Asiel Arce-Sillas

476 total citations
16 papers, 353 citations indexed

About

Asiel Arce-Sillas is a scholar working on Neurology, Pathology and Forensic Medicine and Neurology. According to data from OpenAlex, Asiel Arce-Sillas has authored 16 papers receiving a total of 353 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Neurology, 6 papers in Pathology and Forensic Medicine and 6 papers in Neurology. Recurrent topics in Asiel Arce-Sillas's work include Neuroinflammation and Neurodegeneration Mechanisms (7 papers), Parasitic infections in humans and animals (6 papers) and Parkinson's Disease Mechanisms and Treatments (6 papers). Asiel Arce-Sillas is often cited by papers focused on Neuroinflammation and Neurodegeneration Mechanisms (7 papers), Parasitic infections in humans and animals (6 papers) and Parkinson's Disease Mechanisms and Treatments (6 papers). Asiel Arce-Sillas collaborates with scholars based in Mexico, Germany and Portugal. Asiel Arce-Sillas's co-authors include Laura Adalid‐Peralta, Graciela Cárdenas, Edda Sciutto, Gladis Fragoso, Juan Rodríguez‐Ramírez, Marie‐Catherine Boll, Edgar Sevilla-­Reyes, Agnès Fleury, Marisela Hernández and Laura Uribe-Figueroa and has published in prestigious journals such as SHILAP Revista de lepidopterología, Scientific Reports and Journal of Neuroinflammation.

In The Last Decade

Asiel Arce-Sillas

15 papers receiving 347 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Asiel Arce-Sillas Mexico 8 103 94 83 83 63 16 353
Judith Strauß Austria 7 134 1.3× 74 0.8× 73 0.9× 50 0.6× 34 0.5× 9 345
Michael Happel Germany 7 187 1.8× 57 0.6× 135 1.6× 53 0.6× 23 0.4× 13 414
Marius Birlea United States 12 97 0.9× 24 0.3× 172 2.1× 106 1.3× 17 0.3× 22 418
Sushil Bandodkar Australia 13 104 1.0× 51 0.5× 95 1.1× 200 2.4× 6 0.1× 25 548
Christina N. Como United States 11 53 0.5× 60 0.6× 14 0.2× 38 0.5× 18 0.3× 21 279
J. Robert Chang United States 7 130 1.3× 102 1.1× 102 1.2× 30 0.4× 7 0.1× 12 538
Ingo Gerhauser Germany 6 105 1.0× 189 2.0× 17 0.2× 65 0.8× 7 0.1× 12 375
Carmen M. Doom United States 11 239 2.3× 34 0.4× 21 0.3× 62 0.7× 32 0.5× 12 663
Julia A. Loos Argentina 11 40 0.4× 25 0.3× 193 2.3× 37 0.4× 104 1.7× 20 325
Christopher van Belle United States 6 137 1.3× 24 0.3× 125 1.5× 34 0.4× 16 0.3× 7 507

Countries citing papers authored by Asiel Arce-Sillas

Since Specialization
Citations

This map shows the geographic impact of Asiel Arce-Sillas's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Asiel Arce-Sillas with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Asiel Arce-Sillas more than expected).

Fields of papers citing papers by Asiel Arce-Sillas

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Asiel Arce-Sillas. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Asiel Arce-Sillas. The network helps show where Asiel Arce-Sillas may publish in the future.

Co-authorship network of co-authors of Asiel Arce-Sillas

This figure shows the co-authorship network connecting the top 25 collaborators of Asiel Arce-Sillas. A scholar is included among the top collaborators of Asiel Arce-Sillas based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Asiel Arce-Sillas. Asiel Arce-Sillas is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

