Kim Hue‐Roye

1.1k total citations
51 papers, 800 citations indexed

About

Kim Hue‐Roye is a scholar working on Hematology, Physiology and Genetics. According to data from OpenAlex, Kim Hue‐Roye has authored 51 papers receiving a total of 800 indexed citations (citations by other indexed papers that have themselves been cited), including 46 papers in Hematology, 42 papers in Physiology and 18 papers in Genetics. Recurrent topics in Kim Hue‐Roye's work include Blood groups and transfusion (46 papers), Erythrocyte Function and Pathophysiology (42 papers) and Hemoglobinopathies and Related Disorders (18 papers). Kim Hue‐Roye is often cited by papers focused on Blood groups and transfusion (46 papers), Erythrocyte Function and Pathophysiology (42 papers) and Hemoglobinopathies and Related Disorders (18 papers). Kim Hue‐Roye collaborates with scholars based in United States, Canada and United Kingdom. Kim Hue‐Roye's co-authors include Marion E. Reid, Christine Lomas‐Francis, Sunitha Vege, Connie M. Westhoff, Ghazala Hashmi, Michael Seul, Prabhakar Vissavajjhala, María Rios, Asok Chaudhuri and Carolyn Hoppe and has published in prestigious journals such as Blood, British Journal of Haematology and Transfusion.

In The Last Decade

Kim Hue‐Roye

49 papers receiving 782 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Kim Hue‐Roye United States 15 736 607 342 126 67 51 800
Nicole H. Smith United States 13 518 0.7× 480 0.8× 167 0.5× 128 1.0× 72 1.1× 22 714
Åsa Hellberg Sweden 16 593 0.8× 487 0.8× 175 0.5× 140 1.1× 46 0.7× 46 734
Marilyn Moulds United States 12 532 0.7× 449 0.7× 133 0.4× 127 1.0× 39 0.6× 43 627
Chao‐Peng Shao China 14 340 0.5× 290 0.5× 79 0.2× 117 0.9× 55 0.8× 34 462
M. E. Reid United States 13 337 0.5× 291 0.5× 69 0.2× 94 0.7× 31 0.5× 29 432
DM Lublin United States 8 315 0.4× 229 0.4× 89 0.3× 87 0.7× 32 0.5× 10 469
DJ Anstee United Kingdom 9 357 0.5× 365 0.6× 79 0.2× 87 0.7× 25 0.4× 17 557
T. Seno Japan 9 257 0.3× 192 0.3× 63 0.2× 78 0.6× 24 0.4× 25 414
Jo L. Procter United States 10 322 0.4× 134 0.2× 53 0.2× 71 0.6× 41 0.6× 12 422
ES Thompson United States 7 289 0.4× 210 0.3× 70 0.2× 77 0.6× 21 0.3× 8 390

Countries citing papers authored by Kim Hue‐Roye

Since Specialization
Citations

This map shows the geographic impact of Kim Hue‐Roye's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kim Hue‐Roye with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kim Hue‐Roye more than expected).

Fields of papers citing papers by Kim Hue‐Roye

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Kim Hue‐Roye. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kim Hue‐Roye. The network helps show where Kim Hue‐Roye may publish in the future.

