Axel Seltsam

2.9k total citations
88 papers, 2.0k citations indexed

About

Axel Seltsam is a scholar working on Hematology, Physiology and Biochemistry. According to data from OpenAlex, Axel Seltsam has authored 88 papers receiving a total of 2.0k indexed citations (citations by other indexed papers that have themselves been cited), including 38 papers in Hematology, 25 papers in Physiology and 24 papers in Biochemistry. Recurrent topics in Axel Seltsam's work include Blood groups and transfusion (32 papers), Erythrocyte Function and Pathophysiology (25 papers) and Blood transfusion and management (24 papers). Axel Seltsam is often cited by papers focused on Blood groups and transfusion (32 papers), Erythrocyte Function and Pathophysiology (25 papers) and Blood transfusion and management (24 papers). Axel Seltsam collaborates with scholars based in Germany, United States and Australia. Axel Seltsam's co-authors include Rainer Blasczyk, Thomas Müller, Constança Figueiredo, Ute Gravemann, Wiebke Handke, Britta Eiz‐Vesper, Frank Tolksdorf, Michael Hallensleben, A. Salama and Franz F. Wagner and has published in prestigious journals such as Blood, Annals of the Rheumatic Diseases and Intensive Care Medicine.

In The Last Decade

Axel Seltsam

84 papers receiving 2.0k citations

Peers

Axel Seltsam

HEMAT

PHYSI

BIOCH

José Orlando Bordin Brazil

Philippe Rouger France

Kenji Ikebuchi Japan

Bjarte G. Solheim Norway

Jean‐Yves Muller France

Karina Yazdanbakhsh United States

Ilias I.N. Doxiadis Netherlands

John W. Heitman United States

Ali Akbar Pourfathollah Iran

Connie M. Arthur United States

José Orlando Bordin Brazil

Axel Seltsam

1.2k ×1.4

HEMAT

375 ×0.7

PHYSI

712 ×1.8

BIOCH

155 ×0.5

105 ×0.4

Citations per year, relative to Axel Seltsam Axel Seltsam (= 1×) peers José Orlando Bordin

Countries citing papers authored by Axel Seltsam

Since Specialization

Citations

This map shows the geographic impact of Axel Seltsam's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Axel Seltsam with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Axel Seltsam more than expected).

Fields of papers citing papers by Axel Seltsam

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Axel Seltsam. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Axel Seltsam. The network helps show where Axel Seltsam may publish in the future.

Co-authorship network of co-authors of Axel Seltsam

This figure shows the co-authorship network connecting the top 25 collaborators of Axel Seltsam. A scholar is included among the top collaborators of Axel Seltsam based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Axel Seltsam. Axel Seltsam is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Gravemann, Ute, Wiebke Handke, Torsten J. Schulze, & Axel Seltsam. (2024). Growth and Distribution of Bacteria in Contaminated Whole Blood and Derived Blood Components. Transfusion Medicine and Hemotherapy. 51(2). 75–82. 2 indexed citations

Handke, Wiebke, Axel Seltsam, Iliyana Pepelanova, et al.. (2021). Comparative Analysis of Mesenchymal Stem Cell Cultivation in Fetal Calf Serum, Human Serum, and Platelet Lysate in 2D and 3D Systems. Frontiers in Bioengineering and Biotechnology. 8. 598389–598389. 33 indexed citations

Gravemann, Ute, Wiebke Handke, Thomas Müller, & Axel Seltsam. (2018). Bacterial inactivation of platelet concentrates with the THERAFLEX UV‐Platelets pathogen inactivation system. Transfusion. 59(4). 1324–1332. 20 indexed citations

Faddy, Helen M., Natalie A. Prow, Daniel Watterson, et al.. (2016). Inactivation of dengue, chikungunya, and Ross River viruses in platelet concentrates after treatment with ultraviolet C light. Transfusion. 56(6pt2). 1548–1555. 35 indexed citations