16 of 16 papers shown
1.
Arce-Sillas, Asiel, et al.. (2025). Advanced clinical stage Parkinson’s disease is linked to proinflammatory and regulatory blood cell populations. Neurological Sciences. 46(10). 5081–5091.
2.
Arce-Sillas, Asiel, et al.. (2025). Neurodegeneration models in Parkinson’s disease: cellular and molecular paths to neuron death. Behavioral and Brain Functions. 21(1). 14–14. 2 indexed citations
4.
Arce-Sillas, Asiel, et al.. (2021). Increased Tc17 cell levels and imbalance of naïve/effector immune response in Parkinson’s disease patients in a two-year follow-up: a case control study. Journal of Translational Medicine. 19(1). 378–378. 12 indexed citations
5.
Espitia, Clara, Adrián Ochoa‐Leyva, Filiberto Sánchez, et al.. (2021). A novel, sequencing-free strategy for the functional characterization of Taenia solium proteomic fingerprint. PLoS neglected tropical diseases. 15(2). e0009104–e0009104. 4 indexed citations
6.
Arce-Sillas, Asiel, et al.. (2021). Pro- and anti-inflammatory response in neurological disorders associated to anti-glutamate decarboxylase antibodies. Journal of Neuroimmunology. 355. 577550–577550. 1 indexed citations
7.
Adalid‐Peralta, Laura, Jocelyne Demengeot, Joel Armando Vázquez-Pérez, et al.. (2021). Regulatory T Cells as an Escape Mechanism to the Immune Response in Taenia crassiceps Infection. Frontiers in Cellular and Infection Microbiology. 11. 630583–630583. 6 indexed citations
8.
Arce-Sillas, Asiel, Marie‐Catherine Boll, Elizabeth García-García, et al.. (2020). Immunomodulatory effect and clinical outcome in Parkinson's disease patients on levodopa-pramipexole combo therapy: A two-year prospective study. Journal of Neuroimmunology. 347. 577328–577328. 20 indexed citations
9.
Arce-Sillas, Asiel, et al.. (2019). Biological role of excretory–secretory proteins in endemic parasites of Latin America and the Caribbean. Journal of Helminthology. 94. e53–e53. 4 indexed citations
10.
Arce-Sillas, Asiel, Edgar Sevilla-­Reyes, Marie‐Catherine Boll, et al.. (2019). Regulatory impairment in untreated Parkinson’s disease is not restricted to Tregs: other regulatory populations are also involved. Journal of Neuroinflammation. 16(1). 212–212. 74 indexed citations
11.
Arce-Sillas, Asiel, Edgar Sevilla-­Reyes, Marie‐Catherine Boll, et al.. (2019). Expression of Dopamine Receptors in Immune Regulatory Cells. NeuroImmunoModulation. 26(3). 159–166. 24 indexed citations
12.
Arce-Sillas, Asiel, Graciela Cárdenas, Marisela Hernández, et al.. (2018). Treatment-Resistant Human Extraparenchymal Neurocysticercosis: An Immune-Inflammatory Approach to Cysticidal Treatment Outcome. NeuroImmunoModulation. 25(2). 103–109. 3 indexed citations
13.
Adalid‐Peralta, Laura, Gabriela Rosas, Asiel Arce-Sillas, et al.. (2017). Effect of Transforming Growth Factor-β upon Taenia solium and Taenia crassiceps Cysticerci. Scientific Reports. 7(1). 12345–12345. 23 indexed citations
14.
Arce-Sillas, Asiel, et al.. (2016). Regulatory T Cells: Molecular Actions on Effector Cells in Immune Regulation. Journal of Immunology Research. 2016. 1–12. 117 indexed citations
15.
Arce-Sillas, Asiel, Graciela Cárdenas, Gladis Fragoso, et al.. (2015). Interleukin 10 and dendritic cells are the main suppression mediators of regulatory T cells in human neurocysticercosis. Clinical & Experimental Immunology. 183(2). 271–279. 32 indexed citations
16.
Adalid‐Peralta, Laura, Asiel Arce-Sillas, Gladis Fragoso, et al.. (2013). Cysticerci Drive Dendritic Cells to PromoteIn VitroandIn VivoTregs Differentiation. SHILAP Revista de lepidopterología. 2013. 1–9. 28 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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