Stephen E. Kaiser

2.3k citations
27 papers · 1.9k · h-index 19

Impact in

Papers in

    • Ubiquitin and proteasome pathways 12
    • Epigenetics and DNA Methylation 4
    • Lipid Membrane Structure and Behavior 2
    • Cancer-related gene regulation 2
    • Endoplasmic Reticulum Stress and Disease 3
    • Cellular transport and secretion 3

Stephen E. Kaiser

27 papers receiving 1.8k citations

Peers

Stephen E. Kaiser
Comparison fields: 5 of 112
  • Biological Psychiatry 104
  • Cell Biology 537
  • Molecular Biology 1.3k
  • Physiology 73
  • Epidemiology 470
Replace Paul W. Sheppard with:
Paul W. Sheppard United Kingdom
Hikaru Tsuchiya Japan
Martin A. Augustin Germany
Elizabeth Donohue Canada
Lynn Bedford United Kingdom
Antonello D’Arrigo Italy
Ragna Sannerud Belgium
Luisa L. Gregori United States
Anthony A. High United States
David A. Lomas United Kingdom
Stephen E. Kaiser relative to Paul W. Sheppard United Kingdom Paul W. Sheppard's profile →
Citations per field
00.5×2×3×4×4.6×
Paul W. Sheppard · 1×
Citations per year

Countries citing papers authored by Stephen E. Kaiser

Since Specialization
Citations

This map shows the geographic impact of Stephen E. Kaiser's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Stephen E. Kaiser with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Stephen E. Kaiser more than expected).

Fields of papers citing papers by Stephen E. Kaiser

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Stephen E. Kaiser. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Stephen E. Kaiser. The network helps show where Stephen E. Kaiser may publish in the future.

Co-authors

The 25 scholars most cited alongside Stephen E. Kaiser, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Stephen E. Kaiser Line = papers co-authored together Stephen E. Kaiser links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 27 papers — load more, or switch the sort, to bring in the rest.

#Work
1 2005225
2 2011200
3 2010148
4 2012143
5 2011140
6 2015128
7 2017122
8 2017111
9 201292
10 201188
11 202068
12 201265
13 200150
14 201649
15 200741
16 201338
17 202038
18 197633
19 201120
20 201319

About Stephen E. Kaiser

Stephen E. Kaiser is a scholar working on Molecular Biology, Cell Biology, Epidemiology, Physiology and Cancer Research, having authored 27 papers that have together received 1.9k indexed citations. Recurring topics across this work include Ubiquitin and proteasome pathways (12 papers), Autophagy in Disease and Therapy (9 papers), Epigenetics and DNA Methylation (4 papers), Endoplasmic Reticulum Stress and Disease (3 papers), Cellular transport and secretion (3 papers), Lipid Membrane Structure and Behavior (2 papers), Cancer-related gene regulation (2 papers) and Genetics and Neurodevelopmental Disorders (2 papers). The work is most often cited by research in Biological Psychiatry (104 citations), Cell Biology (537 citations), Molecular Biology (1.3k citations), Physiology (73 citations) and Epidemiology (470 citations). Stephen E. Kaiser has collaborated with scholars based in United States, Germany and United Kingdom. Frequent co-authors include Ron R. Kopito, Thomas A. Shaler, Brigit E. Riley, Axel T. Brünger, Amy R. Reilein, Peter Walter, Jason H. Brickner, Brenda A. Schulman, Christopher H. Becker and Howard Schulman. Their work appears in journals such as Journal of Medicinal Chemistry, The Journal of Cell Biology, Autophagy, Journal of Biological Chemistry and Molecular Cell.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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