Jaimes, Yarúa, Lilia Goudeva, Stefanie Buchholz, et al.. (2012). Semaphorin 7A inhibits platelet production from CD34+ progenitor cells. Journal of Thrombosis and Haemostasis. 10(6). 1100–1108. 10 indexed citations

Wiltshire, Michael, Luke Sonoda, Stephen Thomas, et al.. (2012). Pathogen inactivation of platelets using ultraviolet C light: effect on in vitro function and recovery and survival of platelets. Transfusion. 53(5). 990–1000. 40 indexed citations

Lehmann, Jörg, Janina M. Tomm, Martin von Bergen�, et al.. (2012). Evaluation of the tolerability and immunogenicity of ultraviolet C‐irradiated autologous platelets in a dog model. Transfusion. 52(11). 2414–2426. 13 indexed citations

Raval, Jay S., Jonathan H. Waters, Axel Seltsam, et al.. (2010). Menopausal status affects the susceptibility of stored RBCs to mechanical stress. Vox Sanguinis. 100(4). 418–421. 58 indexed citations

Jaimes, Yarúa, Axel Seltsam, Britta Eiz‐Vesper, Rainer Blasczyk, & Constança Figueiredo. (2010). Regulation of HLA class II expression prevents allogeneic T‐cell responses. Tissue Antigens. 77(1). 36–44. 6 indexed citations

10.

Figueiredo, Constança, Miriam Wittmann, Dong Wang, et al.. (2008). Heat shock protein 70 (HSP70) induces cytotoxicity of T-helper cells. Blood. 113(13). 3008–3016. 77 indexed citations

11.

Seltsam, Axel, et al.. (2008). Rapid detection of anti‐Lu^b with recombinant Lu^b protein and the particle gel immunoassay. Transfusion. 48(4). 731–734. 8 indexed citations

12.

Deng, Zhihui, et al.. (2008). Haemolytic disease of fetus and newborn caused by ABO antibodies in a cisAB offspring. Transfusion and Apheresis Science. 39(2). 123–128. 8 indexed citations

13.

Figueiredo, Constança, Axel Seltsam, & Rainer Blasczyk. (2008). Permanent silencing of NKG2A expression for cell-based therapeutics. Journal of Molecular Medicine. 87(2). 199–210. 35 indexed citations

14.

Seltsam, Axel, et al.. (2007). Weak blood group B phenotypes may be caused by variations in the CCAAT‐binding factor/NF‐Y enhancer region of the ABO gene. Transfusion. 47(12). 2330–2335. 25 indexed citations

15.

Wagner, Franz F., Rainer Blasczyk, & Axel Seltsam. (2005). Nondeletional ABO*O alleles frequently cause blood donor typing problems. Transfusion. 45(8). 1331–1334. 14 indexed citations

16.

Poole, Joyce, Norbert Ahrens, Axel Seltsam, et al.. (2004). SERF: a new antigen in the Cromer blood group system. Transfusion Medicine. 14(4). 313–318. 11 indexed citations

17.

Bade‐Doeding, Christina, Holger-Andreas Elsner, Britta Eiz‐Vesper, et al.. (2004). A single amino-acid polymorphism in pocket�A of HLA-A*6602 alters the auxiliary anchors compared with HLA-A*6601 ligands. Immunogenetics. 56(2). 83–88. 21 indexed citations

18.

Seltsam, Axel, et al.. (2002). Rapid detection of autoantibodies to dsDNA with the particle gel immunoassay (ID-PaGIA). Annals of the Rheumatic Diseases. 61(4). 367–369. 5 indexed citations

19.

Seltsam, Axel, et al.. (2002). A weak blood group A phenotype caused by a new mutation at the ABO locus. Transfusion. 42(3). 294–301. 22 indexed citations

20.

Seltsam, Axel, et al.. (2000). Vaccination‐associated immune hemolytic anemia in two children. Transfusion. 40(8). 907–909. 26 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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