Anil Bagri
- Cellular and Molecular Neuroscience top 0.5%
- Molecular Biology top 5%
- Developmental Neuroscience top 0.2%
- Oncology top 2%
- Cell Biology top 2%
- Co-authors
- Marc Tessier‐LavigneSamuel J. PleasureJohn L.R. RubensteinÒscar MarínAvraham YaronRyan J. WattsHang ChenJudy Mak
- Topics
- Axon Guidance and Neuronal Signaling (15 papers)Angiogenesis and VEGF in Cancer (15 papers)Lymphatic System and Diseases (6 papers)
- Partner nations
- United StatesFranceFinland
In The Last Decade
Anil Bagri
26 papers receiving 4.0k citations
Peers
Comparison fields: 5 of 108
- Cellular and Molecular Neuroscience 2.4k
- Molecular Biology 2.3k
- Developmental Neuroscience 1.2k
- Oncology 990
- Cell Biology 656
Countries citing papers authored by Anil Bagri
This map shows the geographic impact of Anil Bagri's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Anil Bagri with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Anil Bagri more than expected).
Fields of papers citing papers by Anil Bagri
This network shows the impact of papers produced by Anil Bagri. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Anil Bagri. The network helps show where Anil Bagri may publish in the future.
Co-authorship network of co-authors of Anil Bagri
This figure shows the co-authorship network connecting the top 25 collaborators of Anil Bagri. A scholar is included among the top collaborators of Anil Bagri based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Anil Bagri. Anil Bagri is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 3 | |
| 2 | 23 | |
| 3 | 59 | |
| 4 | 14 | |
| 5 | 31 | |
| 6 | 33 | |
| 7 | 110 | |
| 8 | 64 | |
| 9 | 271 | |
| 10 | Mechanistic evaluation of the combination effect of anti-VEGF and chemotherapy | 3 |
| 11 | 425 | |
| 12 | 168 | |
| 13 | 97 | |
| 14 | 253 | |
| 15 | Neuropilins as Semaphorin receptors: in vivo functions in neuronal cell migration and axon guidance. | 96 |
| 16 | 188 | |
| 17 | 44 | |
| 18 | 296 | |
| 19 | 306 | |
| 20 | 249 |
About Anil Bagri
Anil Bagri is a scholar working on Developmental Neuroscience, Cellular and Molecular Neuroscience and Oncology, having authored 26 papers that have together received 4.0k indexed citations. Recurring topics across this work include Axon Guidance and Neuronal Signaling (15 papers), Angiogenesis and VEGF in Cancer (15 papers) and Lymphatic System and Diseases (6 papers). The work is most often cited by research in Developmental Neuroscience (1.2k citations), Cellular and Molecular Neuroscience (2.4k citations) and Cell Biology (656 citations). Anil Bagri has collaborated with scholars based in United States, France and Finland. Frequent co-authors include Marc Tessier‐Lavigne, Samuel J. Pleasure, Marc Tessier‐Lavigne, John L.R. Rubenstein, Òscar Marín, Avraham Yaron, Ryan J. Watts, Hang Chen, Judy Mak and Hwai-Jong Cheng. Their work appears in journals such as Science, Cell and Journal of Biological Chemistry.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.