Dagmar E. Ehrnhoefer

5.2k total citations · 2 hit papers
34 papers, 4.0k citations indexed

About

Dagmar E. Ehrnhoefer is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Physiology. According to data from OpenAlex, Dagmar E. Ehrnhoefer has authored 34 papers receiving a total of 4.0k indexed citations (citations by other indexed papers that have themselves been cited), including 22 papers in Molecular Biology, 21 papers in Cellular and Molecular Neuroscience and 10 papers in Physiology. Recurrent topics in Dagmar E. Ehrnhoefer's work include Mitochondrial Function and Pathology (17 papers), Genetic Neurodegenerative Diseases (17 papers) and Alzheimer's disease research and treatments (10 papers). Dagmar E. Ehrnhoefer is often cited by papers focused on Mitochondrial Function and Pathology (17 papers), Genetic Neurodegenerative Diseases (17 papers) and Alzheimer's disease research and treatments (10 papers). Dagmar E. Ehrnhoefer collaborates with scholars based in Canada, Germany and United States. Dagmar E. Ehrnhoefer's co-authors include Erich E. Wanker, Jan Bieschke, Michael R. Hayden, Sabine Engemann, Rudi Lurz, Annett Boeddrich, Annalisa Pastore, Laura Masino, Martin Herbst and Jenny Russ and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Nature Medicine and Nature Communications.

In The Last Decade

Dagmar E. Ehrnhoefer

33 papers receiving 3.9k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

EGCG redirects amyloidogenic polypeptides into unstructured, off-pathway oligomers

2008 1.2k citations Dagmar E. Ehrnhoefer, Jan Bieschke et al. Nature Structural & Molecular Biology profile →
EGCG remodels mature α-synuclein and amyloid-β fibrils and reduces cellular toxicity

2010 853 citations Jan Bieschke, Jenny Russ et al. profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Dagmar E. Ehrnhoefer Canada	22	2.0k	1.8k	1.2k	827	372	34	4.0k
Annett Boeddrich Germany	10	1.6k 0.8×	1.2k 0.7×	842 0.7×	514 0.6×	249 0.7×	14	2.7k
Yona Levites United States	29	1.3k 0.7×	1.5k 0.8×	728 0.6×	675 0.8×	357 1.0×	54	3.8k
Tiziana Borsello Italy	31	2.1k 1.1×	1.5k 0.8×	1.2k 1.0×	272 0.3×	482 1.3×	82	4.1k
Hindupur K. Anandatheerthavarada United States	35	2.6k 1.3×	1.6k 0.9×	789 0.7×	803 1.0×	347 0.9×	51	4.7k
Valérie Vingtdeux United States	29	1.8k 0.9×	1.7k 0.9×	790 0.7×	293 0.4×	539 1.4×	47	4.2k
Sandra M. Cardoso Portugal	47	2.8k 1.4×	2.3k 1.3×	805 0.7×	1.1k 1.3×	996 2.7×	117	6.0k
Cristina Cecchi Italy	36	2.4k 1.2×	2.4k 1.3×	505 0.4×	889 1.1×	353 0.9×	97	4.3k
Sabine Engemann Germany	13	2.1k 1.1×	1.0k 0.6×	630 0.5×	446 0.5×	203 0.5×	17	3.2k
Shirley ShiDu Yan United States	41	2.9k 1.5×	2.8k 1.5×	716 0.6×	339 0.4×	759 2.0×	72	5.3k
Cassia Overk United States	25	952 0.5×	1.1k 0.6×	678 0.6×	1.2k 1.4×	202 0.5×	47	3.0k

Countries citing papers authored by Dagmar E. Ehrnhoefer

Since Specialization

Citations

This map shows the geographic impact of Dagmar E. Ehrnhoefer's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Dagmar E. Ehrnhoefer with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Dagmar E. Ehrnhoefer more than expected).

Fields of papers citing papers by Dagmar E. Ehrnhoefer

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Dagmar E. Ehrnhoefer. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Dagmar E. Ehrnhoefer. The network helps show where Dagmar E. Ehrnhoefer may publish in the future.

Co-authorship network of co-authors of Dagmar E. Ehrnhoefer

This figure shows the co-authorship network connecting the top 25 collaborators of Dagmar E. Ehrnhoefer. A scholar is included among the top collaborators of Dagmar E. Ehrnhoefer based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Dagmar E. Ehrnhoefer. Dagmar E. Ehrnhoefer is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Neu, Marie A., Gregory K. Potts, Andreas Striebinger, et al.. (2025). A role for the autophagy receptor NBR1 in the degradation of tau aggregates. Neurobiology of Disease. 214. 107060–107060. 1 indexed citations

Taylor, Keenan C., Dagmar E. Ehrnhoefer, Andreas Striebinger, et al.. (2023). Tau seed amplification assay reveals relationship between seeding and pathological forms of tau in Alzheimer’s disease brain. Acta Neuropathologica Communications. 11(1). 181–181. 9 indexed citations

