Arnold D. Essenburg

1.8k total citations
34 papers, 1.6k citations indexed

About

Arnold D. Essenburg is a scholar working on Molecular Biology, Surgery and Cancer Research. According to data from OpenAlex, Arnold D. Essenburg has authored 34 papers receiving a total of 1.6k indexed citations (citations by other indexed papers that have themselves been cited), including 23 papers in Molecular Biology, 20 papers in Surgery and 5 papers in Cancer Research. Recurrent topics in Arnold D. Essenburg's work include Cholesterol and Lipid Metabolism (18 papers), Lipoproteins and Cardiovascular Health (14 papers) and Peroxisome Proliferator-Activated Receptors (10 papers). Arnold D. Essenburg is often cited by papers focused on Cholesterol and Lipid Metabolism (18 papers), Lipoproteins and Cardiovascular Health (14 papers) and Peroxisome Proliferator-Activated Receptors (10 papers). Arnold D. Essenburg collaborates with scholars based in United States, United Kingdom and Russia. Arnold D. Essenburg's co-authors include C L Bisgaier, Charles L. Bisgaier, Katriina Aalto‐Setälä, Jan L. Breslow, Peter Weinstock, Mark R. Emmerling, Alex E. Roher, Yu‐Min Kuo, Denise A. Drumm and Heather C. Lampert and has published in prestigious journals such as Journal of Clinical Investigation, Biochemical and Biophysical Research Communications and Journal of Medicinal Chemistry.

In The Last Decade

Arnold D. Essenburg

34 papers receiving 1.4k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Arnold D. Essenburg United States	20	692	679	372	321	249	34	1.6k
Mark C. Kowala United States	24	618 0.9×	640 0.9×	273 0.7×	362 1.1×	385 1.5×	56	1.9k
J.C. Fruchart France	18	656 0.9×	867 1.3×	611 1.6×	289 0.9×	211 0.8×	44	1.9k
Masataka Yokode Japan	17	684 1.0×	533 0.8×	234 0.6×	296 0.9×	205 0.8×	45	2.0k
Toshihiko Fujiwara Japan	16	328 0.5×	859 1.3×	497 1.3×	337 1.0×	358 1.4×	21	1.6k
Bruce J. Auerbach United States	21	605 0.9×	480 0.7×	407 1.1×	153 0.5×	175 0.7×	40	1.4k
Susan Butler United States	9	704 1.0×	653 1.0×	246 0.7×	283 0.9×	212 0.9×	16	2.7k
Uday Saxena United States	26	542 0.8×	649 1.0×	698 1.9×	214 0.7×	560 2.2×	54	2.1k
William J. Chiou United States	23	418 0.6×	1.2k 1.8×	360 1.0×	736 2.3×	254 1.0×	57	2.2k
Mario Campagnola Italy	18	297 0.4×	515 0.8×	275 0.7×	232 0.7×	236 0.9×	24	1.9k
Louis G. Lange United States	22	661 1.0×	980 1.4×	241 0.6×	192 0.6×	142 0.6×	43	2.2k

Countries citing papers authored by Arnold D. Essenburg

Since Specialization

Citations

This map shows the geographic impact of Arnold D. Essenburg's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Arnold D. Essenburg with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Arnold D. Essenburg more than expected).

Fields of papers citing papers by Arnold D. Essenburg

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Arnold D. Essenburg. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Arnold D. Essenburg. The network helps show where Arnold D. Essenburg may publish in the future.

Co-authorship network of co-authors of Arnold D. Essenburg

This figure shows the co-authorship network connecting the top 25 collaborators of Arnold D. Essenburg. A scholar is included among the top collaborators of Arnold D. Essenburg based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Arnold D. Essenburg. Arnold D. Essenburg is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Hanselman, Jeffrey C., et al.. (2001). Expression of the mRNA encoding truncated PPARα does not correlate with hepatic insensitivity to peroxisome proliferators. Molecular and Cellular Biochemistry. 217(1-2). 91–97. 19 indexed citations

Kuo, Yu‐Min, Mark R. Emmerling, Charles L. Bisgaier, et al.. (1998). Elevated Low-Density Lipoprotein in Alzheimer's Disease Correlates with Brain Aβ 1–42 Levels. Biochemical and Biophysical Research Communications. 252(3). 711–715. 265 indexed citations

Bisgaier, Charles L., Arnold D. Essenburg, Bruce J. Auerbach, et al.. (1998). A novel compound that elevates high density lipoprotein and activates the peroxisome proliferator activated receptor. Journal of Lipid Research. 39(1). 17–30. 53 indexed citations

Essenburg, Arnold D., Katherine L. Hamelehle, Ann Holmes, et al.. (1997). Inhibitors of acyl-CoA:cholesterol acyltransferase: novel trisubstituted ureas as hypocholesterolemic agents. Bioorganic & Medicinal Chemistry. 5(4). 739–747. 3 indexed citations

Bisgaier, C L, Arnold D. Essenburg, Bruce J. Auerbach, et al.. (1997). Attenuation of plasma low density lipoprotein cholesterol by select 3-hydroxy-3-methylglutaryl coenzyme A reductase inhibitors in mice devoid of low density lipoprotein receptors. Journal of Lipid Research. 38(12). 2502–2515. 66 indexed citations

