Andreas Striebinger

855 total citations
8 papers, 690 citations indexed

About

Andreas Striebinger is a scholar working on Physiology, Molecular Biology and Cellular and Molecular Neuroscience. According to data from OpenAlex, Andreas Striebinger has authored 8 papers receiving a total of 690 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Physiology, 4 papers in Molecular Biology and 2 papers in Cellular and Molecular Neuroscience. Recurrent topics in Andreas Striebinger's work include Alzheimer's disease research and treatments (7 papers), Prion Diseases and Protein Misfolding (2 papers) and Neuroscience and Neuropharmacology Research (2 papers). Andreas Striebinger is often cited by papers focused on Alzheimer's disease research and treatments (7 papers), Prion Diseases and Protein Misfolding (2 papers) and Neuroscience and Neuropharmacology Research (2 papers). Andreas Striebinger collaborates with scholars based in Germany, United States and Belgium. Andreas Striebinger's co-authors include Stefan Barghorn, Heinz Hillen, Volker Nimmrich, Ulrich Ebert, Patrick Keller, John E. Harlan, Robert S. Bitner, Eve Barlow, Carsten Krantz and Martin Schmidt and has published in prestigious journals such as Journal of Neuroscience, Journal of Neurochemistry and Neurobiology of Aging.

In The Last Decade

Andreas Striebinger

7 papers receiving 664 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Andreas Striebinger Germany 6 599 363 162 140 96 8 690
Erika N. Cline United States 12 633 1.1× 372 1.0× 136 0.8× 191 1.4× 136 1.4× 20 924
Isam Qahwash United States 8 458 0.8× 376 1.0× 94 0.6× 82 0.6× 81 0.8× 10 655
Ann‐Christin Brorsson Sweden 14 448 0.7× 393 1.1× 103 0.6× 129 0.9× 76 0.8× 27 697
Carsten Krantz Switzerland 6 435 0.7× 292 0.8× 114 0.7× 93 0.7× 72 0.8× 8 554
Ryan Limbocker United States 13 368 0.6× 390 1.1× 109 0.7× 82 0.6× 71 0.7× 26 724
Rie Teraoka Japan 5 726 1.2× 353 1.0× 106 0.7× 182 1.3× 200 2.1× 6 818
Sofia Söllvander Sweden 7 554 0.9× 375 1.0× 132 0.8× 79 0.6× 70 0.7× 7 737
Youssra K. Al‐Hilaly United Kingdom 15 581 1.0× 479 1.3× 72 0.4× 99 0.7× 147 1.5× 29 892
Paul E. Fraser Canada 10 786 1.3× 585 1.6× 115 0.7× 166 1.2× 106 1.1× 12 957
W.S. Wade United States 7 549 0.9× 568 1.6× 123 0.8× 110 0.8× 104 1.1× 7 881

Countries citing papers authored by Andreas Striebinger

Since Specialization
Citations

This map shows the geographic impact of Andreas Striebinger's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Andreas Striebinger with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Andreas Striebinger more than expected).

Fields of papers citing papers by Andreas Striebinger

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Andreas Striebinger. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Andreas Striebinger. The network helps show where Andreas Striebinger may publish in the future.

Co-authorship network of co-authors of Andreas Striebinger

This figure shows the co-authorship network connecting the top 25 collaborators of Andreas Striebinger. A scholar is included among the top collaborators of Andreas Striebinger based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Andreas Striebinger. Andreas Striebinger is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

8 of 8 papers shown
1.
Neu, Marie A., Gregory K. Potts, Andreas Striebinger, et al.. (2025). A role for the autophagy receptor NBR1 in the degradation of tau aggregates. Neurobiology of Disease. 214. 107060–107060. 1 indexed citations
2.
Taylor, Keenan C., Dagmar E. Ehrnhoefer, Andreas Striebinger, et al.. (2023). Tau seed amplification assay reveals relationship between seeding and pathological forms of tau in Alzheimer’s disease brain. Acta Neuropathologica Communications. 11(1). 181–181. 9 indexed citations
3.
Barini, Erica, Mario Mezler, Miroslav Cik, et al.. (2021). Tau in the brain interstitial fluid is fragmented and seeding–competent. Neurobiology of Aging. 109. 64–77. 17 indexed citations
4.
Ibraghimov, Alexander, Edit Tarcsa, Andreas Striebinger, et al.. (2021). Off-target binding of an anti-amyloid beta monoclonal antibody to platelet factor 4 causes acute and chronic toxicity in cynomolgus monkeys. mAbs. 13(1). 14 indexed citations
5.
Hillen, Heinz, Stefan Barghorn, Andreas Striebinger, et al.. (2010). Generation and Therapeutic Efficacy of Highly Oligomer-Specific β-Amyloid Antibodies. Journal of Neuroscience. 30(31). 10369–10379. 85 indexed citations
7.
Gellermann, Gerald, et al.. (2008). Aβ-globulomers are formed independently of the fibril pathway. Neurobiology of Disease. 30(2). 212–220. 92 indexed citations
8.
Barghorn, Stefan, Volker Nimmrich, Andreas Striebinger, et al.. (2005). Globular amyloid β‐peptide1−42 oligomer − a homogenous and stable neuropathological protein in Alzheimer's disease. Journal of Neurochemistry. 95(3). 834–847. 471 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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