John W. Heitman

2.3k total citations · 1 hit paper
37 papers, 1.7k citations indexed

About

John W. Heitman is a scholar working on Virology, Hematology and Epidemiology. According to data from OpenAlex, John W. Heitman has authored 37 papers receiving a total of 1.7k indexed citations (citations by other indexed papers that have themselves been cited), including 15 papers in Virology, 14 papers in Hematology and 11 papers in Epidemiology. Recurrent topics in John W. Heitman's work include HIV Research and Treatment (15 papers), Blood groups and transfusion (14 papers) and Blood transfusion and management (10 papers). John W. Heitman is often cited by papers focused on HIV Research and Treatment (15 papers), Blood groups and transfusion (14 papers) and Blood transfusion and management (10 papers). John W. Heitman collaborates with scholars based in United States, Canada and Poland. John W. Heitman's co-authors include Philip J. Norris, Rachael P. Jackman, Mila Lebedeva, Persephone Borrow, Andrea Stacey, Allan C. deCamp, Dongfeng Li, Steven G. Self, Qin Li and Elizabeth Taylor and has published in prestigious journals such as Journal of Clinical Investigation, Blood and PLoS ONE.

In The Last Decade

John W. Heitman

37 papers receiving 1.7k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Induction of a Striking Systemic Cytokine Cascade prior to Peak Viremia in Acute Human Immunodeficiency Virus Type 1 Infection, in Contrast to More Modest and Delayed Responses in Acute Hepatitis B and C Virus Infections

2009 555 citations Andrea Stacey, Philip J. Norris et al. Journal of Virology profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
John W. Heitman United States	20	660	611	420	387	291	37	1.7k
Eva A. Operskalski United States	24	300 0.5×	454 0.7×	816 1.9×	983 2.5×	86 0.3×	42	2.1k
P.L. Yap United Kingdom	24	434 0.7×	293 0.5×	403 1.0×	904 2.3×	193 0.7×	75	2.0k
Jack R. Davis United States	17	555 0.8×	138 0.2×	401 1.0×	344 0.9×	181 0.6×	32	1.6k
Jean‐Daniel Lelièvre France	26	1.3k 1.9×	1.3k 2.1×	775 1.8×	556 1.4×	520 1.8×	101	2.7k
D. Rigal France	19	466 0.7×	138 0.2×	98 0.2×	229 0.6×	163 0.6×	86	1.4k
J. D’Amaro Netherlands	24	700 1.1×	94 0.2×	158 0.4×	217 0.6×	104 0.4×	64	1.5k
Joanne Becker United States	18	200 0.3×	139 0.2×	170 0.4×	209 0.5×	173 0.6×	35	962
Jean‐Yves Muller France	21	505 0.8×	31 0.1×	259 0.6×	213 0.6×	187 0.6×	72	1.5k
Ann E. Hohenhaus United States	20	319 0.5×	65 0.1×	88 0.2×	90 0.2×	185 0.6×	47	1.3k
C. Gazengel France	15	255 0.4×	294 0.5×	225 0.5×	287 0.7×	166 0.6×	32	989

Countries citing papers authored by John W. Heitman

Since Specialization

Citations

This map shows the geographic impact of John W. Heitman's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by John W. Heitman with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites John W. Heitman more than expected).

Fields of papers citing papers by John W. Heitman

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by John W. Heitman. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by John W. Heitman. The network helps show where John W. Heitman may publish in the future.

Co-authorship network of co-authors of John W. Heitman

This figure shows the co-authorship network connecting the top 25 collaborators of John W. Heitman. A scholar is included among the top collaborators of John W. Heitman based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with John W. Heitman. John W. Heitman is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Kelly, Shannon, Evan Jacobs, Mars Stone, et al.. (2020). Influence of sickle cell disease on susceptibility to HIV infection. PLoS ONE. 15(4). e0218880–e0218880. 8 indexed citations

Tran, Johnson Q., et al.. (2020). Immunodeficient mice are better for modeling the transfusion of human blood components than wild-type mice. PLoS ONE. 15(7). e0237106–e0237106. 11 indexed citations

Jackman, Rachael P., John W. Heitman, & Marcus O. Muench. (2020). A small allelic variant in donor class I MHC is sufficient to induce alloantibodies following transfusion of standard or pathogen‐reduced platelets in mice. Vox Sanguinis. 115(5). 367–376. 2 indexed citations

Norris, Philip J., Ken B. Schechtman, Heather C. Inglis, et al.. (2019). Influence of blood storage age on immune and coagulation parameters in critically ill transfused patients. Transfusion. 59(4). 1223–1232. 7 indexed citations