Limorenko, Galina, Meltem Tatlı, Kolla Rajasekhar, et al.. (2023). Fully co-factor-free ClearTau platform produces seeding-competent Tau fibrils for reconstructing pathological Tau aggregates. Nature Communications. 14(1). 3939–3939. 7 indexed citations

Barini, Erica, Mario Mezler, Miroslav Cik, et al.. (2021). Tau in the brain interstitial fluid is fragmented and seeding–competent. Neurobiology of Aging. 109. 64–77. 17 indexed citations

Schöndorf, David C., Marie A. Neu, Xue Wang, et al.. (2021). SETD7-mediated monomethylation is enriched on soluble Tau in Alzheimer’s disease. Molecular Neurodegeneration. 16(1). 46–46. 19 indexed citations

Skotte, Niels H., Mahmoud A. Pouladi, Dagmar E. Ehrnhoefer, et al.. (2020). Compromised IGF signaling causes caspase-6 activation in Huntington disease. Experimental Neurology. 332. 113396–113396. 8 indexed citations

Behrendt, Annika, et al.. (2019). Asparagine endopeptidase cleaves tau at N167 after uptake into microglia. Neurobiology of Disease. 130. 104518–104518. 22 indexed citations

Ladha, Safia, et al.. (2018). Constitutive ablation of caspase-6 reduces the inflammatory response and behavioural changes caused by peripheral pro-inflammatory stimuli. Cell Death Discovery. 4(1). 40–40. 11 indexed citations

Ercan, Ebru, Sameh Eid, Christian Weber, et al.. (2017). A validated antibody panel for the characterization of tau post-translational modifications. Molecular Neurodegeneration. 12(1). 87–87. 60 indexed citations

10.

Skotte, Niels H., Shaun S. Sanders, Roshni R. Singaraja, et al.. (2016). Palmitoylation of caspase-6 by HIP14 regulates its activation. Cell Death and Differentiation. 24(3). 433–444. 36 indexed citations

11.

Riechers, Sean‐Patrick, Stefanie Butland, Yu Deng, et al.. (2016). Interactome network analysis identifies multiple caspase-6 interactors involved in the pathogenesis of HD. Human Molecular Genetics. 25(8). 1600–1618. 15 indexed citations

12.

Wong, Bibiana K. Y., Dagmar E. Ehrnhoefer, Rona K. Graham, et al.. (2015). Partial rescue of some features of Huntington Disease in the genetic absence of caspase-6 in YAC128 mice. Neurobiology of Disease. 76. 24–36. 41 indexed citations

13.

Ehrnhoefer, Dagmar E., et al.. (2015). A Huntingtin-based peptide inhibitor of caspase-6 provides protection from mutant Huntingtin-induced motor and behavioral deficits. Human Molecular Genetics. 24(9). 2604–2614. 49 indexed citations

14.

Ehrnhoefer, Dagmar E., Niels H. Skotte, Safia Ladha, et al.. (2013). p53 increases caspase-6 expression and activation in muscle tissue expressing mutant huntingtin. Human Molecular Genetics. 23(3). 717–729. 38 indexed citations

15.

Pouladi, Mahmoud A., Yuanyun Xie, Paola Conforti, et al.. (2012). NP03, a novel low-dose lithium formulation, is neuroprotective in the YAC128 mouse model of Huntington disease. Neurobiology of Disease. 48(3). 282–289. 45 indexed citations

16.

Ehrnhoefer, Dagmar E., Niels H. Skotte, Jane Savill, et al.. (2011). A Quantitative Method for the Specific Assessment of Caspase-6 Activity in Cell Culture. PLoS ONE. 6(11). e27680–e27680. 25 indexed citations

17.

Ehrnhoefer, Dagmar E., Bibiana K. Y. Wong, & Michael R. Hayden. (2011). Convergent pathogenic pathways in Alzheimer's and Huntington's diseases: shared targets for drug development. Nature Reviews Drug Discovery. 10(11). 853–867. 67 indexed citations

18.

Okamoto, Shu‐ichi, Mahmoud A. Pouladi, Maria Talantova, et al.. (2009). Balance between synaptic versus extrasynaptic NMDA receptor activity influences inclusions and neurotoxicity of mutant huntingtin. Nature Medicine. 15(12). 1407–1413. 335 indexed citations

19.

Ehrnhoefer, Dagmar E., Jan Bieschke, Annett Boeddrich, et al.. (2008). EGCG redirects amyloidogenic polypeptides into unstructured, off-pathway oligomers. Nature Structural & Molecular Biology. 15(6). 558–566. 1191 indexed citations breakdown →

20.

Ehrnhoefer, Dagmar E., Martin L. Duennwald, Jennifer L. Wacker, et al.. (2006). Green tea (−)-epigallocatechin-gallate modulates early events in huntingtin misfolding and reduces toxicity in Huntington's disease models. Human Molecular Genetics. 15(18). 2743–2751. 332 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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