Moe Tamano

620 total citations
17 papers, 452 citations indexed

About

Moe Tamano is a scholar working on Molecular Biology, Genetics and Pediatrics, Perinatology and Child Health. According to data from OpenAlex, Moe Tamano has authored 17 papers receiving a total of 452 indexed citations (citations by other indexed papers that have themselves been cited), including 13 papers in Molecular Biology, 8 papers in Genetics and 2 papers in Pediatrics, Perinatology and Child Health. Recurrent topics in Moe Tamano's work include CRISPR and Genetic Engineering (9 papers), Pluripotent Stem Cells Research (4 papers) and Genetic and Clinical Aspects of Sex Determination and Chromosomal Abnormalities (4 papers). Moe Tamano is often cited by papers focused on CRISPR and Genetic Engineering (9 papers), Pluripotent Stem Cells Research (4 papers) and Genetic and Clinical Aspects of Sex Determination and Chromosomal Abnormalities (4 papers). Moe Tamano collaborates with scholars based in Japan, United Kingdom and United States. Moe Tamano's co-authors include Shuji Takada, Masafumi Inui, Hiroshi Asahara, Tomoko Kato, Satoshi Yamashita, Mami Miyado, Maki Igarashi, Maki Fukami, Atsushi Kubo and Satoshi Hara and has published in prestigious journals such as PLoS ONE, Scientific Reports and International Journal of Molecular Sciences.

In The Last Decade

Moe Tamano

16 papers receiving 443 citations

Peers

Countries citing papers authored by Moe Tamano

Since Specialization

Citations

This map shows the geographic impact of Moe Tamano's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Moe Tamano with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Moe Tamano more than expected).

Fields of papers citing papers by Moe Tamano

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Moe Tamano. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Moe Tamano. The network helps show where Moe Tamano may publish in the future.

Co-authorship network of co-authors of Moe Tamano

This figure shows the co-authorship network connecting the top 25 collaborators of Moe Tamano. A scholar is included among the top collaborators of Moe Tamano based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Moe Tamano. Moe Tamano is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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17 of 17 papers shown