Co-authorship network of co-authors of Kim Hue‐Roye

This figure shows the co-authorship network connecting the top 25 collaborators of Kim Hue‐Roye. A scholar is included among the top collaborators of Kim Hue‐Roye based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Kim Hue‐Roye. Kim Hue‐Roye is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
2.
Reid, Marion E., et al.. (2013). SC*994C>T causes the Sc(null) phenotype in Pacific Islanders and successful transfusion of Sc3+ blood to a patient with anti-Sc3.. PubMed. 29(2). 69–72. 2 indexed citations
3.
Reid, Marion E., et al.. (2013). Genomic analyses of RH alleles to improve transfusion therapy in patients with sickle cell disease. Blood Cells Molecules and Diseases. 52(4). 195–202. 44 indexed citations
4.
Hue‐Roye, Kim & M. E. Reid. (2012). The molecular basis of the LU:7 and LU:–7 phenotypes. Immunohematology. 28(4). 130–131. 1 indexed citations
5.
Hue‐Roye, Kim, et al.. (2011). Molecular basis of the rare gene complex, DIVa(C)‐, which encodes four low‐prevalence antigens in the Rh blood group system. Vox Sanguinis. 102(2). 167–170. 10 indexed citations
6.
Hue‐Roye, Kim, M. E. Reid, Connie M. Westhoff, & Christine Lomas‐Francis. (2011). Red cells from the original JAL+ proband are also DAK+ and STEM+. Vox Sanguinis. 101(1). 61–64. 2 indexed citations
7.
Hue‐Roye, Kim, et al.. (2010). Absence of DOMR, a new antigen in the Dombrock blood group system that weakens expression of Dob, Gya, Hy, Joa, and DOYA antigens. Transfusion. 50(9). 2026–2031. 9 indexed citations
8.
Mayer, Beate, Nicole Thornton, Salih Yürek, et al.. (2010). New antigen in the Dombrock blood group system, DOYA, ablates expression of Doa and weakens expression of Hy, Joa, and Gya antigens. Transfusion. 50(6). 1295–1302. 10 indexed citations
9.
Hue‐Roye, Kim, Kathryn O’Shea, Roxanne M. Gillett, et al.. (2009). The low prevalence Rh antigen Bea(Rh36) is associated withRHCE*ce662C>G in exon 5, which is predicted to encode Rhce 221Arg. Vox Sanguinis. 98(3p1). e263–e268. 3 indexed citations
10.
Hue‐Roye, Kim, et al.. (2008). Novel GYP(A‐B‐A) hybrid gene in a DANE+ person who made an antibody to a high‐prevalence MNS antigen. Transfusion. 48(12). 2618–2623. 4 indexed citations
11.
Westhoff, Connie M., Sunitha Vege, Dwane E. Wylie, et al.. (2008). The JAL antigen (RH48) is the result of a change in RHCE that encodes Arg114Trp. Transfusion. 49(4). 725–732. 18 indexed citations
12.
Hue‐Roye, Kim, Christine Lomas‐Francis, Larisa Belaygorod, et al.. (2007). Three new high‐prevalence antigens in the Cromer blood group system. Transfusion. 47(9). 1621–1629. 8 indexed citations
13.
Ratliff, Judson, et al.. (2007). An alloantibody to a high-prevalence MNS antigen in a person with a GP.JL/M k phenotype. Immunohematology. 23(4). 146–149. 2 indexed citations
14.
Hashmi, Ghazala, Michael Seul, Prabhakar Vissavajjhala, et al.. (2005). A flexible array format for large‐scale, rapid blood group DNA typing. Transfusion. 45(5). 680–688. 131 indexed citations
15.
Hue‐Roye, Kim, Asok Chaudhuri, Susan Fetics, et al.. (2005). STAR: a novel high‐prevalence antigen in the Scianna blood group system. Transfusion. 45(2). 245–247. 10 indexed citations
16.
Reid, Marion E., Jill R. Storry, María Rios, et al.. (2003). DAK, a new low‐incidence antigen in the Rh blood group system. Transfusion. 43(10). 1394–1397. 18 indexed citations
17.
Storry, Jill R., Kim Hue‐Roye, Morris A. Blajchman, et al.. (2003). GUTI: a new antigen in the Cromer blood group system. Transfusion. 43(3). 340–344. 14 indexed citations
18.
Rios, María, Jill R. Storry, Kim Hue‐Roye, Amy W. Chung, & Marion E. Reid. (2002). Two new molecular bases for the Dombrock null phenotype. British Journal of Haematology. 117(3). 765–767. 18 indexed citations
19.
Rios, María, Kim Hue‐Roye, Jill R. Storry, et al.. (2001). Molecular basis of the Dombrock null phenotype. Transfusion. 41(11). 1405–1407. 20 indexed citations
20.
Rios, María, et al.. (2001). DNA analysis for the Dombrock polymorphism. Transfusion. 41(9). 1143–1146. 28 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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