Takaki Watanabe

931 total citations
22 papers, 512 citations indexed

About

Takaki Watanabe is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Cognitive Neuroscience. According to data from OpenAlex, Takaki Watanabe has authored 22 papers receiving a total of 512 indexed citations (citations by other indexed papers that have themselves been cited), including 13 papers in Molecular Biology, 12 papers in Cellular and Molecular Neuroscience and 4 papers in Cognitive Neuroscience. Recurrent topics in Takaki Watanabe's work include Neuroscience and Neuropharmacology Research (10 papers), Ion channel regulation and function (4 papers) and Genetics and Neurodevelopmental Disorders (4 papers). Takaki Watanabe is often cited by papers focused on Neuroscience and Neuropharmacology Research (10 papers), Ion channel regulation and function (4 papers) and Genetics and Neurodevelopmental Disorders (4 papers). Takaki Watanabe collaborates with scholars based in Japan, United States and France. Takaki Watanabe's co-authors include Masanobu Kano, Naofumi Uesaka, Masahiko Watanabe, Kazuto Sakoori, Kohtarou Konno, Hiromi Hirata, Yasuhito Sasaki, Toshimitsu Momose, J Nishikawa and Ikuo Yokoyama and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Nature Communications and Neuron.

In The Last Decade

Takaki Watanabe

18 papers receiving 503 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Takaki Watanabe Japan 10 252 127 98 96 82 22 512
Jaime Boero United States 10 243 1.0× 116 0.9× 58 0.6× 102 1.1× 41 0.5× 13 548
Makoto Takemoto Japan 11 198 0.8× 235 1.9× 144 1.5× 45 0.5× 45 0.5× 31 509
John D. Tompkins United States 15 398 1.6× 370 2.9× 61 0.6× 68 0.7× 194 2.4× 34 805
Adele Jackson Canada 11 222 0.9× 139 1.1× 51 0.5× 113 1.2× 66 0.8× 14 544
Daniel W. Meechan United States 13 550 2.2× 138 1.1× 122 1.2× 277 2.9× 17 0.2× 17 761
István Adorján Hungary 12 326 1.3× 204 1.6× 60 0.6× 60 0.6× 24 0.3× 29 686
Adolfo E. Cuadra United States 13 517 2.1× 190 1.5× 43 0.4× 49 0.5× 314 3.8× 26 885
Henning Fröhlich Germany 12 225 0.9× 34 0.3× 61 0.6× 73 0.8× 33 0.4× 20 411
Karen M. J. van Loo Germany 15 320 1.3× 231 1.8× 39 0.4× 89 0.9× 19 0.2× 35 595
Yoshimitsu Tokunaga Japan 9 263 1.0× 228 1.8× 82 0.8× 63 0.7× 56 0.7× 19 567

Countries citing papers authored by Takaki Watanabe

Since Specialization
Citations

This map shows the geographic impact of Takaki Watanabe's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Takaki Watanabe with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Takaki Watanabe more than expected).

Fields of papers citing papers by Takaki Watanabe

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Takaki Watanabe. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Takaki Watanabe. The network helps show where Takaki Watanabe may publish in the future.

