Aleksi Lahti

1.7k citations

17 papers · 1.5k indexed · 1 hit paper · h-index 15

Toxicology top 5%
Immunology top 10%
- Neutrophil, Myeloperoxidase and Oxidative Mechanisms 5
Pharmacology top 5%
- Inflammatory mediators and NSAID effects 3
Biochemistry top 5%
Cancer Research top 10%
- NF-κB Signaling Pathways 2
Physiology
- Nitric Oxide and Endothelin Effects 8
- Asthma and respiratory diseases 2
Oncology
- Cytokine Signaling Pathways and Interactions 3
Molecular Biology
- Protein Kinase Regulation and GTPase Signaling 2
- Melanoma and MAPK Pathways 2

Co-authors: Hannu Kankaanranta Riku Korhonen Eeva Moilanen Mari Hämäläinen Ulla Jalonen Riina Nieminen Mark A. Giembycz Richard G. Knowles
Cited by: Toxicology Immunology Pharmacology
Journals: Biochemical Journal (2 papers)Molecular Pharmacology (2 papers)European Journal of Pharmacology (2 papers)
Partner nations: Finland Netherlands Canada

In The Last Decade

Aleksi Lahti

16 papers receiving 1.4k citations

Hit Papers

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Peers

Countries citing papers authored by Aleksi Lahti

Since Specialization

Citations

This map shows the geographic impact of Aleksi Lahti's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Aleksi Lahti with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Aleksi Lahti more than expected).

Fields of papers citing papers by Aleksi Lahti

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Aleksi Lahti. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Aleksi Lahti. The network helps show where Aleksi Lahti may publish in the future.

Co-authorship network

The 25 scholars most cited alongside Aleksi Lahti, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Aleksi Lahti Line = papers co-authored together Aleksi Lahti links everyone, so they are left out of the graph.