Togarrati, Padma Priya, John W. Heitman, Rachael P. Jackman, et al.. (2019). Potential of Membranes Surrounding the Fetus as Immunoprotective Cell-Carriers for Allogeneic Transplantations. Transplantation Direct. 5(6). e460–e460. 2 indexed citations

Danesh, Ali, Heather C. Inglis, Mohamed Abdel‐Mohsen, et al.. (2018). Granulocyte-Derived Extracellular Vesicles Activate Monocytes and Are Associated With Mortality in Intensive Care Unit Patients. Frontiers in Immunology. 9. 956–956. 23 indexed citations

Keating, Sheila M., Jennifer L. Dodge, Philip J. Norris, et al.. (2017). The effect of HIV infection and HCV viremia on inflammatory mediators and hepatic injury—The Women’s Interagency HIV Study. PLoS ONE. 12(9). e0181004–e0181004. 9 indexed citations

Sousa, Francielle Tramontini Gomes de, Gyulnar Baimukanova, Marion C. Lanteri, et al.. (2017). Serum from dengue virus-infected patients with and without plasma leakage differentially affects endothelial cells barrier function in vitro. PLoS ONE. 12(6). e0178820–e0178820. 74 indexed citations

Jackman, Rachael P., Marcus O. Muench, Heather C. Inglis, et al.. (2016). Reduced MHC alloimmunization and partial tolerance protection with pathogen reduction of whole blood. Transfusion. 57(2). 337–348. 12 indexed citations

10.

Keating, Sheila M., John W. Heitman, Shiquan Wu, et al.. (2016). Magnitude and Quality of Cytokine and Chemokine Storm during Acute Infection Distinguish Nonprogressive and Progressive Simian Immunodeficiency Virus Infections of Nonhuman Primates. Journal of Virology. 90(22). 10339–10350. 23 indexed citations

11.

Barbour, Jason D., Emilie Jalbert, Dominic C. Chow, et al.. (2013). Reduced CD14 expression on classical monocytes and vascular endothelial adhesion markers independently associate with carotid artery intima media thickness in chronically HIV-1 infected adults on virologically suppressive anti-retroviral therapy. Atherosclerosis. 232(1). 52–58. 29 indexed citations

12.

Curtis, Kelly A., Melanie S. Kennedy, Sheila M. Keating, et al.. (2013). Inter-Laboratory Assessment of a Prototype Multiplex Kit for Determination of Recent HIV-1 Infection. PLoS ONE. 8(10). e77765–e77765. 7 indexed citations

13.

Owen, Rachel E., John W. Heitman, Dale F. Hirschkorn, et al.. (2010). HIV+ elite controllers have low HIV-specific T-cell activation yet maintain strong, polyfunctional T-cell responses. AIDS. 24(8). 1095–1105. 101 indexed citations

14.

Stacey, Andrea, Philip J. Norris, Qin Li, et al.. (2009). Induction of a Striking Systemic Cytokine Cascade prior to Peak Viremia in Acute Human Immunodeficiency Virus Type 1 Infection, in Contrast to More Modest and Delayed Responses in Acute Hepatitis B and C Virus Infections. Journal of Virology. 83(8). 3719–3733. 555 indexed citations breakdown →

15.

Lanteri, Marion C., Katie M. O’Brien, Whitney E. Purtha, et al.. (2009). Tregs control the development of symptomatic West Nile virus infection in humans and mice. Journal of Clinical Investigation. 119(11). 3266–77. 176 indexed citations

16.

Lanteri, Marion C., John W. Heitman, Rachel E. Owen, et al.. (2008). Comprehensive Analysis of West Nile Virus–Specific T Cell Responses in Humans. The Journal of Infectious Diseases. 197(9). 1296–1306. 36 indexed citations

17.

Owen, Rachel E., Elizabeth Sinclair, Brinda Emu, et al.. (2007). Loss of T cell responses following long-term cryopreservation. Journal of Immunological Methods. 326(1-2). 93–115. 75 indexed citations

18.

Norris, Philip J., Jennifer D. Stone, John W. Heitman, et al.. (2005). Antagonism of HIV-specific CD4+ T cells by C-terminal truncation of a minimum epitope. Molecular Immunology. 43(9). 1349–1357. 12 indexed citations

19.

Busch, Michael P., Simone A. Glynn, David J. Wright, et al.. (2005). Relative sensitivities of licensed nucleic acid amplification tests for detection of viremia in early human immunodeficiency virus and hepatitis C virus infection. Transfusion. 45(12). 1853–1863. 35 indexed citations

20.

Stromberg, Robert R., et al.. (1998). Distribution of HIV type 1 (HIV‐1) in blood components: detection and significance of high levels of HIV‐1 associated with platelets. Transfusion. 38(6). 580–588. 31 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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