#	Work	Indexed citations
1	FMR1 mutant marmosets show fragile X syndrome phenotypes 2025 ·Cell Reports ·(unknown), Zefeng Wei, Harumi Nakao, Yukiko Abe, Motoki Goto, Moe Tamano, (unknown), Kimiko Shimizu, Kazuki Nakao, Yuki Sugaya, Kosuke Itoh, Masanobu Kano, Atsu Aiba	0
2	Efficient marmoset genome engineering by autologous embryo transfer and CRISPR/Cas9 technology 2021 ·Scientific Reports ·Yukiko Abe, Harumi Nakao, Motoki Goto, Moe Tamano, Michinori Koebis, Kazuki Nakao, Atsu Aiba	8
3	Deletion of a Seminal Gene Cluster Reinforces a Crucial Role of SVS2 in Male Fertility 2019 ·International Journal of Molecular Sciences ·(unknown), Masafumi Inui, Woojin Kang, Moe Tamano, Tingwei Cai, Shuji Takada, Taku Hibino, Manabu Yoshida, Kaoru Yoshida, Hiroshi Okada, Teruaki Iwamoto, Kenji Miyado, Natsuko Kawano	11
4	Mapping of a responsible region for sex reversal upstream of Sox9 by production of mice with serial deletion in a genomic locus 2018 ·Scientific Reports ·Yuya Ogawa, (unknown), Satoshi Hara, Moe Tamano, (unknown), Tomoko Kato, Shuji Takada	19
5	A tandem repeat array in IG-DMR is essential for imprinting of paternal allele at the Dlk1–Dio3 domain during embryonic development 2018 ·Human Molecular Genetics ·Takeshi Saito, Satoshi Hara, Tomoko Kato, Moe Tamano, (unknown), Hiroshi Asahara, Shuji Takada	22
6	Creation of mutant mice with megabase-sized deletions containing custom-designed breakpoints by means of the CRISPR/Cas9 system 2017 ·Scientific Reports ·Tomoko Kato, Satoshi Hara, Yuji Goto, Yuya Ogawa, (unknown), Souichirou Kubota, Moe Tamano, (unknown), Shuji Takada	16
7	CRISPR/Cas9-mediated simultaneous knockout of Dmrt1 and Dmrt3 does not recapitulate the 46,XY gonadal dysgenesis observed in 9p24.3 deletion patients 2017 ·Biochemistry and Biophysics Reports ·Masafumi Inui, Moe Tamano, Tomoko Kato, Shuji Takada	18
8	The p.R92W variant of NR5A1/Nr5a1 induces testicular development of 46,XX gonads in humans, but not in mice: phenotypic comparison of human patients and mutation-induced mice 2016 ·Biology of Sex Differences ·Mami Miyado, Masafumi Inui, Maki Igarashi, Yûkô Fukui, Kei Takasawa, (unknown), Junko Kanno, Kenichi Kashimada, Kenji Miyado, Moe Tamano, Tsutomu Ogata, Shuji Takada, Maki Fukami	19
9	Data supporting the role of Fyn in initiating myelination in the peripheral nervous system 2016 ·Data in Brief ·Yuki Miyamoto, Moe Tamano, Tomohiro Torii, (unknown), Kazuaki Nakamura, Akito Tanoue, Shuji Takada, Junji Yamauchi	5
10	Cord blood-derived endothelial colony-forming cell function is disrupted in congenital diaphragmatic hernia 2016 ·American Journal of Physiology-Lung Cellular and Molecular Physiology ·(unknown), (unknown), (unknown), (unknown), Nobuyuki Watanabe, Tomoko Kato, Moe Tamano, (unknown), Shuji Takada, Yushi Ito, Akihiro Umezawa, Masahiko Kuroda	11
11	Deletion of conserved sequences in IG-DMR at <i>Dlk1</i>-<i>Gtl2</i> locus suggests their involvement in expression of paternally expressed genes in mice 2016 ·Journal of Reproduction and Development ·Takeshi Saito, Satoshi Hara, Moe Tamano, Hiroshi Asahara, Shuji Takada	6
12	Microinjection-based generation of mutant mice with a double mutation and a 0.5 Mb deletion in their genome by the CRISPR/Cas9 system 2016 ·Journal of Reproduction and Development ·Satoshi Hara, Tomoko Kato, Yuji Goto, Souichirou Kubota, Moe Tamano, (unknown), Shuji Takada	17
13	Utilization of the CRISPR/Cas9 system for the efficient production of mutant mice using crRNA/tracrRNA with Cas9 nickase and FokI-dCas9 2016 ·EXPERIMENTAL ANIMALS ·(unknown), Moe Tamano, Satoshi Hara, Tomoko Kato, Masato Kinoshita, Shuji Takada	8
14	Generation of mutant mice via the CRISPR/Cas9 system using FokI-dCas9 2015 ·Scientific Reports ·Satoshi Hara, Moe Tamano, Satoshi Yamashita, Tomoko Kato, Takeshi Saito, Tetsushi Sakuma, Takashi Yamamoto, Masafumi Inui, Shuji Takada	36
15	Rapid generation of mouse models with defined point mutations by the CRISPR/Cas9 system 2014 ·Scientific Reports ·Masafumi Inui, Mami Miyado, Maki Igarashi, Moe Tamano, Atsushi Kubo, Satoshi Yamashita, Hiroshi Asahara, Maki Fukami, Shuji Takada	177
16	Production of Sry knockout mouse using TALEN via oocyte injection 2013 ·Scientific Reports ·Tomoko Kato, Kohei Miyata, (unknown), Satoshi Yamashita, Moe Tamano, Kento Miura, Yoshiakira Kanai, Shingo Miyamoto, Tetsushi Sakuma, Takashi Yamamoto, Masafumi Inui, Takefumi Kikusui, Hiroshi Asahara, Shuji Takada	50
17	Targeted Gene Deletion of miRNAs in Mice by TALEN System 2013 ·PLoS ONE ·Shuji Takada, Tempei Sato, Yoshiaki Ito, Satoshi Yamashita, Tomoko Kato, Miyuri Kawasumi, Masami Kanai‐Azuma, Arisa Igarashi, Tomomi Kato, Moe Tamano, Hiroshi Asahara	29

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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