Membrane Structure and Behavior (77 papers), Neuroscience and Neuropharmacology Research (34 papers), RNA and protein synthesis mechanisms (28 papers), Endoplasmic Reticulum Stress and Disease (20 papers) and DNA and Nucleic Acid Chemistry (19 papers). The work is most often cited by research in Cell Biology (12.5k citations), Molecular Biology (42.9k citations) and Structural Biology (726 citations). Axel T. Brünger has collaborated with scholars based in United States, Germany and United Kingdom. Frequent co-authors include Luke M. Rice, Paul D. Adams, Michaël Nilges, Martin Karplus, G. Marius Clore, Jiansheng Jiang, Randy J. Read, Neesh Pannu, Thomas Simonson and John Kuszewski. Their work appears in journals such as Nature, Science and Cell.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.