All Works

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17 of 17 papers shown

#	Work
1	Pupils’ experience of noise in two acoustically different classrooms Facilities ·Jenni Radun,Jeeva Soundarajan,Aleksi Lahti,Marjaana Veermans,Alex Santiago Cevallos Carvajal,Valtteri Hongisto	2023	5
2	Generation of student interest in an inquiry-based mobile learning environment Frontline Learning Research ·Francisco José Escámez Mañas,Marjaana Veermans,Aleksi Lahti,Koen Veermans	2017	14
3	Tumour Necrosis Factor-α Regulates Human Eosinophil Apoptosis via Ligation of TNF-Receptor 1 and Balance between NF-κB and AP-1 PLoS ONE ·Hannu Kankaanranta,Pinja Ilmarinen,Xianzhi Zhang,Ian M. Adcock,Aleksi Lahti,Peter J. Barnes,Mark A. Giembycz,Mark A. Lindsay,Eeva Moilanen	2014	49
4	Regulation of inducible nitric oxide synthase expression and nitric oxide production by mitogen-activated protein kinase pathways Trepo - Institutional Repository of Tampere University ·Aleksi Lahti	2012	0
5	Inhibition of p38 mitogen-activated protein kinase enhances c-Jun N-terminal kinase activity: Implication in inducible nitric oxide synthase expression BMC Pharmacology ·Aleksi Lahti,Outi Sareila,Hannu Kankaanranta,Eeva Moilanen	2006	33
6	JNK inhibitor SP600125 reduces COX-2 expression by attenuating mRNA in activated murine J774 macrophages International Immunopharmacology ·Riina Nieminen,Aleksi Lahti,Ulla Jalonen,Hannu Kankaanranta,Eeva Moilanen	2006	41
7	Nitric Oxide Production and Signaling in Inflammationbreakdown → Current Drug Targets - Inflammation & Allergy ·Riku Korhonen,Aleksi Lahti,Hannu Kankaanranta,Eeva Moilanen	2005	805
8	Inhibitors of Mitogen‐Activated Protein KinasesDownregulate COX‐2 Expression in Human Chondrocytes Mediators of Inflammation ·Riina Nieminen,Elisabet Oktavia,Aleksi Lahti,Katriina Vuolteenaho,Ulla Jalonen,Hannu Kankaanranta,Mary B. Goldring,Eeva Moilanen	2005	56
9	TGFβ inhibits IL-1 -induced iNOS expression and NO production in immortalized chondrocytes Inflammation Research ·Katriina Vuolteenaho,Teemu Moilanen,Ulla Jalonen,Aleksi Lahti,R. Nieminen,Henk M. van Beuningen,P.M. van der Kraan,Eeva Moilanen	2005	25
10	Inhibition of tristetraprolin expression by dexamethasone in activated macrophages Biochemical Pharmacology ·Ulla Jalonen,Aleksi Lahti,Riku Korhonen,Hannu Kankaanranta,Eeva Moilanen	2005	34
11	c-Jun NH2-Terminal Kinase Inhibitor Anthra(1,9-cd)pyrazol-6(2H)-one Reduces Inducible Nitric-Oxide Synthase Expression by Destabilizing mRNA in Activated Macrophages Molecular Pharmacology ·Aleksi Lahti,Ulla Jalonen,Hannu Kankaanranta,Eeva Moilanen	2003	76
12	Regulation of eosinophil apoptosis by nitric oxide: Role of c-Jun-N-terminal kinase and signal transducer and activator of transcription 5 Journal of Allergy and Clinical Immunology ·Xianzhi Zhang,Akancha Pandey,Eeva Moilanen,Aleksi Lahti,Mari Hämäläinen,Mark A. Giembycz,Peter J. Barnes,Mark A. Lindsay,Hannu Kankaanranta	2003	41
13	P38 mitogen-activated protein kinase inhibitor SB203580 has a bi-directional effect on iNOS expression and NO production European Journal of Pharmacology ·Aleksi Lahti,Hannu Kankaanranta,Eeva Moilanen	2002	30
14	Calcineurin inhibitors, cyclosporin A and tacrolimus inhibit expression of inducible nitric oxide synthase in colon epithelial and macrophage cell lines European Journal of Pharmacology ·Mari Hämäläinen,Aleksi Lahti,Eeva Moilanen	2002	42
15	Dexamethasone Inhibits Inducible Nitric-Oxide Synthase Expression and Nitric Oxide Production by Destabilizing mRNA in Lipopolysaccharide-Treated Macrophages Molecular Pharmacology ·Riku Korhonen,Aleksi Lahti,Mari Hämäläinen,Hannu Kankaanranta,Eeva Moilanen	2002	155
16	Bi-directional effects of the elevation of intracellular calcium on the expression of inducible nitric oxide synthase in J774 macrophages exposed to low and to high concentrations of endotoxin Biochemical Journal ·Riku Korhonen,Hannu Kankaanranta,Aleksi Lahti,Han Tu,Richard G. Knowles,Eeva Moilanen	2001	35
17	Bi-directional effects of the elevation of intracellular calcium on the expression of inducible nitric oxide synthase in J774 macrophages exposed to low and to high concentrations of endotoxin Biochemical Journal ·K Saputri,Hannu Kankaanranta,Aleksi Lahti,Han Tu,Richard G. Knowles,Eeva Moilanen	2001	25

About Aleksi Lahti

Aleksi Lahti is a scholar working on Physiology, Immunology and Toxicology, having authored 17 papers that have together received 1.5k indexed citations. Recurring topics across this work include Nitric Oxide and Endothelin Effects (8 papers), Neutrophil, Myeloperoxidase and Oxidative Mechanisms (5 papers), Inflammatory mediators and NSAID effects (3 papers), Cytokine Signaling Pathways and Interactions (3 papers), Protein Kinase Regulation and GTPase Signaling (2 papers), Asthma and respiratory diseases (2 papers), NF-κB Signaling Pathways (2 papers) and Melanoma and MAPK Pathways (2 papers). The work is most often cited by research in Toxicology (59 citations), Immunology (344 citations) and Pharmacology (121 citations). Aleksi Lahti has collaborated with scholars based in Finland, Netherlands and Canada. Frequent co-authors include Hannu Kankaanranta, Riku Korhonen, Eeva Moilanen, Eeva Moilanen, Mari Hämäläinen, Ulla Jalonen, Riina Nieminen, Mark A. Giembycz, Richard G. Knowles and Peter J. Barnes. Their work appears in journals such as Biochemical Journal, Molecular Pharmacology, European Journal of Pharmacology, Inflammation Research and Frontline Learning Research.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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