Antonello Novelli

3.9k total citations · 2 hit papers
75 papers, 3.2k citations indexed

About

Antonello Novelli is a scholar working on Cellular and Molecular Neuroscience, Molecular Biology and Physiology. According to data from OpenAlex, Antonello Novelli has authored 75 papers receiving a total of 3.2k indexed citations (citations by other indexed papers that have themselves been cited), including 43 papers in Cellular and Molecular Neuroscience, 34 papers in Molecular Biology and 15 papers in Physiology. Recurrent topics in Antonello Novelli's work include Neuroscience and Neuropharmacology Research (35 papers), Ion channel regulation and function (16 papers) and Marine Toxins and Detection Methods (13 papers). Antonello Novelli is often cited by papers focused on Neuroscience and Neuropharmacology Research (35 papers), Ion channel regulation and function (16 papers) and Marine Toxins and Detection Methods (13 papers). Antonello Novelli collaborates with scholars based in Spain, Italy and United States. Antonello Novelli's co-authors include Richard C. Henneberry, Paul G. Lysko, Joseph Reilly, M. Teresa Fernández‐Sánchez, Alessandro Guidotti, Ferdinando Nicoletti, E. Costa, Hannu Alho, Ann M. Marini and Robert H. Lipsky and has published in prestigious journals such as Science, Journal of Neuroscience and Analytical Chemistry.

In The Last Decade

Antonello Novelli

73 papers receiving 3.1k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Glutamate becomes neurotoxic via the N-methyl-d-aspartate receptor when intracellular energy levels are reduced

1988 817 citations Antonello Novelli, Joseph Reilly et al. Brain Research profile →
The activation of inositol phospholipid metabolism as a signal- transducing system for excitatory amino acids in primary cultures of cerebellar granule cells

1986 404 citations Antonello Novelli et al. profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Antonello Novelli Spain	26	1.7k	1.5k	581	433	297	75	3.2k
Ragnhild E. Paulsen Norway	33	1.8k 1.0×	1.5k 1.0×	337 0.6×	248 0.6×	87 0.3×	105	3.4k
Gianfranco Di Renzo Italy	38	1.6k 0.9×	2.1k 1.4×	489 0.8×	425 1.0×	333 1.1×	132	4.2k
Robert W. Peoples United States	34	1.7k 1.0×	1.6k 1.1×	373 0.6×	214 0.5×	137 0.5×	81	3.0k
Larry Schmued United States	27	2.1k 1.2×	1.4k 0.9×	670 1.2×	1.0k 2.3×	836 2.8×	56	4.4k
Richard C. Henneberry United States	22	1.3k 0.8×	1.4k 0.9×	358 0.6×	304 0.7×	270 0.9×	37	2.5k
Lourdes Massieu Mexico	36	1.3k 0.8×	1.5k 1.0×	1.0k 1.8×	453 1.0×	311 1.0×	81	3.4k
Jason R. Cannon United States	32	985 0.6×	943 0.6×	473 0.8×	452 1.0×	1.4k 4.6×	74	3.4k
Clorinda Arias Mexico	30	888 0.5×	1.1k 0.8×	1.1k 1.8×	359 0.8×	128 0.4×	83	2.7k
R. Andrew Tasker Canada	23	796 0.5×	715 0.5×	295 0.5×	296 0.7×	107 0.4×	79	1.7k
Rodrigo Bainy Leal Brazil	37	1.0k 0.6×	1.4k 0.9×	444 0.8×	323 0.7×	202 0.7×	136	3.7k

Countries citing papers authored by Antonello Novelli

Since Specialization

Citations

This map shows the geographic impact of Antonello Novelli's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Antonello Novelli with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Antonello Novelli more than expected).

Fields of papers citing papers by Antonello Novelli

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Antonello Novelli. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Antonello Novelli. The network helps show where Antonello Novelli may publish in the future.

Co-authorship network of co-authors of Antonello Novelli

This figure shows the co-authorship network connecting the top 25 collaborators of Antonello Novelli. A scholar is included among the top collaborators of Antonello Novelli based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Antonello Novelli. Antonello Novelli is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Carboni, Ezio, et al.. (2021). Repurposing Ketamine in Depression and Related Disorders: Can This Enigmatic Drug Achieve Success?. Frontiers in Neuroscience. 15. 657714–657714. 17 indexed citations

Cuetos, Fernando, et al.. (2021). A new and short protocol to achieve the early diagnosis of mild cognitive impairment. Neurological Sciences. 42(9). 3687–3694. 5 indexed citations

Chen, Jun, Hongna Pan, Robert H. Lipsky, et al.. (2011). Cellular and Molecular Responses of Cultured Neurons to Stressful Stimuli. Dose-Response. 9(3). 416–33. 3 indexed citations

