Astrid Rohlmann

3.9k citations
35 papers · 3.0k indexed · 1 hit paper · h-index 22

Impact in

Papers in

Astrid Rohlmann

35 papers receiving 2.9k citations

Hit Papers

α-Neurexins couple Ca2+ channels to synaptic vesicle exocytosis 2003 · 527 citations
5272003202620102018100200300400500

Peers

Astrid Rohlmann
Comparison fields: 5 of 106
  • Cellular and Molecular Neuroscience 988
  • Cell Biology 609
  • Developmental Neuroscience 144
  • Molecular Biology 1.6k
  • Neurology 189
Replace Toshifumi Tomoda with:
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Shinichiro Taya Japan
Concepción Lillo United States
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Citations per field
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Citations per year

Countries citing papers authored by Astrid Rohlmann

Since Specialization
Citations

This map shows the geographic impact of Astrid Rohlmann's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Astrid Rohlmann with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Astrid Rohlmann more than expected).

Fields of papers citing papers by Astrid Rohlmann

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Astrid Rohlmann. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Astrid Rohlmann. The network helps show where Astrid Rohlmann may publish in the future.

Co-authorship network

The 25 scholars most cited alongside Astrid Rohlmann, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Astrid Rohlmann Line = papers co-authored together Astrid Rohlmann links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown
#Work
1 20241
2 202316
3 20228
4 20223
5 202115
6 202028
7 202013
8 201678
9 201515
10 201561
11 201155
12 200961
13 200867
14 200770
15 2005118
16 20007
17 199589
18 199462
19 199352
20 199213

About Astrid Rohlmann

Astrid Rohlmann is a scholar working on Cellular and Molecular Neuroscience, Cell Biology, Developmental Neuroscience, Endocrine and Autonomic Systems and Molecular Biology, having authored 35 papers that have together received 3.0k indexed citations. Recurring topics across this work include Neuroscience and Neuropharmacology Research (16 papers), Cellular transport and secretion (10 papers), Connexins and lens biology (6 papers), Photoreceptor and optogenetics research (3 papers), Retinal Development and Disorders (3 papers), Ion channel regulation and function (3 papers), Mitochondrial Function and Pathology (2 papers) and Neurobiology and Insect Physiology Research (2 papers). The work is most often cited by research in Cellular and Molecular Neuroscience (988 citations), Cell Biology (609 citations), Developmental Neuroscience (144 citations), Molecular Biology (1.6k citations) and Neurology (189 citations). Astrid Rohlmann has collaborated with scholars based in Germany, United States and Sweden. Frequent co-authors include Robert E. Hammer, Markus Missler, Joachim Herz, Michael Gotthardt, Thomas E. Willnow, Thomas C. Südhof, Weiqi Zhang, Kurt Gottmann, Dennis K. Burns and Jan Hilpert. Their work appears in journals such as Journal of Neuroscience, Proceedings of the National Academy of Sciences, Nature Communications, Frontiers in Neuroanatomy and Glia.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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