Jocelyne Demengeot
- Immunology top 0.5%
- Molecular Biology top 5%
- Oncology top 5%
- Genetics top 5%
- Rheumatology top 2%
- Co-authors
- Íris CaramalhoSantiago ZelenayMatthias HauryShohei HoriSandra GarcêsThiago CarvalhoFrederick W. AltT Blunt
- Topics
- T-cell and B-cell Immunology (45 papers)Immune Cell Function and Interaction (42 papers)Immunotherapy and Immune Responses (23 papers)
- Cited by
- ImmunologyOncologyMolecular Biology
- Partner nations
- PortugalFranceUnited States
In The Last Decade
Jocelyne Demengeot
80 papers receiving 6.5k citations
Hit Papers
Peers
Comparison fields: 5 of 131
- Immunology 3.7k
- Molecular Biology 2.1k
- Oncology 949
- Genetics 703
- Rheumatology 455
Countries citing papers authored by Jocelyne Demengeot
This map shows the geographic impact of Jocelyne Demengeot's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jocelyne Demengeot with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jocelyne Demengeot more than expected).
Fields of papers citing papers by Jocelyne Demengeot
This network shows the impact of papers produced by Jocelyne Demengeot. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jocelyne Demengeot. The network helps show where Jocelyne Demengeot may publish in the future.
Co-authorship network of co-authors of Jocelyne Demengeot
This figure shows the co-authorship network connecting the top 25 collaborators of Jocelyne Demengeot. A scholar is included among the top collaborators of Jocelyne Demengeot based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jocelyne Demengeot. Jocelyne Demengeot is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 1 | |
| 2 | 1 | |
| 3 | 3 | |
| 4 | 14 | |
| 5 | 0 | |
| 6 | 61 | |
| 7 | 60 | |
| 8 | 2 | |
| 9 | 60 | |
| 10 | 17 | |
| 11 | 4 | |
| 12 | 54 | |
| 13 | EMMA: The European Mouse Mutant Archive. | 1 |
| 14 | 117 | |
| 15 | 18 | |
| 16 | 34 | |
| 17 | 12 | |
| 18 | 205 | |
| 19 | 24 | |
| 20 | Ku80: product of the XRCC5 gene and its role in DNA repair and V(D)J recombinationbreakdown → | 555 |
About Jocelyne Demengeot
Jocelyne Demengeot is a scholar working on Immunology, Rheumatology and Infectious Diseases, having authored 81 papers that have together received 6.6k indexed citations. Recurring topics across this work include T-cell and B-cell Immunology (45 papers), Immune Cell Function and Interaction (42 papers) and Immunotherapy and Immune Responses (23 papers). The work is most often cited by research in Immunology (3.7k citations), Oncology (949 citations) and Molecular Biology (2.1k citations). Jocelyne Demengeot has collaborated with scholars based in Portugal, France and United States. Frequent co-authors include Íris Caramalho, Santiago Zelenay, Matthias Haury, Shohei Hori, Sandra Garcês, Thiago Carvalho, Frederick W. Alt, T Blunt, Stephen P. Jackson and Tanya Gottlieb. Their work appears in journals such as Science, Cell and Proceedings of the National Academy of Sciences.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.