Marini, Ann M., Xiaobing Jiang, Xuan Wu, et al.. (2006). Preconditioning and neurotrophins: a model for brain adaptation to seizures, ischemia and other stressful stimuli. Amino Acids. 32(3). 299–304. 68 indexed citations

Zhu, Daming, Xuan Wu, Kenneth I. Strauss, et al.. (2005). N-methyl-D-aspartate and TrkB receptors protect neurons against glutamate excitotoxicity through an extracellular signal-regulated kinase pathway. Journal of Neuroscience Research. 80(1). 104–113. 60 indexed citations

Novelli, Antonello, et al.. (2005). Nefopam inhibits calcium influx, cGMP formation, and NMDA receptor-dependent neurotoxicity following activation of voltage sensitive calcium channels. Amino Acids. 28(2). 183–191. 46 indexed citations

Dı́az-Trelles, Ramón, M. Teresa Fernández‐Sánchez, Ann M. Marini, & Antonello Novelli. (2002). Antihistamine terfenadine inhibits calcium influx, cGMP formation, and NMDA receptor-dependent neurotoxicity following activation of L-type voltage sensitive calcium channels. Neurotoxicity Research. 4(1). 15–23. 13 indexed citations

Lipsky, Robert H., Ke Xu, Daming Zhu, et al.. (2001). Nuclear factor κB is a critical determinant in N‐methyl‐d‐aspartate receptor‐mediated neuroprotection. Journal of Neurochemistry. 78(2). 254–264. 142 indexed citations

Dı́az-Trelles, Ramón, et al.. (2000). Antihistamine terfenadine potentiates NMDA receptor-mediated calcium influx, oxygen radical formation, and neuronal death. Brain Research. 880(1-2). 17–27. 27 indexed citations

10.

Dı́az-Trelles, Ramón, Antonello Novelli, Giulia Puia, Mario Baraldi, & M. Teresa Fernández‐Sánchez. (2000). NMDA receptor dependent and independent components of veratridine toxicity in cultured cerebellar neurons are prevented by nanomolar concentrations of terfenadine. Amino Acids. 19(1). 263–272. 7 indexed citations

11.

G, Re, et al.. (1997). Effects of clenbuterol as a repartitioning agent onβ-Adrenoceptor concentrations in heart, bronchi and brain of veal calves. The Veterinary Journal. 153(1). 63–70. 32 indexed citations

12.

G, Re, P. Badino, Antonello Novelli, Maria G. Canese, & C. Girardi. (1995). Identification of β-adrenoceptor subtypes in bovine ovarian and myometrial cell membranes. British Veterinary Journal. 151(5). 567–578. 20 indexed citations

13.

G, Re, P. Badino, Antonello Novelli, et al.. (1995). Distribution of cytosolic oestrogen and progesterone receptors in the genital tract of the mare. Research in Veterinary Science. 59(3). 214–218. 9 indexed citations

14.

G, Re, et al.. (1994). Specific binding of dl‐cloprostenol and d‐cloprostenol to PG F_2α receptors in bovine corpus luteum and myometrial cell membranes. Journal of Veterinary Pharmacology and Therapeutics. 17(6). 455–458. 23 indexed citations

15.

Fernández‐Sánchez, M. Teresa & Antonello Novelli. (1993). Basic fibroblast growth factor protects cerebellar neurons in primary culture from NMDA and non‐NMDA receptor mediated neurotoxicity. FEBS Letters. 335(1). 124–131. 68 indexed citations

16.

Novelli, Antonello, et al.. (1992). The amnesic shellfish poison domoic acid enhances neurotoxicity by excitatory amino acids in cultured neurons. Amino Acids. 2(3). 233–244. 7 indexed citations

17.

Marini, Ann M. & Antonello Novelli. (1991). DL-threo-3-hydroxyaspartate reduces NMDA receptor activation by glutamate in cultured neurons. European Journal of Pharmacology. 194(1). 131–132. 21 indexed citations

18.

Fernández‐Sánchez, M. Teresa, V. Žitko, Santiago Gascón, & Antonello Novelli. (1991). The marine toxin okadaic acid is a potent neurotoxin for cultured cerebellar neurons. Life Sciences. 49(19). PL157–PL162. 47 indexed citations

19.

Henneberry, Richard C., Antonello Novelli, Jane A. Cox, & Paul G. Lysko. (1989). Neurotoxicity at the N‐Methyl‐D‐Aspartate Receptor in Energy‐Compromised Neurons An Hypothesis for Cell Death in Aging and Disease. Annals of the New York Academy of Sciences. 568(1). 225–233. 128 indexed citations

20.

Novelli, Antonello, Joseph Reilly, Paul G. Lysko, & Richard C. Henneberry. (1988). Glutamate becomes neurotoxic via the N-methyl-d-aspartate receptor when intracellular energy levels are reduced. Brain Research. 451(1-2). 205–212. 817 indexed citations breakdown →

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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