Rodrigueza, Wendi V., Arnold D. Essenburg, Michael E. Pape, et al.. (1997). Large Versus Small Unilamellar Vesicles Mediate Reverse Cholesterol Transport In Vivo Into Two Distinct Hepatic Metabolic Pools. Arteriosclerosis Thrombosis and Vascular Biology. 17(10). 2132–2139. 41 indexed citations

Krause, Brian R., Arnold D. Essenburg, Karen A. Kieft, et al.. (1996). Opposite effects of bezafibrate and gemfibrozil in both normal and hypertriglyceridemic rats. Atherosclerosis. 127(1). 91–101. 18 indexed citations

Weinstock, Peter, C L Bisgaier, Katriina Aalto‐Setälä, et al.. (1995). Severe hypertriglyceridemia, reduced high density lipoprotein, and neonatal death in lipoprotein lipase knockout mice. Mild hypertriglyceridemia with impaired very low density lipoprotein clearance in heterozygotes.. Journal of Clinical Investigation. 96(6). 2555–2568. 259 indexed citations

Roark, W. Howard, Janak Padia, Gary L. Bolton, et al.. (1995). Bioisosterism in drug design: Identification of and structure-activity relationships in a series of glycine anilide ACAT inhibitors. Bioorganic & Medicinal Chemistry. 3(1). 29–39. 3 indexed citations

10.

Aalto‐Setälä, Katriina, C L Bisgaier, Anthony M.‐H. Ho, et al.. (1994). Intestinal expression of human apolipoprotein A-IV in transgenic mice fails to influence dietary lipid absorption or feeding behavior.. Journal of Clinical Investigation. 93(4). 1776–1786. 66 indexed citations

11.

Trivedi, Bharat K., Ann Holmes, Corinne E. Augelli‐Szafran, et al.. (1994). Inhibitors of Acyl-CoA:Cholesterol Acyltransferase (ACAT). 7. Development of a Series of Substituted N-Phenyl-N'-[(1-phenylcyclopentyl)methyl]ureas with Enhanced Hypocholesterolemic Activity. Journal of Medicinal Chemistry. 37(11). 1652–1659. 37 indexed citations

12.

Trivedi, Bharat K., Ann Holmes, C. John Blankley, et al.. (1993). Inhibitors of acyl-CoA:cholesterol acyltransferase. 4. A novel series of urea ACAT inhibitors as potential hypocholesterolemic agents. Journal of Medicinal Chemistry. 36(22). 3300–3307. 47 indexed citations

13.

Roark, W. Howard, Bruce D. Roth, Ann Holmes, et al.. (1993). Inhibitors of acyl-CoA:cholesterol acyltransferase (ACAT). 2. Modification of fatty acid anilide ACAT inhibitors: bioisosteric replacement of the amide bond. Journal of Medicinal Chemistry. 36(11). 1662–1668. 25 indexed citations

14.

Trivedi, Bharat K., et al.. (1993). Substituted N,N′-diphenyl ureas as potent inhibitors of acyl-CoA:Cholesterol acyltransferase (ACAT). Bioorganic & Medicinal Chemistry Letters. 3(2). 259–262. 4 indexed citations

15.

Augelli‐Szafran, Corinne E., C. John Blankley, Bruce D. Roth, et al.. (1993). Inhibitors of acyl-CoA:cholesterol acyltransferase. 5. Identification and structure-activity relationships of novel .beta.-keto amides as hypocholesterolemic agents. Journal of Medicinal Chemistry. 36(20). 2943–2949. 22 indexed citations

16.

Roth, Bruce D., C. John Blankley, M. L. HOEFLE, et al.. (1992). Inhibitors of acyl-CoA:cholesterol acyltransferase. 1. Identification and structure-activity relationships of a novel series of fatty acid anilide hypocholesterolemic agents. Journal of Medicinal Chemistry. 35(9). 1609–1617. 57 indexed citations

17.

Kaplan, Harvey, et al.. (1984). CI-906 and CI-907: new orally active nonsulfhydryl angiotensin-converting enzyme inhibitors.. PubMed. 43(5). 1326–9. 17 indexed citations

18.

Meyer, Robert, Arnold D. Essenburg, Ronald D. Smith, & Harvey Kaplan. (1982). Angiotensin converting enzyme inhibitors: modifications of a tripeptide analog. Journal of Medicinal Chemistry. 25(8). 996–999. 19 indexed citations

19.

Almquist, Ronald G., Clive Jennings-White, Robert Meyer, et al.. (1982). Derivatives of the potent angiotensin converting enzyme inhibitor 5(S)-benzamido-4-oxo-6-phenylhexanoyl-L-proline: effect of changes at postions 2 and 5 of the hexanoic acid portion. Journal of Medicinal Chemistry. 25(11). 1292–1299. 34 indexed citations

20.

Meyer, Robert, Ernest D. Nicolaides, Elizabeth A. Lunney, et al.. (1981). Novel synthesis of (S)-1-[5-(benzoylamino)-1,4-dioxo-6-phenylhexyl]-L-proline and analogs: potent angiotensin converting enzyme inhibitors. Journal of Medicinal Chemistry. 24(8). 964–969. 33 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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