Co-authorship network of co-authors of Takaki Watanabe

This figure shows the co-authorship network connecting the top 25 collaborators of Takaki Watanabe. A scholar is included among the top collaborators of Takaki Watanabe based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Takaki Watanabe. Takaki Watanabe is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Okuno, Y., et al.. (2025). Synaptic transmission is dispensable for selecting the winner input but is crucial for the subsequent events of synapse elimination. Proceedings of the National Academy of Sciences. 122(34). e2416797122–e2416797122.
2.
Zhang, Jianling, Takaki Watanabe, Taisuke Miyazaki, et al.. (2025). The transcription factor ZFP64 promotes activity-dependent synapse elimination during postnatal cerebellar development. iScience. 28(6). 112746–112746.
3.
Watanabe, Takaki & Masanobu Kano. (2024). Molecular and cellular mechanisms of developmental synapse elimination in the cerebellum: Involvement of autism spectrum disorder-related genes. Proceedings of the Japan Academy Series B. 100(9). 508–523. 2 indexed citations
4.
Okuno, Y., Kazuto Sakoori, Miwako Yamasaki, et al.. (2023). PTPδ is a presynaptic organizer for the formation and maintenance of climbing fiber to Purkinje cell synapses in the developing cerebellum. Frontiers in Molecular Neuroscience. 16. 1206245–1206245. 4 indexed citations
5.
6.
Toriumi, Kazuya, Stefano Berto, Shin Koike, et al.. (2021). Combined glyoxalase 1 dysfunction and vitamin B6 deficiency in a schizophrenia model system causes mitochondrial dysfunction in the prefrontal cortex. Redox Biology. 45. 102057–102057. 13 indexed citations
7.
Sakoori, Kazuto, Kohtarou Konno, Takaki Watanabe, et al.. (2020). Autism spectrum disorder-like behavior caused by reduced excitatory synaptic transmission in pyramidal neurons of mouse prefrontal cortex. Nature Communications. 11(1). 5140–5140. 94 indexed citations
8.
Hori, Kei, Ryo Aoki, Nariko Arimura, et al.. (2020). AUTS2 Governs Cerebellar Development, Purkinje Cell Maturation, Motor Function and Social Communication. iScience. 23(12). 101820–101820. 29 indexed citations
9.
Sakoori, Kazuto, Takaki Watanabe, Yusuke Kishi, et al.. (2020). Setd1a Insufficiency in Mice Attenuates Excitatory Synaptic Function and Recapitulates Schizophrenia-Related Behavioral Abnormalities. Cell Reports. 32(11). 108126–108126. 42 indexed citations
10.
11.
Uesaka, Naofumi, Manabu Abe, Kohtarou Konno, et al.. (2018). Retrograde Signaling from Progranulin to Sort1 Counteracts Synapse Elimination in the Developing Cerebellum. Neuron. 97(4). 796–805.e5. 29 indexed citations
12.
Sugio, Shouta, Kenji F. Tanaka, Takaki Watanabe, et al.. (2018). Ectopic positioning of Bergmann glia and impaired cerebellar wiring in Mlc1‐over‐expressing mice. Journal of Neurochemistry. 147(3). 344–360. 2 indexed citations
13.
Kano, Masanobu, Takaki Watanabe, Naofumi Uesaka, & Masahiko Watanabe. (2018). Multiple Phases of Climbing Fiber Synapse Elimination in the Developing Cerebellum. The Cerebellum. 17(6). 722–734. 56 indexed citations
14.
Aikawa, Tomonori, Takaki Watanabe, Taisuke Miyazaki, et al.. (2017). Alternative splicing in the C-terminal tail of Cav2.1 is essential for preventing a neurological disease in mice. Human Molecular Genetics. 26(16). 3094–3104. 8 indexed citations
15.
16.
Watanabe, Takaki, et al.. (2013). Coexpression of auxiliary Kvβ2 subunits with Kv1.1 channels is required for developmental acquisition of unique firing properties of zebrafish Mauthner cells. Journal of Neurophysiology. 111(6). 1153–1164. 14 indexed citations
17.
Hirata, Hiromi, Takaki Watanabe, Jun Hatakeyama, et al.. (2007). Zebrafishrelatively relaxedmutants have a ryanodine receptor defect, show slow swimming and provide a model of multi-minicore disease. Development. 134(15). 2771–2781. 97 indexed citations
18.
Teramoto, Atsushi, et al.. (1993). [Clinical application of 11C-NMSP to the patients with pituitary adenoma other than prolactinoma].. PubMed. 30(6). 627–35.
19.
Takayama, Satoshi, Takaki Watanabe, Yutaro Akiyama, et al.. (1986). Reproductive toxicity of ofloxacin.. PubMed. 36(8). 1244–8. 20 indexed citations
20.
Watanabe, Takaki, et al.. (1978). Teratogenicity study of oxepinac in mice and rabbits.. PubMed. 28(3). 451–5. 2 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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